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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://link.springer.com/article/10.1007/s00415-013-7169-7, but it redirected us to https://link.springer.com/article/10.1007/s00415-013-7169-7. The analysis below is for the second page.

Title[redir]:
Update on the diagnosis and treatment of neuromyelitis optica: Recommendations of the Neuromyelitis Optica Study Group (NEMOS) | Journal of Neurology
Description:
Neuromyelitis optica (NMO, Devic’s syndrome), long considered a clinical variant of multiple sclerosis, is now regarded as a distinct disease entity. Major progress has been made in the diagnosis and treatment of NMO since aquaporin-4 antibodies (AQP4-Ab; also termed NMO-IgG) were first described in 2004. In this review, the Neuromyelitis Optica Study Group (NEMOS) summarizes recently obtained knowledge on NMO and highlights new developments in its diagnosis and treatment, based on current guidelines, the published literature and expert discussion at regular NEMOS meetings. Testing of AQP4-Ab is essential and is the most important test in the diagnostic work-up of suspected NMO, and helps to distinguish NMO from other autoimmune diseases. Furthermore, AQP4-Ab testing has expanded our knowledge of the clinical presentation of NMO spectrum disorders (NMOSD). In addition, imaging techniques, particularly magnetic resonance imaging of the brain and spinal cord, are obligatory in the diagnostic workup. It is important to note that brain lesions in NMO and NMOSD are not uncommon, do not rule out the diagnosis, and show characteristic patterns. Other imaging modalities such as optical coherence tomography are proposed as useful tools in the assessment of retinal damage. Therapy of NMO should be initiated early. Azathioprine and rituximab are suggested as first-line treatments, the latter being increasingly regarded as an established therapy with long-term efficacy and an acceptable safety profile in NMO patients. Other immunosuppressive drugs, such as methotrexate, mycophenolate mofetil and mitoxantrone, are recommended as second-line treatments. Promising new therapies are emerging in the form of anti-IL6 receptor, anti-complement or anti-AQP4-Ab biologicals.

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 8,426,036 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

The income method remains a mystery to us.

Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Doi.org could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {🔍}

pubmed, google, scholar, article, neuromyelitis, optica, cas, nmo, patients, neurol, treatment, central, neurology, disease, jarius, aquaporin, therapy, mult, scler, antibodies, clinical, multiple, diagnosis, sclerosis, study, spectrum, universität, wildemann, brain, lesions, kim, antibody, case, weinshenker, arch, aqpab, paul, studies, relapse, patient, mri, cases, diagnostic, acute, lennon, pittock, nmoigg, disorders, imaging, criteria,

Topics {✒️}

nmo-igg/aquaporin-4-antibody-positive neuromyelitis optica 1 mg/kg body weight/day enzymatic aqp4-igg deglycosylation t-cell-depleting antibody previously article download pdf /kg body weight/day central nervous system nmo-igg-positive patient collective aqp4-ab-positive classical nmo open-label pilot study steroid-responsive hearing impairment enzyme-linked immunosorbent assay white matter lesions myelin-oligodendrocyte glycoprotein antibodies steroid-unresponsive optic neuritis aqp4-ab remain detectable anti-human aquaporin-4 antibody pencil-thin ependymal enhancement anti-ds-dna antibodies anti-aqp4-ab biologicals �nmo-spectrum disorder”—abortive anti-ssa antibody detection anti-aquaporin-4 antibodies measured exceed 100 mg/m2 bsa true-positive test results magnetic resonance imaging apq4-ab-negative nmo patients apq4-ab-positive nmo open-access network optical coherence tomography apq4-ab-positive patients therapy-related acute leukaemia full-length human aqp4 called cell-based assays aquaporin-4 antibody-positive patients pathogenic aqp4-specific antibodies comparative cross-sectional study krankenhaus martha-maria halle aqp4-ab-positive hrs intermittent long-term therapy myelin oligodendrocyte glycoprotein aqp4-ab serum levels aqp4-igg-positive nmo aqp4-ab-positive nmosd igm csf/serum ratios aqp4-ab-positive nmo aqp4-ab-positive nmo [1 interferon beta-1b treatment heinrich-heine-universität düsseldorf related subjects

Questions {❓}

  • Cosgrove J, Alli S, Ramadan H, Ford HL (2013) Myocarditis and diffuse skeletal muscle oedema: new features of neuromyelitis optica spectrum disorder?
  • Higgs JE, Payne K, Roberts C, Newman WG (2010) Are patients with intermediate TPMT activity at increased risk of myelosuppression when taking thiopurine medications?
  • Jacob A, Hutchinson M, Elsone L, Kelly S, Ali R, Saukans I, Tubridy N, Boggild M (2012) Does natalizumab therapy worsen neuromyelitis optica?
  • Kim S-H, Kim W, Li XF, Jung I-J, Kim HJ (2012) Does interferon beta treatment exacerbate neuromyelitis optica spectrum disorder?
  • Vaknin-Dembinsky A, Abramsky O, Petrou P, Ben-Hur T, Gotkine M, Brill L, Brenner T, Argov Z, Karussis D (2011) Myasthenia gravis-associated neuromyelitis optica-like disease: an immunological link between the central nervous system and muscle?

