Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2803035/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC2803035/. The analysis below is for the second page.

Title[redir]:
Inflammation and cancer - PMC
Description:
Recent data have expanded the concept that inflammation is a critical component of tumour progression. Many cancers arise from sites of infection, chronic irritation and inflammation. It is now becoming clear that the tumour microenvironment, which ...

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Ncbi.nlm.nih.gov Make Money? {💸}

The income method remains a mystery to us.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Ncbi.nlm.nih.gov might be making money, but it's not detectable how they're doing it.

Keywords {🔍}

doi, cells, google, scholar, pubmed, cancer, inflammatory, inflammation, tumour, growth, cell, neoplastic, cytokines, chemokines, factors, factor, chronic, tissue, carcinoma, pmc, response, metastasis, activation, angiogenesis, article, macrophages, progression, development, human, free, receptors, tumours, wound, endothelial, chemokine, tumor, expression, vascular, tgfβ, macrophage, mice, proliferation, dna, mechanisms, role, healing, virus, nature, migration, antiinflammatory,

Topics {✒️}

transmembrane-spanning g-protein-coupled receptors mip-3α/ccl20 stimulates growth granulocyte–macrophage colony-stimulating factor macrophage-deficient op/op mouse pmc beta search pro-versus anti-inflammatory cytokines t-cell stimulatory capacity macrophage pro-inflammatory chemokine-3α pro-inflammatory cytokine tnf-α carbohydrate structure sialyl-lewis mip-3α/larc/exodus nonsteroidal anti-inflammatory drugs cell-dependent tumor rejection anti-cancer therapeutic opportunity anti-inflammatory therapeutic approaches glu-leu-arg/elr transforming growth factor-α transforming growth factor-β murine mcp-5/ccl12 exhibits epstein-barr virus aids platelet-derived growth factor small-cell lung carcinoma44 early-response pro-inflammatory cytokines /p-selectin-deficient mice52 tumour necrosis factor-α tumour–platelet–leukocyte emboli strong anti-metastatic effects csf-1-dependent infiltrating macrophages macrophage-regulated tumour stromal-cell-derived factor 1 stimulate t-lymphocyte activation melanocytes express il-8 pymt/csf1op/csf1op mice urokinase-type plasminogen activator late-stage invasive carcinoma distinct tumour-mediated mechanisms colony-stimulating factor 1 th2-type inflammatory response platelet-derived angiogenesis factor macrophage inflammatory protein-1α colony-forming activity epithelial-neutrophil activating peptide mip-3α/ccl20 express sialyl-lewis enhanced tumor-forming capacity keratinocyte growth factor regulate vascular permeability epstein-barr virus cxc chemokine-mediated angiogenesis tgf-β receptors change

Questions {❓}

  • Can we apply these new insights for targeting metastases?
  • Chemokines: agents for the immunotherapy of cancer?
  • Does the tumour need inflammation to help foster angiogenesis?
  • How are inflammatory cells co-opted into the neoplastic process?
  • Inflammation and cancer: back to Virchow?
  • Inflammatory cells and cancer: friend or foe?
  • Tumor necrosis factor or tumor promoting factor?
  • Tumors and inflammatory infiltrates: friends or foes?
  • Why does the inflammatory response to tumours persist?

Social Networks {👍}(6)

External Links {🔗}(149)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

3.68s.