Here's how NCBI.NLM.NIH.GOV makes money* and how much!

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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5829048/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC5829048/. The analysis below is for the second page.

Title[redir]:
Blood–brain barrier breakdown in Alzheimer’s disease and other neurodegenerative disorders - PMC
Description:
The blood–brain barrier (BBB) is a continuous endothelial membrane within brain microvessels that has sealed cell-to-cell contacts, and is sheathed by mural vascular cells and perivascular astrocyte end-feet. The BBB protects neurons from factors ...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We don't see any clear sign of profit-making.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Ncbi.nlm.nih.gov could have a money-making trick up its sleeve, but it's undetectable for now.

Keywords {🔍}

doi, google, scholar, pubmed, brain, bbb, disease, pmc, free, article, barrier, alzheimers, patients, bloodbrain, blood, vascular, breakdown, increased, flow, studies, transport, cells, cerebral, neurodegenerative, endothelial, neurol, dementia, glucose, csf, reduced, proteins, degeneration, microbleeds, neurodegeneration, mri, med, disorders, tight, levels, perivascular, cognitive, nat, multiple, dysfunction, risk, pericyte, cereb, clearance, metab, factors,

Topics {✒️}

[14c]2-deoxy-d-glucose-6-phosphate luminal sodium-potassium-chloride cotransporter [14c]2-deoxy-d-glucose ldl receptor-related protein-1 high-resolution confocal microscopy pmc beta search bone marrow-derived macrophages232 pericyte-derived soluble factors amyloid-β peptide protects apoeε4 transgenic mice71 transgenic apoeε4 mice71 enzyme-linked immunosorbent assay allan–herndon–dudley syndrome allan-herndon-dudley syndrome fluid-attenuated inversion recovery impaired p-glycoprotein function pressure-driven fluid flow blood-brain barrier permeability microbleed t2-weighted mri87 late-middle-aged persons altered p-glycoprotein expression autosomal-dominant neurodegenerative disease 3t susceptibility-weighted imaging pericapillary haem-rich deposits small lipid-soluble molecules removes toxic amyloid-β blood-brain barrier disturbance anatomical blood-brain barrier leaky blood-brain barrier plasma amyloid-beta efflux perform amyloid-pet imaging86–93 astrocyte end-feet wrapping apoe isoform-specific disruption gov/ct2/show/nct02008357 gov/ct2/show/nct02916056 p-glycoprotein clears aβ symptomatic apoeε4 carriers diminished p-glycoprotein activity solute carrier-mediated transport amyloid-beta peptide neurotoxicity dynamic contrast-enhanced mri neurotoxic blood-derived products reduced p-glycoprotein expression neurotoxic blood-derived debris deep-brain stimulation associates bbb-specific glucose transporter109–112 blood–brain barrier disruption blood-brain barrier disruption rhoa-rock signaling pathway blood-brain barrier function

Questions {❓}

  • Alzheimer’s disease: a matter of blood-brain barrier dysfunction?
  • Cerebral microbleeds as a biomarker in Alzheimer’s disease?
  • If CNS vascular changes drive the initial pathogenic events that contribute to onset and progression of neurodegeneration, loss of brain connectivity, and neuronal injury and loss in complex neurodegenerative disorders, as they do in human monogenic neurological BBB diseases6, the question persists will therapeutic targeting of BBB arrest and reverse the course of neurological disorder in humans in a way as shown in some animal models71,73,192,196,199?
  • Transmission of α-synuclein-containing erythrocyte-derived extracellular vesicles across the blood-brain barrier via adsorptive mediated transcytosis: another mechanism for initiation and progression of Parkinson’s disease?

Social Networks {👍}(6)

External Links {🔗}(516)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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