Here's how NATURE.COM makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

NATURE . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Nature.com Make Money
  6. How Much Does Nature.com Make
  7. Keywords
  8. Topics
  9. Questions
  10. Schema
  11. Social Networks
  12. External Links
  13. Analytics And Tracking
  14. Libraries
  15. Hosting Providers
  16. CDN Services

We are analyzing https://www.nature.com/articles/nri.2017.142.

Title:
NF-κB, inflammation, immunity and cancer: coming of age | Nature Reviews Immunology
Description:
Here, Koji Taniguchi and Michael Karin discuss the key roles of the nuclear factor-κB (NF-κB) transcription factor in cancer-related inflammation and consider the potential of targeting NF-κB in different types of cancer. Fourteen years have passed since nuclear factor-κB (NF-κB) was first shown to serve as a molecular lynchpin that links persistent infections and chronic inflammation to increased cancer risk. The young field of inflammation and cancer has now come of age, and inflammation has been recognized by the broad cancer research community as a hallmark and cause of cancer. Here, we discuss how the initial discovery of a role for NF-κB in linking inflammation and cancer led to an improved understanding of tumour-elicited inflammation and its effects on anticancer immunity.
Website Age:
30 years and 10 months (reg. 1994-08-11).

Matching Content Categories {📚}

  • Education
  • Health & Fitness
  • Science

Content Management System {📝}

What CMS is nature.com built with?

Custom-built

No common CMS systems were detected on Nature.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of nature.com audience?

🌆 Monumental Traffic: 20M - 50M visitors per month


Based on our best estimate, this website will receive around 42,194,993 visitors per month in the current month.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Nature.com Make Money? {💸}


Display Ads {🎯}


The website utilizes display ads within its content to generate revenue. Check the next section for further revenue estimates.

Ads are managed by yourbow.com. Particular relationships are as follows:

Direct Advertisers (10)
google.com, pmc.com, doceree.com, yourbow.com, audienciad.com, onlinemediasolutions.com, advibe.media, aps.amazon.com, getmediamx.com, onomagic.com

Reseller Advertisers (38)
conversantmedia.com, rubiconproject.com, pubmatic.com, appnexus.com, openx.com, smartadserver.com, lijit.com, sharethrough.com, video.unrulymedia.com, google.com, yahoo.com, triplelift.com, onetag.com, sonobi.com, contextweb.com, 33across.com, indexexchange.com, media.net, themediagrid.com, adform.com, richaudience.com, sovrn.com, improvedigital.com, freewheel.tv, smaato.com, yieldmo.com, amxrtb.com, adyoulike.com, adpone.com, criteo.com, smilewanted.com, 152media.info, e-planning.net, smartyads.com, loopme.com, opera.com, mediafuse.com, betweendigital.com

How Much Does Nature.com Make? {💰}


Display Ads {🎯}

$531,700 per month
Estimations show Nature.com's display ad online revenue falls between $354,480 and $974,820 per month.

Keywords {🔍}

pubmed, google, scholar, cas, cancer, central, nfκb, cell, inflammation, cells, rev, immunol, nat, nature, activation, signaling, karin, role, biol, tumor, immunity, sci, res, factor, tumorigenesis, progression, metastasis, promotes, development, nuclear, function, research, immune, colorectal, foundation, stem, clin, regulation, pathway, med, pancreatic, article, tumour, stat, therapy, interleukin, science, liver, review, taniguchi,

Topics {✒️}

krasg12d-induced ikk2/β/nf-κb activation permissions reprints privacy policy nature portfolio journals nature portfolio editing ikkα-e2f1-bmi1 cascade activated epstein–barr virus-mediated transformation author information authors advertising il-6 feed-forward loop cytokine-driven compensatory proliferation k-ras-induced lung tumorigenesis tgf-β-driven program social media lipopolysaccharide-induced nf-κb activation nf-κb-activating complex engaged rmd open 2 tnf-mediated nf-κb signalling nf-κb subunit p65/rela interferon-sensitive response element interleukin-6-induced epithelial-mesenchymal transition chemotherapy-induced alimentary mucositis nf-κb-dependent induction nf-κb-dependent manner tumour necrosis factor-alpha adenoma-linked barrier defects activation-induced cytidine deaminase steroidal anti-inflammatory drugs nf-κb activation induces carcinogen-induced colorectal tumors myd88-dependent il-6 production hgf/c-met axis mdr2 p-glycoprotein gene anti-cancer agent dmapt c-myc sensitizes cells preventing protein binding transcription factor nf-κb classical nf-κb pathway alternative nf-κb pathway japan cancer society lymphotoxin-driven pathway gp130-mediated stat3 activation nf-κb activation occurs nf-κb activation pathway nf-κb activation triggered nf-κb subunit specificity nf-κb response pathways dissecting molecular cross-talk il-22/il-22r1 axis

Questions {❓}

  • Angiogenesis and chronic inflammation: cause or consequence?
  • Inflammation and cancer: back to Virchow?
  • NF-κB signaling in cancer stem cells: a promising therapeutic target?
  • NSAIDs inhibit tumorigenesis, but how?
  • Notch and NFκB signaling pathways: Do they collaborate in normal vertebrate brain development and function?
  • Redox activation of Nrf2 and NF-κB: a double end sword?
  • Targeting the alternative NF-κB pathway in pancreatic cancer: a new direction for therapy?
  • Therapeutic interventions for gut dysbiosis and related disorders in the elderly: antibiotics, probiotics or faecal microbiota transplantation?

