Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5864239/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5864239/. The analysis below is for the second page.

Title[redir]:
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 - PMC
Description:
Over the past decade, the Nomenclature Committee on Cell Death (NCCD) has formulated guidelines for the definition and interpretation of cell death from morphological, biochemical, and functional perspectives. Since the field continues to expand and ...

Matching Content Categories {πŸ“š}

  • Education
  • Telecommunications
  • Fitness & Wellness

Content Management System {πŸ“}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,694,437 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {πŸ’Έ}

We don’t know how the website earns money.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Ncbi.nlm.nih.gov might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {πŸ”}

doi, pubmed, google, scholar, cell, pmc, article, free, death, cells, apoptosis, biol, activation, cancer, mitochondrial, rcd, nat, protein, nature, ripk, casp, bcl, caspase, signaling, necroptosis, mol, autophagy, membrane, sci, differ, receptor, activity, bax, cdd, kinase, family, ferroptosis, apoptotic, science, role, cellular, proteins, specific, including, necrosis, complex, rev, bak, chem, mechanisms,

Topics {βœ’οΈ}

amino]butyl]amino]-4-oxo-pentanoic acid hydrate l-leucyl-l-leucine methyl ester n-benzyloxycarbonyl-val-ala-asp phorbol-12-myristate-13-acetate-induced protein 1 macrophage-induced glutamate-dependent cytotoxicity raf-mek-erk pathway engage bax/bak-dependent momp-driven nf-kappab-independent cell-death switch g-protein-coupled receptor signaling voltage-dependent anion channels voltage-dependent anion channel rip1-parp-aif-dependent pathway n-terminal pore-forming domain p19arf/p53-mediated apoptotic checkpoint oncogenic-ras-harboring cancer cells tnf-induced nf-kappab signal engineered small-molecule-activated protease genuine multi-bh-domain protein reaper-grim-hid-mediated suppression including cd4+cd25+foxp3+ regulatory gram-negative bacteria-induced pyroptosis p62-keap1-nrf2 pathway protects cyclic gmp-amp synthase erk-regulated alphab-crystallin induction mediates alpha-synuclein cytotoxicity triple-negative breast carcinoma inflammasome components cias1/cryopyrin/nlrp3 autophagy-inflammation-cell death axis p53-mediated cell-cycle arrest apaf-1/caspase-9-mediated caspase activity autocrine/paracrine signaling pathway fda-approved alkylating agent lysosome-dependent cell death pmc beta search mg2+/mn2+ dependent 1b regulate mpt-driven necrosis transcription factor nf-kappab abolishes mpt-driven necrosis znf304-integrin axis protects high-quality articles dealing reverts niemann-pick disease interferon-beta pathways mediated mediates resveratrol-induced autophagy apoptosis-inducing factor substitutes 29362479 abstract feed-forward amplification circuitry concluding remarks fadd-homologous ice/ced-3 mir-133a tumor suppressor diffuse large b-cell

Questions {❓}

  • Ferroptosis: bug or feature?
  • How neutrophils die or survive along NET release and is "NETosis" = necroptosis?
  • Metabolism during ECM Detachment: Achilles Heel of Cancer Cells?
  • Neutrophil extracellular traps: is immunity the second function of chromatin?
  • Pyroptosis - a cell death modality of its kind?
  • Questions and controversies in innate immune research: what is the physiological role of NLRP3?
  • Why does the specific activation of RCD (that should not be confounded with the alteration of normal cellular functions, although this also can lead to RCD) appear as a much simpler clinical objective than its inhibition?

External Links {πŸ”—}(2131)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {πŸ“¦}

  • Ncbi

9.81s.