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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. Social Networks
  11. External Links
  12. Analytics And Tracking
  13. Libraries
  14. Hosting Providers
  15. CDN Services

We began analyzing https://www.nature.com/articles/nrgastro.2012.152, but it redirected us to https://www.nature.com/articles/nrgastro.2012.152. The analysis below is for the second page.

Title[redir]:
The gut microbiota in IBD | Nature Reviews Gastroenterology & Hepatology
Description:
IBD is emerging as a worldwide epidemic. Patients with IBD often have an abnormal gut microbiota; however, it is unknown whether this feature is a cause or a consequence of disease. In this article, Francisco Guarner and colleagues review our current knowledge of the human gut microbiota, describe changes observed in patients with IBD and discuss whether such changes might explain the pathophysiological characteristics of IBD. IBD—ulcerative colitis and Crohn

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 8,426,036 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We don't see any clear sign of profit-making.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Doi.org might be plotting its profit, but the way they're doing it isn't detectable yet.

Keywords {🔍}

article, google, scholar, cas, gut, disease, bowel, microbiota, nature, human, colitis, inflammatory, microbial, crohns, ulcerative, gastroenterology, patients, dis, ibd, access, inflamm, intestinal, gastroenterol, bacteria, content, mucosal, diseases, mucosa, microbiome, cookies, analysis, data, nat, clin, science, coli, privacy, borruel, guarner, colonization, immune, commensal, inflammation, online, sci, hepatol, colonic, bacterial, review, manichanh,

Topics {✒️}

nature portfolio permissions reprints privacy policy advertising nature 453 nature 464 nature 473 nature social media regional ileitis rural africa powerful bioinformatic tools author information authors cyclic-di-amp confers author correspondence regulating nuclear-cytoplasmic shuttling grant agreement health-f4-2007-201052 socioeconomic development tools gastrointestinal microbiology enters springerlink instant access permissions molecular biological access ribosomal database project adherent-invasive escherichia coli development personal data active ulcerative colitis data protection distal digestive tract big data human microbiome project privacy fusobacterium varium localized fusobacterium varium isolated inflammatory bowel diseases francisco guarner european economic area systematic review review series indigenous clostridium species de novo assembly long polar fimbriae ministerio de ciencia competing financial interests human gut microbiome desulfovibrio bacterial species dysregulated immune response inflammatory bowel disease enteric commensal flora

Questions {❓}

  • Do metalloproteinases contribute to tissue destruction or remodeling in the inflamed gut?
  • TH17 cells in autoimmunity and immunodeficiency: protective or pathogenic?
  • What is the role of the enteric commensal flora in IBD?

Schema {🗺️}

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         description:IBD is emerging as a worldwide epidemic. Patients with IBD often have an abnormal gut microbiota; however, it is unknown whether this feature is a cause or a consequence of disease. In this article, Francisco Guarner and colleagues review our current knowledge of the human gut microbiota, describe changes observed in patients with IBD and discuss whether such changes might explain the pathophysiological characteristics of IBD. IBD—ulcerative colitis and Crohn's disease—is emerging as a worldwide epidemic. An association between the increased incidence of IBD and environmental factors linked to socioeconomic development has been persistently detected in different parts of the world. The lifestyle in developed countries might impair the natural patterns of microbial colonization of the human gut. The interaction of microbes with mucosal immune compartments in the gut seems to have a major role in priming and regulating immunity. In IBD, mucosal lesions are generated by an excessive or dysregulated immune response against commensal microbes in the gut. In individuals with a genetic susceptibility to IBD, abnormal microbial colonization of the gastrointestinal tract might be the origin of such dysregulation. Developments in gene-sequencing technologies, as well as increased availability of powerful bioinformatic tools, have enabled novel insights into the microbial composition of the human gut microbiota and the effect of microbial communities on human physiology and disease. Studies that used these technologies indicate that dysbiosis (that is, abnormal microbiota composition) and decreased complexity of the gut microbial ecosystem are common features in patients with Crohn's disease or ulcerative colitis. Whether such changes are a cause or a consequence of the disease remains to be elucidated.
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Social Networks {👍}(2)

External Links {🔗}(246)

Analytics and Tracking {📊}

  • Google Tag Manager

Libraries {📚}

  • Prism.js
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com

CDN Services {📦}

  • Crossref

5.14s.