Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3454471/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3454471/. The analysis below is for the second page.

Title[redir]:
Reactive oxygen species (ROS) homeostasis and redox regulation in cellular signaling - PMC
Description:
Reactive oxygen species (ROS) are generated during mitochondrial oxidative metabolism as well as in cellular response to xenobiotics, cytokines, and bacterial invasion. Oxidative stress refers to the imbalance due to excess ROS or oxidants over the ...

Matching Content Categories {๐Ÿ“š}

  • Education
  • Science
  • Fitness & Wellness

Content Management System {๐Ÿ“}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of ncbi.nlm.nih.gov audience?

๐ŸŒ  Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 6,694,437 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {๐Ÿ’ธ}

We see no obvious way the site makes money.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.

Keywords {๐Ÿ”}

doi, pubmed, google, scholar, ros, free, pmc, article, oxidative, protein, redox, pshc, fig, biol, signaling, cell, cys, stress, irp, iron, ref, activation, regulation, atm, binding, domain, kinase, cellular, pathways, nrf, oxidation, response, activated, shown, transcription, dna, mol, chem, regulated, phosphorylation, activity, results, acid, mapk, factor, proteins, cluster, factors, conditions, formation,

Topics {โœ’๏ธ}

/v-h-c-x-x-g-x-x-r f-box/lrr-repeat protein 5 keap1/cullin-3 e3-ubiquitin ligase c-jun n-terminal kinases cullin-3 e3-ubiquitin ligase p66shc phospho-ser36-pro37 bond double-strand dna breaks basal c-terminal interaction extracellular signal-related kinases pmc beta search b-zip transcription factor platelet-derived growth factor mrn/ser-1981 autophosphorylation-independent manner primary post-transcriptional mechanism b-zip transcriptional repressor b-zip transcription factors iron-deficient conditions facilitate cgmp-dependent protein kinase iron-rich cells synthesize normal p66shc fibro-blasts mitogen-activated protein kinase 4feโ€“4s cluster implies iron-catalyzed oxidative stress phosphoinositide-dependent protein kinase n-terminus ch2 domain p66shc-deficient mouse fibroblasts hypoxia inducible factor-2ฮฑ ireโ€“irp regulatory system redox-dependent reversible tetramerization ironโ€“sulfur cluster proteins indirect ros-mediated damage iron-deficient cells produce family serine/threonine mapkkk apurinic/apyrimidinic endonuclease domain copper-dependent ros generation c-terminal region ros-mediated phosphorylation sites blocking n-terminal b-zip family ap-1/nfkb-independent manner ap-endonuclease activity domain increasing irpโ€“ire interaction stem-loop structure composed preventing fbxl5-dependent ubiquitination c-terminal domain human shca gene n-terminus region p66shc-deficient mice reactive oxygen species amino acid sequence

Questions {โ“}

  • What downstream signaling events are regulated by activated ATM in response to ROS?
  • What is the initial and direct impact of oxidative or genotoxic stress on p66Shc and how is p66Shc activated?
  • While there exists ample evidence demonstrating the role of ROS in regulating cellular signaling pathways, the question that is raised is exactly how do ROS initiate cellular signaling?

External Links {๐Ÿ”—}(307)

Analytics and Tracking {๐Ÿ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {โœ‰๏ธ}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {๐Ÿ“ฆ}

  • Ncbi

3.97s.