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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
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We began analyzing https://www.tandfonline.com/doi/abs/10.4161/cam.20377, but it redirected us to https://www.tandfonline.com/doi/abs/10.4161/cam.20377. The analysis below is for the second page.

Title[redir]:
Full article: The role of the myofibroblast in tumor stroma remodeling
Description:
Tissue-resident stromal cells, often loosely defined under the term “fibroblast,” form the extracellular matrix (ECM) supportive scaffold of connective tissues during development and maintain the s...

Matching Content Categories {📚}

  • Dating & Relationships
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  • Humor

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Doi.org Make Money? {💸}

We're unsure how the site profits.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Doi.org might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

open, window, citation, google, scholar, pmid, pubmed, science, web, cells, cell, tumor, cancer, stroma, tissue, myofibroblasts, tgfβ, ecm, growth, myofibroblast, biol, activation, fibrosis, stromal, human, wound, carcinoma, fibroblasts, stem, liver, epithelial, matrix, hepatocellular, role, mesenchymal, latent, expression, factor, development, normal, rev, res, healing, progression, αsma, mechanical, collagen, cholangiocarcinoma, remodeling, bone,

Topics {✒️}

androgen-receptor-positive wild-type mesenchyme exploring anti-tgf-β therapies alpha-smooth muscle actin tgf-β-mediated biochemical signals medical sciences transforming growth factor-beta transforming growth factor-β1 thioacetamide-induced intestinal-type cholangiocarcinoma α-smooth muscle actin platelet-derived growth factor references tomasek jj latent tgf-beta1 activation protease-independent tgfβ1-activating integrins peripheral bile-duct carcinoma display full size skin flap-induced regression free-floating relaxed gels stromal cell-derived factor-1 bone marrow-derived cells neural crest-related precursors photon laser-generated microtracks alcohol-induced fatty liver extra-hepatic bile ducts bone marrow-derived stroma α-sma expressing cells increased apoptotic figures activate latent tgf-β numerous α-sma expressing google scholar paquet-fifield google scholar dore-duffy google scholar piera-velazquez google scholar andersson-sjöland google scholar barisic-dujmovic google scholar ruiz-ortega fibroblast-led collective invasion bone-marrow-derived myofibroblasts multipotent skin-derived precursors latent tgf-beta activation mediate epithelial-mesenchymal interactions α-sma positive myofibroblasts α-sma-positive myofibroblasts pro-tumorigenic factor tgfβ1 androgen-receptor-negative epithelium latent tgf-β1 complex cell-basement membrane junctions cross-linked fibrin ecm dermal-epidermal junction result latent tgf-β structure autocrine feed-forward loop pro-inflammatory growth factors

Questions {❓}

  • Epithelial-mesenchymal transition in chronic liver disease: fibrogenesis or escape from death?
  • Epithelial-to-mesenchymal transition in liver fibrosis: dead or alive?
  • Hepatic stellate cells: the only cells involved in liver fibrogenesis?
  • Matrix elasticity, cytoskeletal forces and physics of the nucleus: how deeply do cells ‘feel’ outside and in?
  • Origins and roles of cells in the stroma of normal organs: all fibroblasts?
  • Transforming growth factor beta: tumor suppressor or promoter?
  • Why are MSCs therapeutic?
  • Why don’t we get more cancer?
  • Why publish with us?

Schema {🗺️}

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            name:The role of the myofibroblast in tumor s ....
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      mainEntityOfPage:https://www.tandfonline.com/doi/full/10.4161/cam.20377
      url:https://www.tandfonline.com/doi/full/10.4161/cam.20377
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      articleSection:Review
      name:The role of the myofibroblast in tumor stroma remodeling
      headline:The role of the myofibroblast in tumor stroma remodeling
      abstract:Since its first description in wound granulation tissue, the myofibroblast has been recognized to be a key actor in the epithelial-mesenchymal cross-talk that plays a crucial role in many physiological and pathological situations, such as regulation of prostate development, ventilation-perfusion in lung alveoli or organ fibrosis. The presence of myofibroblasts in the stroma reaction to epithelial tumors is well established and many data are accumulating which suggest that the stroma compartment is an active participant in tumor onset and/or evolution. In this review we summarize the evidence in favor of this concept, the main mechanisms that regulate myofibroblast differentiation and function, as well as the biophysical and biochemical factors possibly involved in epithelial-stroma interactions, using liver carcinoma as main model, in view of achieving a better understanding of tumor progression mechanisms and of tools directed toward stroma as eventual therapeutic target.
      description:Since its first description in wound granulation tissue, the myofibroblast has been recognized to be a key actor in the epithelial-mesenchymal cross-talk that plays a crucial role in many physiological and pathological situations, such as regulation of prostate development, ventilation-perfusion in lung alveoli or organ fibrosis. The presence of myofibroblasts in the stroma reaction to epithelial tumors is well established and many data are accumulating which suggest that the stroma compartment is an active participant in tumor onset and/or evolution. In this review we summarize the evidence in favor of this concept, the main mechanisms that regulate myofibroblast differentiation and function, as well as the biophysical and biochemical factors possibly involved in epithelial-stroma interactions, using liver carcinoma as main model, in view of achieving a better understanding of tumor progression mechanisms and of tools directed toward stroma as eventual therapeutic target.
      author:
            type:Person
            name:Giulio Gabbiani
            type:Person
            name:Vincent Sarrazy
            type:Person
            name:Boris Hinz
            type:Person
            name:Marcela Otranto
            type:Person
            name:Frédéric Bonté
            type:Person
            name:Alexis Desmouliere
            type:Person
            name:Alexis Desmouliere
      keywords:mechanosensing, extracellular matrix, transforming growth factor-beta, wound healing, fibrosis
      pageStart:203
      pageEnd:219
      datePublished:2012-06-01
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      name:Boris Hinz
      name:Marcela Otranto
      name:Frédéric Bonté
      name:Alexis Desmouliere
      name:Alexis Desmouliere
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