DOI . ORG {
}
We began analyzing https://link.springer.com/article/10.1007/s10068-022-01114-y, but it redirected us to https://link.springer.com/article/10.1007/s10068-022-01114-y. The analysis below is for the second page.
Title[redir]:
Nutritional components as mitigators of cellular senescence in organismal aging: a comprehensive review | Food Science and Biotechnology
Description:
The process of cellular senescence is rapidly emerging as a modulator of organismal aging and disease. Targeting the development and removal of senescent cells is considered a viable approach to achieving improved organismal healthspan and lifespan. Nutrition and health are intimately linked and an appropriate dietary regimen can greatly impact organismal response to stress and diseases including during aging. With a renewed focus on cellular senescence, emerging studies demonstrate that both primary and secondary nutritional elements such as carbohydrates, proteins, fatty acids, vitamins, minerals, polyphenols, and probiotics can influence multiple aspects of cellular senescence. The present review describes the recent molecular aspects of cellular senescence-mediated understanding of aging and then studies available evidence of the cellular senescence modulatory attributes of major and minor dietary elements. Underlying pathways and future research directions are deliberated to promote a nutrition-centric approach for targeting cellular senescence and thus improving human health and longevity.
Title[redir]:
Nutritional components as mitigators of cellular senescence in organismal aging: a comprehensive review | Food Science and Biotechnology
Description:
The process of cellular senescence is rapidly emerging as a modulator of organismal aging and disease. Targeting the development and removal of senescent cells is considered a viable approach to achieving improved organismal healthspan and lifespan. Nutrition and health are intimately linked and an appropriate dietary regimen can greatly impact organismal response to stress and diseases including during aging. With a renewed focus on cellular senescence, emerging studies demonstrate that both primary and secondary nutritional elements such as carbohydrates, proteins, fatty acids, vitamins, minerals, polyphenols, and probiotics can influence multiple aspects of cellular senescence. The present review describes the recent molecular aspects of cellular senescence-mediated understanding of aging and then studies available evidence of the cellular senescence modulatory attributes of major and minor dietary elements. Underlying pathways and future research directions are deliberated to promote a nutrition-centric approach for targeting cellular senescence and thus improving human health and longevity.
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Keywords {🔍}
pubmed, article, google, scholar, cas, central, senescence, aging, cell, cellular, cells, journal, senescent, wang, disease, mice, human, nutrition, sharma, nature, ageing, research, health, chen, zhang, liu, kim, polyphenols, endothelial, biology, dietary, fatty, effects, sciences, food, science, acids, molecular, gerontology, van, medicine, oxidative, review, telomere, healthy, kirkland, immune, clinical, american, gut,
Topics {✒️}
late-onset doxorubicin-induced cardiotoxicity ampk/sirt1/nf-κb signaling pathway fish-oil supplementation enhances pampkα/sirt1/foxo3a signaling pathway oxidative-stress-induced endothelial senescence d-galactose-induced subacute aging low-protein high-carbohydrate diets month download article/chapter suppressing ros-akt-mtor axis h2o2-induced premature senescence fish oil supplementation stress-induced premature senescence omega-3 fatty acids oxidative stress-induced apoptosis le couteur dg berberine suppresses gero-conversion sirt1-nad cd38 axis irreparable dna damage endothelial colony-forming cells cellular senescence-mediated understanding nestin-gfp mice induced p62-dependent selective autophagy nampt/sirt1/nlrp3 pathway daf-16/foxo transcription factor kiecolt-glaser jk pi3k/akt/mtor pathway natural anti-oxidant quercetin th1/th2 immune homeostasis hla-e-mediated nk cell-free culture supernatant central nervous system redox-telomere-antioncogene axis full article pdf essential fatty acids article diwan ameliorates cartilage degeneration high dietary protein main age-related diseases aging-related vascular dysfunction blagosklonny mv hpa axis functions therapy-induced senescence aneuploidy-induced senescence bleomycin-induced senescence long-chain n-3 article food science increased molecular damage high protein intake phytochemical epigallocatechin gallate tocotrienol-rich fraction
Questions {❓}
- Are we ill because we age?
- Do senescent cells play a role in Alzheimer’s disease?
- Senescence and apoptosis: dueling or complementary cell fates?
- Senescence in post-mitotic cells: a driver of aging?
- Senescent microglia: the key to the ageing brain?
- Telomere length: a marker of disease susceptibility?
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