NCBI . NLM . NIH . GOV {
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We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2860265/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC2860265/. The analysis below is for the second page.
Title[redir]:
Autophagy Protects against Sindbis Virus Infection of the Central Nervous System - PMC
Description:
Autophagy functions in antiviral immunity. However, it is not yet known whether endogenous autophagy genes protect against viral disease in vertebrates. Using three different approaches to inactivate the autophagy gene Atg5 in virally-infected ...
Title[redir]:
Autophagy Protects against Sindbis Virus Infection of the Central Nervous System - PMC
Description:
Autophagy functions in antiviral immunity. However, it is not yet known whether endogenous autophagy genes protect against viral disease in vertebrates. Using three different approaches to inactivate the autophagy gene Atg5 in virally-infected ...
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Keywords {🔍}
atg, sin, mice, cell, cells, autophagy, doi, viral, infection, neuronal, capsid, protein, fig, mefs, virus, infected, increased, replication, pubmed, google, scholar, gfplc, death, neurons, function, mouse, data, autophagic, observed, results, control, pmc, clearance, studies, atgfloxflox, vivo, shown, sirna, levels, vitro, role, mizushima, experimental, cns, proteins, levine, cellular, mortality, svia, sindbis,
Topics {✒️}
hela/gfp-lc3 cells mock-infected tetracycline-repressible atg5-expressing plasmid hela/gpf-lc3 cells treated atg5-atg12-atg16 conjugation system hela/gfp-lc3 cells treated sin/atg5k130r versus sin/atg5 polyclonal anti-sin antibody anti-sin polyclonal antibody sin/cre-infected atg5flox/flox mice cns-specific atg5-deficient mice svia-infected gfp-lc3 mefs pmc beta search stably-expressing gfp-lc3 retinoic acid-inducible gene calpain-mediated cleavage fragment huntingtin-induced cell death c-terminal uba domain gfp-lc3-positive membranes anti-sin polyclonal antibodies older neuron-specific atg5 cd4+ t-cell responses studying virus-host interactions double membrane-bound vesicles hela/gfp-lc3 cells virus-induced cell death mosquito-borne arboviral infections atg5-deficient cell lines gfp-lc3 punctate structures sin-induced cell death cell-autonomous defense system cell type-dependent manner autophagy-dependent viral recognition atg5−/− mefs stably-transfected detect gfp-lc3 punctae undergo neuron-specific deletion viral antigen-positive cells specific gfp-lc3 aggregates virally-infected hela cells sin antigen-positive cells anti-sin virus capsid exhibits age-dependent neurovirulence neuronal atg5-dependent control primary atg5flox/flox mefs p62-mediated autophagic clearance contained sindbis virions detergent-insoluble protein fraction viral antigen-positive neurons virally-infected neurons exclusively bcl-2-interacting protein increased sin-induced mortality
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