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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
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We began analyzing https://www.nature.com/articles/nature09782, but it redirected us to https://www.nature.com/articles/nature09782. The analysis below is for the second page.

Title[redir]:
Autophagy in immunity and inflammation | Nature
Description:
Autophagy is an essential, homeostatic process by which cells break down their own components. Perhaps the most primordial function of this lysosomal degradation pathway is adaptation to nutrient deprivation. However, in complex multicellular organisms, the core molecular machinery of autophagy — the

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Doi.org Make Money? {💸}

We don't see any clear sign of profit-making.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Doi.org could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {🔍}

pubmed, google, scholar, autophagy, cas, cell, central, nature, ads, biol, cells, immunity, levine, usa, article, proteins, host, inflammation, mizushima, infection, access, disease, atgl, study, essential, pathway, microbe, innate, atg, protein, proc, natl, acad, sci, viral, science, content, mammalian, immune, class, regulates, presentation, replication, intracellular, virus, cookies, machinery, responses, immunol, research,

Topics {✒️}

scientific research privacy policy nature portfolio mammalian autophagy research midwest regional center author information authors advertising permissions reprints social media ifnγ-inducible immunity-related gtpases genome-wide association defines genome-wide association scan nlrp1 inflammasome-linked depressive genome-wide association study autophagy gene-dependent clearance anti-acute peritonitis agent viral bcl-2-mediated evasion mammalian development ros-mediated autophagy inhibition scientific illustration autophagy-dependent viral recognition lc3-i-positive edemosomes nrf2-mediated autophagy pathway intracellular pathogen induced embryonic development autophagy-related pathways autophagy-related candidate genes autophagosome-independent essential function nature cell biol personal data atg5–atg12 conjugate associates flip-mediated autophagy regulation central nervous system genome-wide analysis springerlink instant access japan normal nutritional conditions signaling adapter p62 core molecular machinery data protection viral rna translation systemic lupus erythematosus permissions information human tectonin protein nature rev nature genet subscribe nature nature immunol cd4+ t-cell repertoire vesicular stomatitis virus

Questions {❓}

  • Antigen processing via autophagy—not only for MHC class II presentation anymore?

Schema {🗺️}

WebPage:
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         headline:Autophagy in immunity and inflammation
         description:Autophagy is an essential, homeostatic process by which cells break down their own components. Perhaps the most primordial function of this lysosomal degradation pathway is adaptation to nutrient deprivation. However, in complex multicellular organisms, the core molecular machinery of autophagy — the 'autophagy proteins' — orchestrates diverse aspects of cellular and organismal responses to other dangerous stimuli such as infection. Recent developments reveal a crucial role for the autophagy pathway and proteins in immunity and inflammation. They balance the beneficial and detrimental effects of immunity and inflammation, and thereby may protect against infectious, autoimmune and inflammatory diseases.
         datePublished:2011-01-20T00:00:00Z
         dateModified:2011-01-20T00:00:00Z
         pageStart:323
         pageEnd:335
         sameAs:https://doi.org/10.1038/nature09782
         keywords:
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            Immunological disorders
            Signal transduction
            Science
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      headline:Autophagy in immunity and inflammation
      description:Autophagy is an essential, homeostatic process by which cells break down their own components. Perhaps the most primordial function of this lysosomal degradation pathway is adaptation to nutrient deprivation. However, in complex multicellular organisms, the core molecular machinery of autophagy — the 'autophagy proteins' — orchestrates diverse aspects of cellular and organismal responses to other dangerous stimuli such as infection. Recent developments reveal a crucial role for the autophagy pathway and proteins in immunity and inflammation. They balance the beneficial and detrimental effects of immunity and inflammation, and thereby may protect against infectious, autoimmune and inflammatory diseases.
      datePublished:2011-01-20T00:00:00Z
      dateModified:2011-01-20T00:00:00Z
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                     name:Department of Microbiology, University of Texas Southwestern Medical Center, Dallas, USA
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         name:Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, USA
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         name:Department of Microbiology, University of Texas Southwestern Medical Center, Dallas, USA
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         name:Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, USA
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         name:Department of Physiology and Cell Biology, Tokyo Medical and Dental University, Tokyo, Japan
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            address:
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      name:Department of Physiology and Cell Biology, Tokyo Medical and Dental University, Tokyo, Japan
      name:Department of Pathology and Immunology, Washington University School of Medicine and Midwest Regional Center of Excellence for Biodefense and Emerging Infectious Diseases Research, Campus Box 8118, 660 South Euclid Avenue, Saint Louis, Missouri 63110, USA. [email protected],
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Social Networks {👍}(2)

External Links {🔗}(464)

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