We are analyzing https://link.springer.com/chapter/10.1007/978-3-030-62026-4_7.

Title:
Necroptosis , the Other Main Caspase-Independent Cell Death | SpringerLink
Description:
The past decades witnessed the discovery of novel modes of cell death, such as ferroptosis, pyroptosis and necroptosis, all of them presenting common necrotic traits. In this chapter, we revisit the early discoveries that unveiled necroptosis as a distinct cell death...
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Keywords {🔍}

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Topics {✒️}

nuclear factor kappa-light-chain-enhancer nf-kappab-independent cell-death switch receptor-interacting kinase-3-mediated pathway alix-syntenin-1-mediated exocytosis protect caspase-mediated p65/rela cleavage month download article/chapter ch'en il caspase-independent apoptosis pathway radiation-induced mutator phenotype escrt-iii acts downstream cell death-independent neuroinflammation tnfα-induced necroptotic signaling ubiquitylation-dependent proteasomal degradation death-receptor-induced apoptosis cellular flice-inhibitory protein dai/zbp1/dlm-1 complexes rip3-induced programmed necrosis receptor-interacting protein 1 z-dna binding domain fas-induced dna fragmentation murine cytomegalovirus vira phosphorylation-driven assembly receptor 3-mediated necrosis anti-viral inflammation heighten intestinal inflammation rip1/rip3 necrosome forms necroptotic cell death apoptosis-inducing kinase dysfunctional hiv-specific cd8+ tumor necrosis factor virus-induced inflammation necrotic cell death privacy choices/manage cookies cochaperone cdc37 complex thapa rj chronic lymphocytic leukemia tnf-induced nf petrie ej device instant download hydrogen peroxide mediates regulates endosomal trafficking blunt mechanical impact nf-kappab signaling dna-dependent activator caspase-8-deficient mice ripk1-mediated neuroinflammation upton jw flotillin-mediated endocytosis pattern recognition receptors helix bundle domain

Questions {❓}

Kelliher MA, Grimm S, Ishida Y, Kuo F, Stanger BZ, Leder P (1998) The death domain kinase RIP mediates the TNF-induced NF-?
Marshall KD, Baines CP (2014) Necroptosis: is there a role for mitochondria?
Wang T, Jin Y, Yang W, Zhang L, Jin X, Liu X et al (2017) Necroptosis in cancer: an angel or a demon?

Schema {🗺️}

ScholarlyArticle:
      headline:Necroptosis , the Other Main Caspase-Independent Cell Death
      pageEnd:138
      pageStart:123
      image:https://media.springernature.com/w153/springer-static/cover/book/978-3-030-62026-4.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Ferroptosis: Mechanism and Diseases
         isbn:
            978-3-030-62026-4
            978-3-030-62025-7
         type:Book
      publisher:
         name:Springer International Publishing
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Larissa C. Zanetti
            affiliation:
                  name:Hospital Israelita Albert Einstein. Av. Albert Einstein
                  address:
                     name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
            name:Ricardo Weinlich
            affiliation:
                  name:Hospital Israelita Albert Einstein. Av. Albert Einstein
                  address:
                     name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
                     type:PostalAddress
                  type:Organization
            type:Person
      keywords:Necroptosis, RIPK3, MLKL, RIPK1, Inflammation, TNF
      description:The past decades witnessed the discovery of novel modes of cell death, such as ferroptosis, pyroptosis and necroptosis, all of them presenting common necrotic traits. In this chapter, we revisit the early discoveries that unveiled necroptosis as a distinct cell death mechanism. We describe necroptosis, its main regulators and their role in maintaining cellular homeostasis and in the disease state. We conclude by discussing its phenotypic similarities with ferroptosis and the possible crosstalk between these pathways.
      datePublished:2021
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         978-3-030-62025-7
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      name:Springer International Publishing
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            name:Hospital Israelita Albert Einstein. Av. Albert Einstein
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               name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:Ricardo Weinlich
      affiliation:
            name:Hospital Israelita Albert Einstein. Av. Albert Einstein
            address:
               name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
               type:PostalAddress
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      name:Hospital Israelita Albert Einstein. Av. Albert Einstein, São Paulo, Brazil
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