Schema {🗺️}

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         headline:Update on the diagnosis and treatment of neuromyelitis optica: Recommendations of the Neuromyelitis Optica Study Group (NEMOS)
         description: Neuromyelitis optica (NMO, Devic’s syndrome), long considered a clinical variant of multiple sclerosis, is now regarded as a distinct disease entity. Major progress has been made in the diagnosis and treatment of NMO since aquaporin-4 antibodies (AQP4-Ab; also termed NMO-IgG) were first described in 2004. In this review, the Neuromyelitis Optica Study Group (NEMOS) summarizes recently obtained knowledge on NMO and highlights new developments in its diagnosis and treatment, based on current guidelines, the published literature and expert discussion at regular NEMOS meetings. Testing of AQP4-Ab is essential and is the most important test in the diagnostic work-up of suspected NMO, and helps to distinguish NMO from other autoimmune diseases. Furthermore, AQP4-Ab testing has expanded our knowledge of the clinical presentation of NMO spectrum disorders (NMOSD). In addition, imaging techniques, particularly magnetic resonance imaging of the brain and spinal cord, are obligatory in the diagnostic workup. It is important to note that brain lesions in NMO and NMOSD are not uncommon, do not rule out the diagnosis, and show characteristic patterns. Other imaging modalities such as optical coherence tomography are proposed as useful tools in the assessment of retinal damage. Therapy of NMO should be initiated early. Azathioprine and rituximab are suggested as first-line treatments, the latter being increasingly regarded as an established therapy with long-term efficacy and an acceptable safety profile in NMO patients. Other immunosuppressive drugs, such as methotrexate, mycophenolate mofetil and mitoxantrone, are recommended as second-line treatments. Promising new therapies are emerging in the form of anti-IL6 receptor, anti-complement or anti-AQP4-Ab biologicals.
         datePublished:2013-11-23T00:00:00Z
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      headline:Update on the diagnosis and treatment of neuromyelitis optica: Recommendations of the Neuromyelitis Optica Study Group (NEMOS)
      description: Neuromyelitis optica (NMO, Devic’s syndrome), long considered a clinical variant of multiple sclerosis, is now regarded as a distinct disease entity. Major progress has been made in the diagnosis and treatment of NMO since aquaporin-4 antibodies (AQP4-Ab; also termed NMO-IgG) were first described in 2004. In this review, the Neuromyelitis Optica Study Group (NEMOS) summarizes recently obtained knowledge on NMO and highlights new developments in its diagnosis and treatment, based on current guidelines, the published literature and expert discussion at regular NEMOS meetings. Testing of AQP4-Ab is essential and is the most important test in the diagnostic work-up of suspected NMO, and helps to distinguish NMO from other autoimmune diseases. Furthermore, AQP4-Ab testing has expanded our knowledge of the clinical presentation of NMO spectrum disorders (NMOSD). In addition, imaging techniques, particularly magnetic resonance imaging of the brain and spinal cord, are obligatory in the diagnostic workup. It is important to note that brain lesions in NMO and NMOSD are not uncommon, do not rule out the diagnosis, and show characteristic patterns. Other imaging modalities such as optical coherence tomography are proposed as useful tools in the assessment of retinal damage. Therapy of NMO should be initiated early. Azathioprine and rituximab are suggested as first-line treatments, the latter being increasingly regarded as an established therapy with long-term efficacy and an acceptable safety profile in NMO patients. Other immunosuppressive drugs, such as methotrexate, mycophenolate mofetil and mitoxantrone, are recommended as second-line treatments. Promising new therapies are emerging in the form of anti-IL6 receptor, anti-complement or anti-AQP4-Ab biologicals.
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         Differential diagnosis
         Diagnostic tests
         Therapy
         Neurology
         Neurosciences
         Neuroradiology
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      name:Orhan Aktas
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      name:Department of Neurology, Hannover Medical School, Hannover, Germany
      name:Division of Molecular Neuroimmunology, Department of Neurology, University of Heidelberg, Heidelberg, Germany
      name:Department of Neurology, Klinikum rechts der Isar, Technische Universität München, Munich, Germany
      name:Clinical and Experimental Multiple Sclerosis Research Center, Department of Neurology, Charité-Universitätsmedizin Berlin, Berlin, Germany
      name:NeuroCure Clinical Research Center and Experimental and Clinical Research Center, Charité-Universitätsmedizin Berlin and Max Delbrück Center for Molecular Medicine, Berlin, Germany
      name:Neuroimmunology and Multiple Sclerosis Research Section, Department of Neurology, University Hospital Zürich, Zürich, Switzerland
      name:Division of Molecular Neuroimmunology, Department of Neurology, University of Heidelberg, Heidelberg, Germany
      name:NeuroCure Clinical Research Center and Experimental and Clinical Research Center, Charité-Universitätsmedizin Berlin and Max Delbrück Center for Molecular Medicine, Berlin, Germany
      name:Department of Neurology, St. Josef-Hospital, Ruhr-Universität, Bochum, Germany
      name:Department of Neurology, Medical Faculty, Heinrich-Heine-Universität Düsseldorf, Düsseldorf, Germany
      name:Institute of Clinical Neuroimmunology, Medical Campus Grosshadern, Ludwig-Maximilians-Universität, Munich, Germany

External Links {🔗}(934)

Analytics and Tracking {📊}

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Libraries {📚}

  • Clipboard.js
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Emails and Hosting {✉️}

Mail Servers:

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Name Servers:

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CDN Services {📦}

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