Here's how NATURE.COM makes money* and how much!

*Please read our disclaimer before using our estimates.
Loading...

NATURE . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Nature.com Make Money
  6. How Much Does Nature.com Make
  7. Keywords
  8. Topics
  9. Questions
  10. Schema
  11. Social Networks
  12. External Links
  13. Analytics And Tracking
  14. Libraries
  15. Hosting Providers
  16. CDN Services

We are analyzing https://www.nature.com/articles/nrn3745.

Title:
Neurobiology of food intake in health and disease | Nature Reviews Neuroscience
Description:
The energy homeostasis system maintains stability of body fat stores over time but can be overridden by activation of feeding neurocircuits during emergent or stressful conditions. In this Review, Schwartz and colleagues highlight crosstalk between homeostatic and emergency feeding circuits in the regulation of energy balance. Under normal conditions, food intake and energy expenditure are balanced by a homeostatic system that maintains stability of body fat content over time. However, this homeostatic system can be overridden by the activation of
Website Age:
30 years and 10 months (reg. 1994-08-11).

Matching Content Categories {πŸ“š}

  • Education
  • Health & Fitness
  • Fitness & Wellness

Content Management System {πŸ“}

What CMS is nature.com built with?

Custom-built

No common CMS systems were detected on Nature.com, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of nature.com audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

check SE Ranking
check Ahrefs
check Similarweb
check Ubersuggest
check Semrush

How Does Nature.com Make Money? {πŸ’Έ}


Display Ads {🎯}


The website utilizes display ads within its content to generate revenue. Check the next section for further revenue estimates.

Ads are managed by yourbow.com. Particular relationships are as follows:

Direct Advertisers (10)
google.com, pmc.com, doceree.com, yourbow.com, audienciad.com, onlinemediasolutions.com, advibe.media, aps.amazon.com, getmediamx.com, onomagic.com

Reseller Advertisers (38)
conversantmedia.com, rubiconproject.com, pubmatic.com, appnexus.com, openx.com, smartadserver.com, lijit.com, sharethrough.com, video.unrulymedia.com, google.com, yahoo.com, triplelift.com, onetag.com, sonobi.com, contextweb.com, 33across.com, indexexchange.com, media.net, themediagrid.com, adform.com, richaudience.com, sovrn.com, improvedigital.com, freewheel.tv, smaato.com, yieldmo.com, amxrtb.com, adyoulike.com, adpone.com, criteo.com, smilewanted.com, 152media.info, e-planning.net, smartyads.com, loopme.com, opera.com, mediafuse.com, betweendigital.com

How Much Does Nature.com Make? {πŸ’°}


Display Ads {🎯}

$63,100 per month
Our analysis indicates Nature.com generates between $42,042 and $115,616 monthly online from display ads.

Keywords {πŸ”}

pubmed, google, scholar, article, cas, central, leptin, nature, food, energy, neurons, obesity, intake, physiol, hypothalamic, feeding, clin, schwartz, insulin, body, control, weight, diabetes, response, brain, neurosci, rats, receptor, metab, homeostasis, role, invest, agrp, mice, nucleus, cell, balance, melanocortin, system, neuronal, circuits, med, oxytocin, neuron, obese, adiposity, endocrinology, satiety, reward, homeostatic,

Topics {βœ’οΈ}

permissions reprints nature portfolio privacy policy body mass index diabetes research center advertising regional brain response medical research service sandra social media author information authors hypothalamic ikkΞ²/nf-ΞΊb arcuate pro-opiomelanocortin neurons author correspondence palatable high-fat diets insulin activates atp-sensitive physical activity decision-making central leptin infusion atp-sensitive potassium channels fat-rich diet activates cck-induced meal suppression emergency-activated circuits interact personal data serum immunoreactive-leptin concentrations high-fat feeding underlies central hormone resistance body fat content springerlink instant access permissions hypothalamic paraventricular nucleus pro-opiomelanocortin neurons oxytocin-deficient mice data protection fasting-activated hypothalamic neurons long-term orexigenic effects lean-body-mass composition 2-deoxy-d-glucose administration central neural networks obesity-related insulin resistance streptozotocin-induced diabetic rats normal body-weight homeostasis nature genet oxytocin inhibits food nature neurosci express pro-opiomelanocortin nature med nature rev increases food intake early-onset obesity agouti-related protein

Questions {❓}

  • Adiposity signaling and biological defense against weight gain: absence of protection or central hormone resistance?
  • Glucose-sensing neurons: are they physiologically relevant?
  • Hypothalamic inflammation: marker or mechanism of obesity pathogenesis?

