We are analyzing https://link.springer.com/article/10.1007/s10555-022-10068-w.

Title:
Natural compounds targeting nuclear receptors for effective cancer therapy | Cancer and Metastasis Reviews
Description:
Human nuclear receptors (NRs) are a family of forty-eight transcription factors that modulate gene expression both spatially and temporally. Numerous biochemical, physiological, and pathological processes including cell survival, proliferation, differentiation, metabolism, immune modulation, development, reproduction, and aging are extensively orchestrated by different NRs. The involvement of dysregulated NRs and NR-mediated signaling pathways in driving cancer cell hallmarks has been thoroughly investigated. Targeting NRs has been one of the major focuses of drug development strategies for cancer interventions. Interestingly, rapid progress in molecular biology and drug screening reveals that the naturally occurring compounds are promising modern oncology drugs which are free of potentially inevitable repercussions that are associated with synthetic compounds. Therefore, the purpose of this review is to draw our attention to the potential therapeutic effects of various classes of natural compounds that target NRs such as phytochemicals, dietary components, venom constituents, royal jelly–derived compounds, and microbial derivatives in the establishment of novel and safe medications for cancer treatment. This review also emphasizes molecular mechanisms and signaling pathways that are leveraged to promote the anti-cancer effects of these natural compounds. We have also critically reviewed and assessed the advantages and limitations of current preclinical and clinical studies on this subject for cancer prophylaxis. This might subsequently pave the way for new paradigms in the discovery of drugs that target specific cancer types.
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pubmed, google, scholar, article, cas, cancer, receptor, journal, cells, central, cell, nuclear, prostate, research, human, molecular, wang, chen, expression, lee, breast, natural, chemistry, inhibits, receptors, androgen, activity, apoptosis, growth, acid, effects, marine, activation, signaling, curcumin, letters, international, httpsdoiorg, biology, anticancer, pharmacology, liu, induces, kim, zhang, lin, yang, medicinal, estrogen, therapy,

Topics {✒️}

bt/556/ne/u-excel/2016 grant awarded pd-l1/itgb6/stat3 signaling axis mediated phosphoinositide-3-kinase/akt/mammalian target peroxisome proliferator-activated receptor-gamma attenuates tnf-alpha-mediated lipolysis bcr/abl-regulated set protein nf-kappab-regulated inflammatory cytokines acetyl-11-keto-β-boswellic acid wnt/β-catenin signaling pathway wnt/beta-catenin signaling pathways 17beta-oestradiol-treated mcf-7 cells month download article/chapter akt/mtor/s6k1 signaling cascades mitogen-activated protein kinase n-nitrosodiethylamine-induced lung tumorigenesis mir-181c-3p/pparalpha axis nf-κb-regulated gene expression nur77/hif-1alpha axis si-wu-tang series decoctions improves lps-induced inflammation natural pregnane-x-receptor agonists γ-tocopherol inhibits tumorigenesis fxr/nf-κb signalling pathway homocysteine-induced endothelial dysfunction x-ray-induced killing nf-kappab signaling pathways stat3/jak2 signaling cascade potent pregnane-x-receptor agonist thyroid-hormone receptor-mediated castration-resistant prostate cancer suppress beta-catenin signaling androgen-dependent lncap cells trans-11-conjugated linoleic acid inhibits lysosomal-dependent migration pi3k-induced ap1 cre modulates hormone-dependent transactivation neo-tanshinlactone selectively inhibits nr-mediated signaling pathways fxr-regulated bile acid inhibiting bfgf-induced lymphangiogenesis tlr4-androgen receptor axis estrogen-mediated breast cancer 26s proteasome-mediated degradation nuclear receptor-driven alterations cutaneous t-cell lymphomas nuclear-receptor interacting protein x-irradiated hela cells il-4rα-tyk2-stat6 depurinating estrogen-dna adducts pi3k/akt signaling pathway

Questions {❓}

Regulation of nuclear factor-kappaB (NF-κB) signaling pathway by non-coding RNAs in cancer: Inhibiting or promoting carcinogenesis?

Schema {🗺️}

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      description:Human nuclear receptors (NRs) are a family of forty-eight transcription factors that modulate gene expression both spatially and temporally. Numerous biochemical, physiological, and pathological processes including cell survival, proliferation, differentiation, metabolism, immune modulation, development, reproduction, and aging are extensively orchestrated by different NRs. The involvement of dysregulated NRs and NR-mediated signaling pathways in driving cancer cell hallmarks has been thoroughly investigated. Targeting NRs has been one of the major focuses of drug development strategies for cancer interventions. Interestingly, rapid progress in molecular biology and drug screening reveals that the naturally occurring compounds are promising modern oncology drugs which are free of potentially inevitable repercussions that are associated with synthetic compounds. Therefore, the purpose of this review is to draw our attention to the potential therapeutic effects of various classes of natural compounds that target NRs such as phytochemicals, dietary components, venom constituents, royal jelly–derived compounds, and microbial derivatives in the establishment of novel and safe medications for cancer treatment. This review also emphasizes molecular mechanisms and signaling pathways that are leveraged to promote the anti-cancer effects of these natural compounds. We have also critically reviewed and assessed the advantages and limitations of current preclinical and clinical studies on this subject for cancer prophylaxis. This might subsequently pave the way for new paradigms in the discovery of drugs that target specific cancer types.
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      name:Department of Studies in Molecular Biology, University of Mysore, Manasagangotri, Mysore, India
      name:Division of Cancer Stem Cells and Cardiovascular Regeneration, School of Regenerative Medicine, Manipal Academy of Higher Education (MAHE), Bangalore, India
      name:Cuor Stem Cellutions Pvt Ltd, Manipal Institute of Regenerative Medicine, Manipal Academy of Higher Education (MAHE), Bangalore, India
      name:Division of Cellular and Molecular Research, Humphrey Oei Institute of Cancer Research, National Cancer Centre, Singapore, Singapore
      name:Institution of Excellence, University of Mysore, Manasagangotri, Mysore, India
      name:Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore, Singapore, Singapore
      name:Cancer Biology Laboratory, Department of Biosciences and Bioengineering, Indian Institute of Technology Guwahati, Guwahati, India
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