DOI . ORG {
}
We began analyzing https://link.springer.com/chapter/10.1007/978-3-030-62026-4_7, but it redirected us to https://link.springer.com/chapter/10.1007/978-3-030-62026-4_7. The analysis below is for the second page.
Title[redir]:
Necroptosis , the Other Main Caspase-Independent Cell Death | SpringerLink
Description:
The past decades witnessed the discovery of novel modes of cell death, such as ferroptosis, pyroptosis and necroptosis, all of them presenting common necrotic traits. In this chapter, we revisit the early discoveries that unveiled necroptosis as a distinct cell death...
Title[redir]:
Necroptosis , the Other Main Caspase-Independent Cell Death | SpringerLink
Description:
The past decades witnessed the discovery of novel modes of cell death, such as ferroptosis, pyroptosis and necroptosis, all of them presenting common necrotic traits. In this chapter, we revisit the early discoveries that unveiled necroptosis as a distinct cell death...
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Keywords {🔍}
pubmed, article, google, scholar, cas, cell, necroptosis, central, death, protein, rip, cancer, mlkl, necrosis, kinase, ferroptosis, biol, ripk, wang, cells, zhang, sci, httpsdoiorgs, chen, apoptosis, mol, nat, receptor, domain, chapter, disease, virus, immunity, liu, programmed, signaling, necrotic, immunol, expression, httpsdoiorgjcell, mediates, chem, proc, natl, acad, httpsdoiorgpnas, cellular, molecular, activation, inflammation,
Topics {✒️}
nuclear factor kappa-light-chain-enhancer nf-kappab-independent cell-death switch receptor-interacting kinase-3-mediated pathway alix-syntenin-1-mediated exocytosis protect caspase-mediated p65/rela cleavage month download article/chapter ch'en il caspase-independent apoptosis pathway radiation-induced mutator phenotype escrt-iii acts downstream cell death-independent neuroinflammation tnfα-induced necroptotic signaling ubiquitylation-dependent proteasomal degradation death-receptor-induced apoptosis cellular flice-inhibitory protein dai/zbp1/dlm-1 complexes rip3-induced programmed necrosis receptor-interacting protein 1 z-dna binding domain fas-induced dna fragmentation murine cytomegalovirus vira phosphorylation-driven assembly receptor 3-mediated necrosis anti-viral inflammation heighten intestinal inflammation rip1/rip3 necrosome forms necroptotic cell death apoptosis-inducing kinase dysfunctional hiv-specific cd8+ tumor necrosis factor virus-induced inflammation necrotic cell death privacy choices/manage cookies cochaperone cdc37 complex thapa rj chronic lymphocytic leukemia tnf-induced nf petrie ej device instant download hydrogen peroxide mediates regulates endosomal trafficking blunt mechanical impact nf-kappab signaling dna-dependent activator caspase-8-deficient mice ripk1-mediated neuroinflammation upton jw flotillin-mediated endocytosis pattern recognition receptors helix bundle domain
Questions {❓}
- Kelliher MA, Grimm S, Ishida Y, Kuo F, Stanger BZ, Leder P (1998) The death domain kinase RIP mediates the TNF-induced NF-?
- Marshall KD, Baines CP (2014) Necroptosis: is there a role for mitochondria?
- Wang T, Jin Y, Yang W, Zhang L, Jin X, Liu X et al (2017) Necroptosis in cancer: an angel or a demon?
Schema {🗺️}
ScholarlyArticle:
headline:Necroptosis , the Other Main Caspase-Independent Cell Death
pageEnd:138
pageStart:123
image:https://media.springernature.com/w153/springer-static/cover/book/978-3-030-62026-4.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:Ferroptosis: Mechanism and Diseases
isbn:
978-3-030-62026-4
978-3-030-62025-7
type:Book
publisher:
name:Springer International Publishing
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:Larissa C. Zanetti
affiliation:
name:Hospital Israelita Albert Einstein. Av. Albert Einstein
address:
name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
type:PostalAddress
type:Organization
email:[email protected]
type:Person
name:Ricardo Weinlich
affiliation:
name:Hospital Israelita Albert Einstein. Av. Albert Einstein
address:
name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
type:PostalAddress
type:Organization
type:Person
keywords:Necroptosis, RIPK3, MLKL, RIPK1, Inflammation, TNF
description:The past decades witnessed the discovery of novel modes of cell death, such as ferroptosis, pyroptosis and necroptosis, all of them presenting common necrotic traits. In this chapter, we revisit the early discoveries that unveiled necroptosis as a distinct cell death mechanism. We describe necroptosis, its main regulators and their role in maintaining cellular homeostasis and in the disease state. We conclude by discussing its phenotypic similarities with ferroptosis and the possible crosstalk between these pathways.
datePublished:2021
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address:
name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
type:PostalAddress
type:Organization
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