Here's how LINK.SPRINGER.COM makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Schema
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://link.springer.com/article/10.1007/s00401-006-0156-7.

Title:
Neurofibrillary tangle-predominant dementia: comparison with classical Alzheimer disease | Acta Neuropathologica
Description:
Neurofibrillary tangle predominant dementia (NFTPD) is a subset of late onset dementia, clinically different from traditional “plaque and tangle” Alzheimer disease (AD): later onset, shorter duration, less severe cognitive impairment, and almost absence of ApoE ε4. Neuropathology reveals abundant allocortical neurofibrillary pathology with no or few isocortical tau lesions, absence of neuritic plaques, absence or scarcity of amyloid deposits, but neurofibrillary changes comprising both 3 and 4 repeat (3R and 4R) tau immunohistochemistry are not significantly different from those in classical AD. Comparing 51 autopsy cases of NFTPD with 244 classical AD subjects, the nosology of NFTPD and its differences from AD are discussed.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of link.springer.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Link.springer.com Make Money? {💸}

The income method remains a mystery to us.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Link.springer.com might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

google, scholar, cas, article, pubmed, disease, dementia, neuropathol, neurofibrillary, alzheimers, acta, tau, senile, dickson, alzheimer, berl, jellinger, neurol, tangles, tangle, neuropathology, pathology, degeneration, type, apolipoprotein, corticobasal, brain, pathol, abstract, study, ikeda, eds, attems, progressive, supranuclear, palsy, argyrophilic, grain, diagnosis, disorders, akiyama, iseki, silva, tauopathies, amyloid, repeat, neurology, allele, tauopathy, morris,

Topics {✒️}

month download article/chapter paired helical filament-tau mutation-specific functional impairments alpha-synuclein brainstem pathology microtubule-binding repeat domains neurofibrillary tangle-predominant dementia 4r tau-specific immunohistochemistry glial fibrillary tangles age-related biochemical modifications full article pdf specific monoclonal antibodies tangle predominant form privacy choices/manage cookies tangle-predominant variants alzheimer-type neurofibrillary lesions 4-repeat tau isoforms argentophilic glial inclusions differential laminar distributions abundant neurofibrillary tangles tau protein isoforms frontotemporal lobar degenerations region-specific dissociation widespread neurofibrillary tangles microtubule-binding region microtubule-binding repeats distinct tau isoforms neurofibrillary tangle type atypical parkinsonian disorders pericak-vance ma neurodegenerative diseases extracellular neurofibrillary tangles subcortical neurofibrillary tangles proteins alpha 1-antichymotrypsin de vos ra tangle-bearing neurons beta a4 deposits late onset families european economic area scope submit manuscript severe cognitive impairment progressive supranuclear palsy common pathological feature thorn-shaped astrocytes transgenic animal models additional 74-kda component widespread argentophilic threads jansen steur en selective neurofibrillary degeneration 1007/s00401-006-0146-9 attems isocortical tau lesions

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Neurofibrillary tangle-predominant dementia: comparison with classical Alzheimer disease
         description:Neurofibrillary tangle predominant dementia (NFTPD) is a subset of late onset dementia, clinically different from traditional “plaque and tangle” Alzheimer disease (AD): later onset, shorter duration, less severe cognitive impairment, and almost absence of ApoE ε4. Neuropathology reveals abundant allocortical neurofibrillary pathology with no or few isocortical tau lesions, absence of neuritic plaques, absence or scarcity of amyloid deposits, but neurofibrillary changes comprising both 3 and 4 repeat (3R and 4R) tau immunohistochemistry are not significantly different from those in classical AD. Comparing 51 autopsy cases of NFTPD with 244 classical AD subjects, the nosology of NFTPD and its differences from AD are discussed.
         datePublished:2006-11-07T00:00:00Z
         dateModified:2006-11-07T00:00:00Z
         pageStart:107
         pageEnd:117
         sameAs:https://doi.org/10.1007/s00401-006-0156-7
         keywords:
            Senile dementia with tangles
            Alzheimer disease
            3 and 4 repeat tau isoforms
            Tauopathy
            Differential diagnosis
            Neuropathology
            Pathology
            Neurosciences
         image:
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig1_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig2_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig3_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig4_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig5_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig6_HTML.jpg
         isPartOf:
            name:Acta Neuropathologica
            issn:
               1432-0533
               0001-6322
            volumeNumber:113
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Springer-Verlag
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:K. A. Jellinger
               affiliation:
                     name:Institute of Clinical Neurobiology
                     address:
                        name:Institute of Clinical Neurobiology, Vienna, Austria
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
               name:J. Attems
               affiliation:
                     name:OWS Hospital (JA)
                     address:
                        name:Department of Pathology, OWS Hospital (JA), Vienna, Austria
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Neurofibrillary tangle-predominant dementia: comparison with classical Alzheimer disease
      description:Neurofibrillary tangle predominant dementia (NFTPD) is a subset of late onset dementia, clinically different from traditional “plaque and tangle” Alzheimer disease (AD): later onset, shorter duration, less severe cognitive impairment, and almost absence of ApoE ε4. Neuropathology reveals abundant allocortical neurofibrillary pathology with no or few isocortical tau lesions, absence of neuritic plaques, absence or scarcity of amyloid deposits, but neurofibrillary changes comprising both 3 and 4 repeat (3R and 4R) tau immunohistochemistry are not significantly different from those in classical AD. Comparing 51 autopsy cases of NFTPD with 244 classical AD subjects, the nosology of NFTPD and its differences from AD are discussed.
      datePublished:2006-11-07T00:00:00Z
      dateModified:2006-11-07T00:00:00Z
      pageStart:107
      pageEnd:117
      sameAs:https://doi.org/10.1007/s00401-006-0156-7
      keywords:
         Senile dementia with tangles
         Alzheimer disease
         3 and 4 repeat tau isoforms
         Tauopathy
         Differential diagnosis
         Neuropathology
         Pathology
         Neurosciences
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig1_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig2_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig3_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig4_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig5_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00401-006-0156-7/MediaObjects/401_2006_156_Fig6_HTML.jpg
      isPartOf:
         name:Acta Neuropathologica
         issn:
            1432-0533
            0001-6322
         volumeNumber:113
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Springer-Verlag
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:K. A. Jellinger
            affiliation:
                  name:Institute of Clinical Neurobiology
                  address:
                     name:Institute of Clinical Neurobiology, Vienna, Austria
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
            name:J. Attems
            affiliation:
                  name:OWS Hospital (JA)
                  address:
                     name:Department of Pathology, OWS Hospital (JA), Vienna, Austria
                     type:PostalAddress
                  type:Organization
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Acta Neuropathologica
      issn:
         1432-0533
         0001-6322
      volumeNumber:113
Organization:
      name:Springer-Verlag
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Institute of Clinical Neurobiology
      address:
         name:Institute of Clinical Neurobiology, Vienna, Austria
         type:PostalAddress
      name:OWS Hospital (JA)
      address:
         name:Department of Pathology, OWS Hospital (JA), Vienna, Austria
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:K. A. Jellinger
      affiliation:
            name:Institute of Clinical Neurobiology
            address:
               name:Institute of Clinical Neurobiology, Vienna, Austria
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:J. Attems
      affiliation:
            name:OWS Hospital (JA)
            address:
               name:Department of Pathology, OWS Hospital (JA), Vienna, Austria
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Institute of Clinical Neurobiology, Vienna, Austria
      name:Department of Pathology, OWS Hospital (JA), Vienna, Austria
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

External Links {🔗}(207)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • Prism.js

CDN Services {📦}

  • Crossref

4.5s.