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  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
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We began analyzing https://www.nature.com/articles/nrendo.2013.29, but it redirected us to https://www.nature.com/articles/nrendo.2013.29. The analysis below is for the second page.

Title[redir]:
Oxidative stress and the ageing endocrine system | Nature Reviews Endocrinology
Description:
A progressive decline in cellular function and organismal fitness and an increase in age-related diseases are associated with ageing; many theories have attempted to explain this phenomenon. This Review outlines the current knowledge of the effects of oxidative stress on endocrine organs and tissues and suggests that inflammation is the link between oxidative stress and ageing. Ageing is a process characterized by a progressive decline in cellular function, organismal fitness and increased risk of age-related diseases and death. Several hundred theories have attempted to explain this phenomenon. One of the most popular is the

Matching Content Categories {📚}

  • Education
  • Health & Fitness
  • Science

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 80,479,999 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We see no obvious way the site makes money.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Doi.org might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {🔍}

pubmed, google, scholar, article, cas, oxidative, stress, ageing, central, aging, thyroid, endocrinol, cell, human, clin, biol, cancer, free, rev, endocrine, role, nature, reactive, oxygen, med, cells, metab, antioxidant, species, res, function, factor, disease, franceschi, radic, adipose, obesity, sci, centenarians, expression, tissue, diabetes, exp, rat, dna, system, agerelated, inflammation, growth, mol,

Topics {✒️}

nature portfolio permissions reprints privacy policy cancer research editing research leading advertising nature 345 nature hypothalamic–pituitary-adrenal axis activity author information authors tumor necrosis factor-alpha social media unrepairable double-strand breaks hypothalamic–pituitary-adrenal activity author correspondence castration-resistant prostate cancer hypothalamic–pituitary-adrenocortical axis salvioli researched data st-elevation myocardial infarction advanced glycation end-products total oxidant/antioxidant status hydrogen peroxide-induced production hypothalamus–pituitary-adrenal axis springerlink instant access nuclear factor κb senescence-accelerated mice analyzed permissions tumor necrosis factor pancreatic β-cell function glucose-stimulated insulin secretion development age-related endocrine diseases reactive oxygen species tissue-specific micrornas italy tgf-beta signaling nitrogen species generation increased lipid peroxidation growth hormone deficiency females live longer von willebrand factor improves exercise capacity reactive oxygen intermediates privacy monocyte chemoattractant protein-1 androgen receptor signaling nf-kb signaling decreased oxidative damage testosterone-producing cells

Questions {❓}

  • Age-related increase in mitochondrial superoxide generation in the testosterone-producing cells of Brown Norway rat testes: relationship to reduced steroidogenic function?
  • Beta-cell deterioration during diabetes: what's in the gun?
  • Hydrogen peroxide-induced production of a 40 kDa immunoreactive thyroglobulin fragmentin human thyroid cells: the onset of thyroid autoimmunity?
  • Is PCOS an inflammatory process?
  • Mitochondrial hormesis in pancreatic β cells: does uncoupling protein 2 play a role?
  • Reactive oxygen species level in follicular fluid—embryo quality marker in IVF?
  • The “multiple hormone deficiency” theory of ageing: is human senescence caused mainly by multiple hormone deficiencies?
  • Thyroid iodide efflux: a team effort?
  • Update on the oxidative stress theory of aging: does oxidative stress play a role in aging or healthy aging?

Schema {🗺️}

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         description:A progressive decline in cellular function and organismal fitness and an increase in age-related diseases are associated with ageing; many theories have attempted to explain this phenomenon. This Review outlines the current knowledge of the effects of oxidative stress on endocrine organs and tissues and suggests that inflammation is the link between oxidative stress and ageing. Ageing is a process characterized by a progressive decline in cellular function, organismal fitness and increased risk of age-related diseases and death. Several hundred theories have attempted to explain this phenomenon. One of the most popular is the 'oxidative stress theory', originally termed the 'free radical theory'. The endocrine system seems to have a role in the modulation of oxidative stress; however, much less is known about the role that oxidative stress might have in the ageing of the endocrine system and the induction of age-related endocrine diseases. This Review outlines the interactions between hormones and oxidative metabolism and the potential effects of oxidative stress on ageing of endocrine organs. Many different mechanisms that link oxidative stress and ageing are discussed, all of which converge on the induction or regulation of inflammation. All these mechanisms, including cell senescence, mitochondrial dysfunction and microRNA dysregulation, as well as inflammation itself, could be targets of future studies aimed at clarifying the effects of oxidative stress on ageing of endocrine glands.
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