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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://link.springer.com/article/10.1007/s40139-017-0139-5, but it redirected us to https://link.springer.com/article/10.1007/s40139-017-0139-5. The analysis below is for the second page.

Title[redir]:
Autophagy and Ferroptosis—What Is the Connection? | Current Pathobiology Reports
Description:
Purpose of Review Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferroptosis and discuss their crosstalk mechanisms at the molecular level. Recent Findings The original study shows that ferroptosis is morphologically, biochemically, and genetically distinct from autophagy and other types of cell death. However, recent studies demonstrate that activation of ferroptosis is indeed dependent on the induction of autophagy. Additionally, many ferroptosis regulators such as SLC7A11, GPX4, NRF2, p53, HSPB1, CISD1, FANCD2, and ACSL4 have been identified as potential regulators of autophagy. Summary This review not only highlights the importance of autophagy as an emerging mechanism of ferroptosis but also raises new insights regarding regulated cell death.

Matching Content Categories {📚}

  • Education
  • Telecommunications
  • Science

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 80,486,609 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We're unsure how the site profits.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

pubmed, article, scholar, google, cas, cell, ferroptosis, autophagy, central, death, importance, cancer, study, regulation, demonstrated, kang, cells, tang, biol, nat, sun, lipid, acsl, xie, journal, daolin, regulated, protein, song, res, yang, chem, chen, privacy, cookies, function, content, peroxidation, molecular, gpx, access, glutathione, differ, doicdd, rev, skouta, doijcell, lotze, doi, biochem,

Topics {✒️}

oncogenic-ras-harboring cancer cells p62-keap1-nrf2 pathway protects month download article/chapter iron-dependent cell death acyl-coa synthetase-4 research scholar grant daolin tang declare mitochondrial lipid peroxidation bravo-san pedro jm p53-mediated ferroptotic responses genotype-selective antitumor agents cell signal 28 full article pdf iron-dependent form daolin tang lipid peroxidation molecular life sciences adaptive stress responses privacy choices/manage cookies dixon sj autophagy-related proteins author information authors integrated strategy reveals bone marrow injury animal subjects performed related subjects ros-induced autophagy p53-mediated activity autophagic cell death negative breast cancer check access instant access autophagy promotes ferroptosis pancreatic cancer cells cancer cells assists hepatocellular carcinoma cells cell death subroutines nonapoptotic cell death immunogenic cell death pathobiology rights cancer targets bcl-2 american cancer society regulated cell death p53-mediated ferroptosis article kang angeli jp article log yang ws european economic area maintaining mitochondrial homeostasis

Questions {❓}

  • Autophagy and Ferroptosis—What Is the Connection?
  • Autophagy and Ferroptosis—What Is the Connection?
  • Cox AD, Fesik SW, Kimmelman AC, Luo J, Der CJ (2014) Drugging the undruggable RAS: mission possible?
  • Green DR, Levine B (2014) To be or not to be?

Schema {🗺️}

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         headline:Autophagy and Ferroptosis—What Is the Connection?
         description:Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferroptosis and discuss their crosstalk mechanisms at the molecular level. The original study shows that ferroptosis is morphologically, biochemically, and genetically distinct from autophagy and other types of cell death. However, recent studies demonstrate that activation of ferroptosis is indeed dependent on the induction of autophagy. Additionally, many ferroptosis regulators such as SLC7A11, GPX4, NRF2, p53, HSPB1, CISD1, FANCD2, and ACSL4 have been identified as potential regulators of autophagy. This review not only highlights the importance of autophagy as an emerging mechanism of ferroptosis but also raises new insights regarding regulated cell death.
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      headline:Autophagy and Ferroptosis—What Is the Connection?
      description:Autophagy is a conserved intracellular degradation system and plays a dual role in cell death, depending on context and phase. Ferroptosis is a new form of regulated cell death that mainly depends on iron accumulation and lipid peroxidation. In this review, we summarize the processes of autophagy and ferroptosis and discuss their crosstalk mechanisms at the molecular level. The original study shows that ferroptosis is morphologically, biochemically, and genetically distinct from autophagy and other types of cell death. However, recent studies demonstrate that activation of ferroptosis is indeed dependent on the induction of autophagy. Additionally, many ferroptosis regulators such as SLC7A11, GPX4, NRF2, p53, HSPB1, CISD1, FANCD2, and ACSL4 have been identified as potential regulators of autophagy. This review not only highlights the importance of autophagy as an emerging mechanism of ferroptosis but also raises new insights regarding regulated cell death.
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External Links {🔗}(282)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • Prism.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Crossref

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