Schema {πŸ—ΊοΈ}

WebPage:
      mainEntity:
         headline:Neurobiology of food intake in health and disease
         description: The energy homeostasis system maintains stability of body fat stores over time but can be overridden by activation of feeding neurocircuits during emergent or stressful conditions. In this Review, Schwartz and colleagues highlight crosstalk between homeostatic and emergency feeding circuits in the regulation of energy balance. Under normal conditions, food intake and energy expenditure are balanced by a homeostatic system that maintains stability of body fat content over time. However, this homeostatic system can be overridden by the activation of 'emergency response circuits' that mediate feeding responses to emergent or stressful stimuli. Inhibition of these circuits is therefore permissive for normal energy homeostasis to occur, and their chronic activation can cause profound, even life-threatening, changes in body fat mass. This Review highlights how the interplay between homeostatic and emergency feeding circuits influences the biologically defended level of body weight under physiological and pathophysiological conditions.
         datePublished:2014-05-20T00:00:00Z
         dateModified:2014-05-20T00:00:00Z
         pageStart:367
         pageEnd:378
         sameAs:https://doi.org/10.1038/nrn3745
         keywords:
            Feeding behaviour
            Homeostasis
            Hypothalamus
            Biomedicine
            general
            Neurosciences
            Behavioral Sciences
            Biological Techniques
            Neurobiology
            Animal Genetics and Genomics
         image:
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig1_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig2_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig3_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig4_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Figa_HTML.jpg
            https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig5_HTML.jpg
         isPartOf:
            name:Nature Reviews Neuroscience
            issn:
               1471-0048
               1471-003X
            volumeNumber:15
            type:
               Periodical
               PublicationVolume
         publisher:
            name:Nature Publishing Group UK
            logo:
               url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
               type:ImageObject
            type:Organization
         author:
               name:Gregory J. Morton
               affiliation:
                     name:Diabetes and Obesity Center of Excellence, University of Washington
                     address:
                        name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Thomas H. Meek
               affiliation:
                     name:Diabetes and Obesity Center of Excellence, University of Washington
                     address:
                        name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Michael W. Schwartz
               affiliation:
                     name:Diabetes and Obesity Center of Excellence, University of Washington
                     address:
                        name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
                        type:PostalAddress
                     type:Organization
               email:[email protected]
               type:Person
         isAccessibleForFree:
         hasPart:
            isAccessibleForFree:
            cssSelector:.main-content
            type:WebPageElement
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Neurobiology of food intake in health and disease
      description: The energy homeostasis system maintains stability of body fat stores over time but can be overridden by activation of feeding neurocircuits during emergent or stressful conditions. In this Review, Schwartz and colleagues highlight crosstalk between homeostatic and emergency feeding circuits in the regulation of energy balance. Under normal conditions, food intake and energy expenditure are balanced by a homeostatic system that maintains stability of body fat content over time. However, this homeostatic system can be overridden by the activation of 'emergency response circuits' that mediate feeding responses to emergent or stressful stimuli. Inhibition of these circuits is therefore permissive for normal energy homeostasis to occur, and their chronic activation can cause profound, even life-threatening, changes in body fat mass. This Review highlights how the interplay between homeostatic and emergency feeding circuits influences the biologically defended level of body weight under physiological and pathophysiological conditions.
      datePublished:2014-05-20T00:00:00Z
      dateModified:2014-05-20T00:00:00Z
      pageStart:367
      pageEnd:378
      sameAs:https://doi.org/10.1038/nrn3745
      keywords:
         Feeding behaviour
         Homeostasis
         Hypothalamus
         Biomedicine
         general
         Neurosciences
         Behavioral Sciences
         Biological Techniques
         Neurobiology
         Animal Genetics and Genomics
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig1_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig2_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig3_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig4_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Figa_HTML.jpg
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1038%2Fnrn3745/MediaObjects/41583_2014_Article_BFnrn3745_Fig5_HTML.jpg
      isPartOf:
         name:Nature Reviews Neuroscience
         issn:
            1471-0048
            1471-003X
         volumeNumber:15
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Nature Publishing Group UK
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Gregory J. Morton
            affiliation:
                  name:Diabetes and Obesity Center of Excellence, University of Washington
                  address:
                     name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Thomas H. Meek
            affiliation:
                  name:Diabetes and Obesity Center of Excellence, University of Washington
                  address:
                     name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Michael W. Schwartz
            affiliation:
                  name:Diabetes and Obesity Center of Excellence, University of Washington
                  address:
                     name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
                     type:PostalAddress
                  type:Organization
            email:[email protected]
            type:Person
      isAccessibleForFree:
      hasPart:
         isAccessibleForFree:
         cssSelector:.main-content
         type:WebPageElement
["Periodical","PublicationVolume"]:
      name:Nature Reviews Neuroscience
      issn:
         1471-0048
         1471-003X
      volumeNumber:15
Organization:
      name:Nature Publishing Group UK
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Diabetes and Obesity Center of Excellence, University of Washington
      address:
         name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
         type:PostalAddress
      name:Diabetes and Obesity Center of Excellence, University of Washington
      address:
         name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
         type:PostalAddress
      name:Diabetes and Obesity Center of Excellence, University of Washington
      address:
         name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
         type:PostalAddress
ImageObject:
      url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
Person:
      name:Gregory J. Morton
      affiliation:
            name:Diabetes and Obesity Center of Excellence, University of Washington
            address:
               name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
               type:PostalAddress
            type:Organization
      name:Thomas H. Meek
      affiliation:
            name:Diabetes and Obesity Center of Excellence, University of Washington
            address:
               name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
               type:PostalAddress
            type:Organization
      name:Michael W. Schwartz
      affiliation:
            name:Diabetes and Obesity Center of Excellence, University of Washington
            address:
               name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
PostalAddress:
      name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
      name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
      name:Department of Medicine, Diabetes and Obesity Center of Excellence, University of Washington, Seattle, USA
WebPageElement:
      isAccessibleForFree:
      cssSelector:.main-content

Social Networks {πŸ‘}(2)

External Links {πŸ”—}(562)

Analytics and Tracking {πŸ“Š}

  • Google Tag Manager

Libraries {πŸ“š}

  • Prism.js
  • Zoom.js

Emails and Hosting {βœ‰οΈ}

Mail Servers:

  • mxa-002c5801.gslb.pphosted.com
  • mxb-002c5801.gslb.pphosted.com

Name Servers:

  • pdns1.ultradns.net
  • pdns2.ultradns.net
  • pdns3.ultradns.org
  • pdns4.ultradns.org
  • pdns5.ultradns.info
  • pdns6.ultradns.co.uk

CDN Services {πŸ“¦}

  • Crossref

6.76s.