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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers

We began analyzing https://pnas.org/doi/full/10.1073/pnas.1301218110, but it redirected us to https://pnas.org/doi/full/10.1073/pnas.1301218110. The analysis below is for the second page.

Title[redir]:
Interferon-induced RIP1/RIP3-mediated necrosis requires PKR and is licensed by FADD and caspases | PNAS
Description:
Interferons (IFNs) are cytokines with powerful immunomodulatory and antiviral properties, but less is known about how they induce cell death. Here, we show that both type I (α/β) and type II (γ) IF...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🚦 Initial Traffic: less than 1k visitors per month


Based on our best estimate, this website will receive around 19 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

We can't see how the site brings in money.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Doi.org might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

fadd, rip, pkr, mefs, fig, necrosis, ifnγ, death, formation, cells, kinase, cell, necrosome, riprip, scholar, crossref, pubmed, google, ifns, phosphorylation, ngml, complex, necrotic, protein, represent, inhibitor, type, wildtype, treated, bars, nec, caspases, caspase, ifninduced, viability, error, zvad, signaling, jak, ifn, kinases, transcription, activate, treatment, association, mechanism, activity, stat, ifnβ, expression,

Topics {✒️}

ifn-γ–induced necrosis—rela−/− mefs ifn-γ—induces progressive rip1–rip3 induce pkr–rip1–rip3-dependent necrosis tnf-α–stimulated rip1–rip3 complex ifn-γplus high-dose z-vad tnf-α–induced rip1–rip3 association ifn-γ–driven rip1–rip3 association ifn-γ–driven necrosome formation t-cell receptor-mediated necroptosis g2/m-arrested wild-type mefs tnf-α–induced necrotic death ifn-γ–activated necrosome formation jak/stat-dependent transcriptional activation classical ifn-γ–induced signaling ifn–pkr–rip kinase axis requires jak1/stat1-dependent transcription z-vad–exposed wild-type mefs ifn-γ–induced cell death ifn-γ–driven transcriptional mechanism licenses interferon-induced necrosis ifn-β–triggered cell death ifn-γ–triggered necrosis inhibited ifn-driven necrosome formation rna-dependent protein kinase rip1–rip3 complex formation licenses ifn-mediated elimination pkr-driven necrosome formation interferon-induced necrosis proceeds 1/rip3 kinase-mediated necrosis rip1–rip3 “necrosome” complex rna-responsive kinase pkr rip1–rip3 kinase complex rip1–rip3 necrosome formation dsrna-dependent protein kinase pkr–rip1 complex formation social media facebook rip1/rip3-dependent necrosis tnf-α–induced necrosome ifn-triggered necrotic death rip1 kinase-dependent necrosis classical jak–stat signaling progressive transcription-dependent mechanism phospho-rip1–rip3 complex ifn-induced necrotic death ifn-driven cell death noc-induced necrosome formation ifn-triggered necrosis differs order ifn-triggered necrosis tnf-α–induced necrosis nascent rip1–rip3 necrosome

Questions {❓}

  • We have identified phosphorylation of FADD as a putative mechanism that permits IFN-activated necrosis, but how and when does phosphorylation of FADD license necrosis?

Social Networks {👍}(4)

External Links {🔗}(325)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Tag Manager

Libraries {📚}

  • Dropzone.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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