Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC8346021/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC8346021/. The analysis below is for the second page.

Title[redir]:
Untangling the Cooperative Role of Nuclear Receptors in Cardiovascular Physiology and Disease - PMC
Description:
The heart is the first organ to acquire its physiological function during development, enabling it to supply the organism with oxygen and nutrients. Given this early commitment, cardiomyocytes were traditionally considered transcriptionally stable ...

Matching Content Categories {📚}

  • Health & Fitness
  • Education
  • Fitness & Wellness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,005 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We're unsure how the site profits.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Ncbi.nlm.nih.gov has a revenue plan, but it's either invisible or we haven't found it.

Keywords {🔍}

doi, pubmed, google, scholar, cardiac, pmc, article, free, receptor, heart, expression, mice, vitamin, receptors, signaling, cell, myocardial, acid, hypertrophy, role, pparα, activation, rxr, retinoid, cardiomyocytes, thyroid, nuclear, function, cardiomyocyte, development, atherosclerosis, peroxisome, cells, proliferatoractivated, retinoic, effects, hormone, transcriptional, homeostasis, genes, stress, injury, mol, increased, liver, rats, rat, res, zhang, biol,

Topics {✒️}

int/news-room/fact-sheets/detail/cardiovascular-diseases pparγ/pgc-1α/tnf-α signaling pathway peroxisome proliferator-activated receptor-alpha c-reactive protein-induced pro-inflammation peroxisome proliferator-activated receptor peroxisome proliferator-activated receptors long-term post-myocardial infarction camkii/nf-κb/tgf-β1 regulating pten/akt/fak pathway high-fat/high-cholesterol diet increased heart-weight/body-weight ratio endocardium-specific rxrα-ko mice mitogen-activated protein kinase liver-x-receptor β protects ldl receptor-deficient mice cardiac-specific rxrα-ko mice long-chain fatty acid pmc beta search epicardium-specific rxrα-ko mice lkb1-dependent anti-fibrosis effects 3-hydroxy-3-methylglutaryl-coa synthase 2 pparγ-dependent anti-inflammatory mechanisms cyclic-amp-dependent pathways ppar-γ signaling pathway crp-induced nf-kb atra-catabolizing enzyme cyp26 double rxrαβ-ko mice transforming growth factor-beta cardiac renin-angiotensin systems nf-kb pathway repression high glucose-induced repression ventricular muscle-restricted targeting systemic rxrαβ-ko embryos inhibit monocyte/macrophage recruitment long-chain polyunsaturated fa long-chain fa c24 anti-inflammatory mechanisms activated 9-cis-retinoic acid receptor isoproterenol-induced cardiac hypertrophy 1alpha-hydroxylase knockout mice renin-angiotensin-aldosterone system cardiac malonyl-coa levels c-jun/ap-1 signaling [81] ]pyridine-6-carboxylic acid derivatives 1α-hydroxylase ko mice suppressing fundc1-mediated mitophagy camp response element-binding half-sites forming direct prevents phenylephrine-induced hypertrophy liver-x-receptor α

Questions {❓}

  • Circulating MMP9, vitamin D and variation in the TIMP-1 response with VDR genotype: Mechanisms for inflammatory damage in chronic disorders?

External Links {🔗}(559)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Foundation
  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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