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Keywords {🔍}

ferroptosis, pubmed, cancer, article, cell, google, scholar, cells, cas, central, death, iron, expression, ros, apoptosis, erastin, protein, autophagy, ferroptotic, pathway, targeting, tumor, nrf, wang, gsh, zhang, induce, system, breast, res, cys, lipid, kinase, treatment, role, slca, activation, stress, activity, level, mol, regulation, process, cellular, human, promote, regulate, gpx, accumulation, receptor,

Topics {✒️}

ras-mitogen-activated protein kinase oncogenic-ras-harboring cancer cells ras/raf/mek pathway overactive p62-keap1-nrf2 pathway rna-binding protein elavl1/hur k-ras oncogenic signal p62-keap1-nrf2 pathway plays ferroptotic agent-mediated sensitization article download pdf overactive ras-mek pathway dmt1-iron-responsive element ras-mutant cancer cells specific light-chain submission lysosome-dependent degradation pathway small heat-shock protein death-inducing signaling complex iron-dependent ros accretion oxidative stress-induced ferroptosis ros-induced stress signaling pi3k-dependent proliferation hsf1-dependent hspb1 contributes iron metabolism-related genes cystine/glutamate exchanger slc7a11 high-efficiency anticancer therapy lymphoid-specific helicase genotype-selective antitumor agents xct/slc7a11 dependent manner central nervous system mon-p53-treated cells bcl-2 family proteins p53-mediated tumor suppression hepatic stellate cells nutrient-deprived cancer cells mir-200a readily induce mt-1g knockdown contributed central south university metal-organic network suppresses ferritin degradation heat shock factor-1 p53-mediated tumour suppression anti-malarial drug artesunate oncogenic ras mutation mir-20a regulates expression cystathionine-γ-lyase function pro-apoptotic member anti-tumor immunotherapy treatment ampk-mediated becn1 phosphorylation high-level ros result killing mesenchymal-state cells ferroptosis-based cancer therapy

Questions {❓}

• Are the ferroptosis inducers effective in killing the cancer cells in pre-clinical or clinical trials?
• Can ferroptosis enhance the cell immunogenicity to the host and thereby evoke an adaptive immune response, as shown in necroptotic cell death?
• Ferroptosis and apoptosis: switch, synergism, or antagonism?
• Ferroptosis: bug or feature?
• How do we fit ferroptosis in the family of regulated cell death?
• How does ferroptosis interact with other cell death at the molecular level and how could these pathways be mapped and integrated into the cellular events?
• How does ncRNA regulate the process of ferroptotic cell death?
• How does p53 induce apoptosis and how does this relate to p53-mediated tumour suppression?
• Moreover, it is still obscure that what kind of role P53-target genes take part in manipulating of the ferroptotic cell death [17]?
• Tackling the cancer stem cells—what challenges do they pose?

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      headline:Ferroptosis, a new form of cell death: opportunities and challenges in cancer
      description:Ferroptosis is a novel type of cell death with distinct properties and recognizing functions involved in physical conditions or various diseases including cancers. The fast-growing studies of ferroptosis in cancer have boosted a perspective for its usage in cancer therapeutics. Here, we review the current findings of ferroptosis regulation and especially focus on the function of ncRNAs in mediating the process of cell ferroptotic death and on how ferroptosis was in relation to other regulated cell deaths. Aberrant ferroptosis in diverse cancer types and tissues were summarized, and we elaborated recent data about the novel actors of some “conventional” drugs or natural compounds as ferroptosis inducers in cancer. Finally, we deliberate future orientation for ferroptosis in cancer cells and current unsettled issues, which may forward the speed of clinical use of ferroptosis induction in cancer treatment.
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         Apoptosis
         Autophagy
         NcRNAs
         Cancers
         P53
         Oncology
         Hematology
         Cancer Research
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