Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3427235/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3427235/. The analysis below is for the second page.

Title[redir]:
The role of the myofibroblast in tumor stroma remodeling - PMC
Description:
Since its first description in wound granulation tissue, the myofibroblast has been recognized to be a key actor in the epithelial-mesenchymal cross-talk that plays a crucial role in many physiological and pathological situations, such as regulation ...

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't figure out the monetization strategy.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

doi, google, scholar, pubmed, cells, cell, tumor, cancer, pmc, free, article, stroma, tissue, myofibroblasts, tgfβ, ecm, growth, myofibroblast, biol, activation, fibrosis, stromal, wound, human, stem, carcinoma, fibroblasts, liver, matrix, epithelial, hepatocellular, role, mesenchymal, latent, expression, factor, development, normal, healing, rev, res, progression, αsma, mechanical, collagen, cholangiocarcinoma, remodeling, bone, tgfbeta, differentiation,

Topics {✒️}

platelet-derived growth factor alpha-smooth muscle actin α-smooth muscle actin androgen-receptor-positive wild-type mesenchyme transforming growth factor-beta transforming growth factor-β1 envisaged msc therapy platelet-derived microparticles matrix metalloproteinase type-3 epidermal growth factor small latent complex exploring anti-tgf-β therapies tgf-β-mediated biochemical signals hepatocyte growth factor stromal cell-derived factor-1 increased metalloproteinase expression free-floating relaxed gels thioacetamide-induced intestinal-type cholangiocarcinoma matricellular protein ccn2 tumor necrosis factor latent tgf-beta1 activation tgfβ fibroblast growth factor bone marrow-derived cells reduced growth factor latent tgf-β1 complex lysyl oxidases bone marrow-derived stroma hsc-derived myofibroblasts pmc beta search latent tgf-beta activation dermal-epidermal junction result pro-tumorigenic factor tgfβ1 α-sma expressing cells smooth muscle α-sm actin bone-marrow-derived myofibroblasts numerous α-sma expressing protease-independent tgfβ1-activating integrins activate latent tgf-β cells secrete slc α-sma positive myofibroblasts α-sma-positive myofibroblasts skin flap-induced regression mt1-mmp-dependent activation small-molecule inhibitors fibrillin protein family latent tgf-β structure mediate epithelial-mesenchymal interactions fibroblast-led collective invasion

Questions {❓}

  • Epithelial-mesenchymal transition in chronic liver disease: fibrogenesis or escape from death?
  • Epithelial-to-mesenchymal transition in liver fibrosis: dead or alive?
  • Hepatic stellate cells: the only cells involved in liver fibrogenesis?
  • Matrix elasticity, cytoskeletal forces and physics of the nucleus: how deeply do cells ‘feel’ outside and in?
  • Origins and roles of cells in the stroma of normal organs: all fibroblasts?
  • Transforming growth factor beta: tumor suppressor or promoter?
  • Why are MSCs therapeutic?
  • Why don’t we get more cancer?

Social Networks {👍}(6)

External Links {🔗}(568)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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