Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC7969520/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC7969520/. The analysis below is for the second page.

Title[redir]:
Neutrophils: Many Ways to Die - PMC
Description:
Neutrophils or polymorphonuclear leukocytes (PMN) are key participants in the innate immune response for their ability to execute different effector functions. These cells express a vast array of membrane receptors that allow them to recognize and ...

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't see how the site brings in money.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Ncbi.nlm.nih.gov might be earning cash quietly, but we haven't detected the monetization method.

Keywords {🔍}

pubmed, google, scholar, doi, neutrophils, cell, apoptosis, neutrophil, death, pmc, free, article, activation, cells, immunol, extracellular, human, receptors, caspase, biol, necrosis, inflammation, autophagy, ros, netosis, process, intracellular, blood, membrane, formation, nadph, role, phagocytosis, protein, nets, proteins, pyroptosis, traps, mechanisms, oxidase, production, regulation, inflammatory, pathway, factor, survival, pathways, molecules, release, expression,

Topics {✒️}

granulocyte/macrophage colony–stimulating factor granulocyte-macrophage colony-stimulating factor granulocyte-monocyte colony-stimulating factor tnf-related apopotosis-inducing ligand granulocyte-colony stimulating factor granulocyte colony-stimulating factor tnf-α-induced neutrophil apoptosis tnf-related apoptosis-inducing ligand heat-killed escherichia coli ps-binding proteins mfg-e8 tumor necrosis factor-α tnf-α-mediated neutrophil apoptosis tnf-α-mediated antiapoptotic signaling tumor necrosis factor-alpha nf-κb-dependent il-8 release caspase-1-dependent necrotic-type death idiosyncratic drug-induced agranulocytosis caspase-independent cell-death pathway muckle-wells autoinflammatory disorder cytokine-prestimulated endothelial cells gram-negative bacteria dai/zbp1/dlm-1 complexes severe multi-organ consequences pmc beta search sars-cov-2 infection raf-mek-erk pathway β-glucan-bearing particles heat-killed staphylococcus aureus nf-κb-dependent pathway small-molecular-weight colorants helicobacter pylori-infected neutrophils endotoxin-induced pulmonary inflammation nf-κb survival factor lipoprotein receptor-related protein infection-prone cellular objective anti-inflammatory signals start pi3-kinase/pkb contributes caspase-1-dependent pore formation pmn-specific serine protease il-1beta-processing inflammasome ia-type phosphoinositide 3-kinase nf-κb signaling pathways neutrophil-predominant severe asthma mitogen-activated protein kinases host-defense integrated system tnf-α-stimulated cells nf-κb-independent role induce nadph-dependent netosis calcium-activated potassium channels caspase-independent death pathway

Questions {❓}

  • Haemophilus influenzae induces neutrophil necrosis: a role in chronic obstructive pulmonary disease?
  • How neutrophils die or survive along NET release and is “NETosis” = necroptosis?
  • NLRP3 inflammasome activation: the convergence of multiple signalling pathways on ROS production?
  • ROS induced p53 activation: DNA damage, redox signaling or both?

Social Networks {👍}(6)

External Links {🔗}(577)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Particles.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

5.2s.