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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers

We began analyzing https://link.springer.com/article/10.1007/s00401-015-1407-2, but it redirected us to https://link.springer.com/article/10.1007/s00401-015-1407-2. The analysis below is for the second page.

Title[redir]:
PART, a distinct tauopathy, different from classical sporadic Alzheimer disease | Acta Neuropathologica
Description:
Acta Neuropathologica -

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 80,904,851 visitors per month in the current month.

check SE Ranking
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How Does Doi.org Make Money? {💸}

We can't see how the site brings in money.

Many websites are intended to earn money, but some serve to share ideas or build connections. Websites exist for all kinds of purposes. This might be one of them. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

pubmed, article, google, scholar, disease, cas, neuropathol, central, usa, department, acta, pathology, university, dementia, alzheimers, alzheimer, neurol, brain, neurofibrillary, neurology, jellinger, thal, institute, van, neuropathology, medicine, research, attems, thomas, tau, school, center, york, part, dickson, jack, knopman, nelson, trojanowski, senile, braak, aging, masliah, study, preclinical, privacy, cookies, cairns, hof, jicha,

Topics {✒️}

month download article/chapter jack cr jr primary age-related tauopathy thr113-114ins presenilin-1 mutation age-specific population frequencies amyloid-beta peptide aggregation full article pdf neurofibrillary tangle-predominant dementia ad-related pathological process privacy choices/manage cookies high-morbidity brain disease albert serrano-pozo cerebral beta-amyloidosis hauw jj predicting cognitive impairment van duijn cm van hoeven kh neurofibrillary tangle type references bancher neuritic amyloid plaques article jellinger jack jr van de loo van eldik lj harvard medical school tangle-predominant dementia neurodegenerative disease research european economic area year autopsy population soto-ortolaza ai perforant pathway neurons lewy body variant graff-radford nr neuropathologically defined subtypes nondemented aged subjects george-hyslop ps guamanian chamorros free overt neurologic dysfunction california san diego author correspondence article log hamilton-nelson kl sanders-brown center conditions privacy policy classical alzheimer disease neuronal injury biomarkers corticobasal degeneration brains white cl 3rd chartier-harlin mc de bruijn rfag

Questions {❓}

  • Braak H, Del Tredici K (2014) Are cases with tau pathology occurring in the absence of Abeta deposits part of the AD-related pathological process?
  • Knopman DS (2014) beta-Amyloidosis and neurodegeneration in Alzheimer disease: who’s on first?

Schema {🗺️}

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