Schema {🗺️}

WebPage:
      mainEntity:
         headline:NF-κB, inflammation, immunity and cancer: coming of age
         description: Here, Koji Taniguchi and Michael Karin discuss the key roles of the nuclear factor-κB (NF-κB) transcription factor in cancer-related inflammation and consider the potential of targeting NF-κB in different types of cancer. Fourteen years have passed since nuclear factor-κB (NF-κB) was first shown to serve as a molecular lynchpin that links persistent infections and chronic inflammation to increased cancer risk. The young field of inflammation and cancer has now come of age, and inflammation has been recognized by the broad cancer research community as a hallmark and cause of cancer. Here, we discuss how the initial discovery of a role for NF-κB in linking inflammation and cancer led to an improved understanding of tumour-elicited inflammation and its effects on anticancer immunity.
         datePublished:2018-01-22T00:00:00Z
         dateModified:2018-01-22T00:00:00Z
         pageStart:309
         pageEnd:324
         sameAs:https://doi.org/10.1038/nri.2017.142
         keywords:
            Inflammation
            NF-kappaB
            Tumour immunology
            Biomedicine
            general
            Immunology
         image:
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig1_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig2_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig3_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig4_HTML.jpg
         isPartOf:
            name:Nature Reviews Immunology
            issn:
               1474-1741
               1474-1733
            volumeNumber:18
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Nature Publishing Group UK
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:Koji Taniguchi
               affiliation:
                     name:Keio University School of Medicine
                     address:
                        name:Department of Microbiology and Immunology, Keio University School of Medicine, Shinjyuku-ku, Japan
                        type:PostalAddress
                     type:Organization
                     name:Graduate School of Medical Sciences, Kyushu University
                     address:
                        name:Department of Surgery and Science, Graduate School of Medical Sciences, Kyushu University, Higashi-ku, Japan
                        type:PostalAddress
                     type:Organization
                     name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
                     address:
                        name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
               name:Michael Karin
               affiliation:
                     name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
                     address:
                        name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:NF-κB, inflammation, immunity and cancer: coming of age
      description: Here, Koji Taniguchi and Michael Karin discuss the key roles of the nuclear factor-κB (NF-κB) transcription factor in cancer-related inflammation and consider the potential of targeting NF-κB in different types of cancer. Fourteen years have passed since nuclear factor-κB (NF-κB) was first shown to serve as a molecular lynchpin that links persistent infections and chronic inflammation to increased cancer risk. The young field of inflammation and cancer has now come of age, and inflammation has been recognized by the broad cancer research community as a hallmark and cause of cancer. Here, we discuss how the initial discovery of a role for NF-κB in linking inflammation and cancer led to an improved understanding of tumour-elicited inflammation and its effects on anticancer immunity.
      datePublished:2018-01-22T00:00:00Z
      dateModified:2018-01-22T00:00:00Z
      pageStart:309
      pageEnd:324
      sameAs:https://doi.org/10.1038/nri.2017.142
      keywords:
         Inflammation
         NF-kappaB
         Tumour immunology
         Biomedicine
         general
         Immunology
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig1_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig2_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig3_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnri.2017.142/MediaObjects/41577_2018_Article_BFnri2017142_Fig4_HTML.jpg
      isPartOf:
         name:Nature Reviews Immunology
         issn:
            1474-1741
            1474-1733
         volumeNumber:18
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Nature Publishing Group UK
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Koji Taniguchi
            affiliation:
                  name:Keio University School of Medicine
                  address:
                     name:Department of Microbiology and Immunology, Keio University School of Medicine, Shinjyuku-ku, Japan
                     type:PostalAddress
                  type:Organization
                  name:Graduate School of Medical Sciences, Kyushu University
                  address:
                     name:Department of Surgery and Science, Graduate School of Medical Sciences, Kyushu University, Higashi-ku, Japan
                     type:PostalAddress
                  type:Organization
                  name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
                  address:
                     name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
            name:Michael Karin
            affiliation:
                  name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
                  address:
                     name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Nature Reviews Immunology
      issn:
         1474-1741
         1474-1733
      volumeNumber:18
Organization:
      name:Nature Publishing Group UK
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Keio University School of Medicine
      address:
         name:Department of Microbiology and Immunology, Keio University School of Medicine, Shinjyuku-ku, Japan
         type:PostalAddress
      name:Graduate School of Medical Sciences, Kyushu University
      address:
         name:Department of Surgery and Science, Graduate School of Medical Sciences, Kyushu University, Higashi-ku, Japan
         type:PostalAddress
      name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
      address:
         name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
         type:PostalAddress
      name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
      address:
         name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Koji Taniguchi
      affiliation:
            name:Keio University School of Medicine
            address:
               name:Department of Microbiology and Immunology, Keio University School of Medicine, Shinjyuku-ku, Japan
               type:PostalAddress
            type:Organization
            name:Graduate School of Medical Sciences, Kyushu University
            address:
               name:Department of Surgery and Science, Graduate School of Medical Sciences, Kyushu University, Higashi-ku, Japan
               type:PostalAddress
            type:Organization
            name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
            address:
               name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:Michael Karin
      affiliation:
            name:Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California
            address:
               name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Microbiology and Immunology, Keio University School of Medicine, Shinjyuku-ku, Japan
      name:Department of Surgery and Science, Graduate School of Medical Sciences, Kyushu University, Higashi-ku, Japan
      name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
      name:Departments of Pharmacology and Pathology, Laboratory of Gene Regulation and Signal Transduction, School of Medicine, University of California, La Jolla, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(761)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Prism.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mxa-002c5801.gslb.pphosted.com
  • mxb-002c5801.gslb.pphosted.com

Name Servers:

  • pdns1.ultradns.net
  • pdns2.ultradns.net
  • pdns3.ultradns.org
  • pdns4.ultradns.org
  • pdns5.ultradns.info
  • pdns6.ultradns.co.uk

CDN Services {📦}

  • Crossref

5.79s.