DOI . ORG {
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We began analyzing https://link.springer.com/chapter/10.1007/978-3-319-07320-0_9, but it redirected us to https://link.springer.com/chapter/10.1007/978-3-319-07320-0_9. The analysis below is for the second page.
Title[redir]:
Serotonin Modulation of Macrophage Polarization: Inflammation and Beyond | SpringerLink
Description:
Macrophages display a ample plethora of effector functions whose acquisition is promoted by the surrounding cytokine and cellular environment. Depending on the stimulus, macrophages become specialized (“polarized”) for either pathogen elimination, tissue...
Title[redir]:
Serotonin Modulation of Macrophage Polarization: Inflammation and Beyond | SpringerLink
Description:
Macrophages display a ample plethora of effector functions whose acquisition is promoted by the surrounding cytokine and cellular environment. Depending on the stimulus, macrophages become specialized (“polarized”) for either pathogen elimination, tissue...
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Keywords {🔍}
pubmed, google, scholar, cas, central, serotonin, macrophages, macrophage, receptor, immunol, cells, cancer, cell, polarization, human, receptors, activation, med, res, htb, nat, inflammation, immune, blood, tumor, factor, role, sci, pharmacol, expression, rev, liver, clin, tissue, inflammatory, immunity, biol, chem, chapter, mantovani, tumorassociated, mice, invest, regulation, growth, functions, responses, gene, dendritic, system,
Topics {✒️}
ccaat/enhancer-binding protein beta granulocyte-macrophage colony-stimulating factor human m-csf-generated macrophages 5-ht7-receptor-mediated gastric relaxation macrophage-csf-dependent macrophage responses chemotherapy drug-induced apoptosis transforming growth factor-beta1 m2 anti-inflammatory/regulatory macrophages macrophage colony-stimulating factor structure-activity relationship study lipopolysaccharide-induced gene activation serotonin-selective antidepressant actions 5-ht7 receptor-mediated relaxation month download article/chapter t-cell priming capacity lps-primed human monocytes serum-free tissue culture anti-inflammatory macrophage markers muscle layer-dependent manner selective serotonin-reuptake inhibitors central nervous system ras-dependent erk activation lps-induced il-10 release cytokine-driven compensatory proliferation enhanced irf-3/stat1 activation high-affinity serotonin receptor enhance t-cell activation adipocyte-derived th2 cytokines antiapoptotic mediator sphingosine-1-phosphate g-protein signal transduction resolution-phase macrophages possess cytokine-induced sickness behaviour central 5-ht7 receptors serotonin/5-ht2b receptor signaling macrophage-csf dependent nebigil cg 5-hydroxytryptamine induced relaxation tumor-induced immune suppression receptor gene products anti-inflammatory state van ginderachter ja m-csf origin interferon-gamma induced phagocytosis palsson-mcdermott em phosphatidylinositol-specific phospholipase privacy choices/manage cookies 5-ht7 receptor research differential macrophage programming primary pulmonary hypertension skews macrophage polarization
Questions {❓}
- 5-HT2B Receptor on Macrophages: What for?
- Functional consequence of serotonin/5-HT2B receptor signaling in heart: role of mitochondria in transition between hypertrophy and heart failure?
- Serotonin: a double-edged sword for the liver?
Schema {🗺️}
ScholarlyArticle:
headline:Serotonin Modulation of Macrophage Polarization: Inflammation and Beyond
pageEnd:115
pageStart:89
image:https://media.springernature.com/w153/springer-static/cover/book/978-3-319-07320-0.jpg
genre:
Biomedical and Life Sciences
Biomedical and Life Sciences (R0)
isPartOf:
name:Oxidative Stress and Inflammation in Non-communicable Diseases - Molecular Mechanisms and Perspectives in Therapeutics
isbn:
978-3-319-07320-0
978-3-319-07319-4
type:Book
publisher:
name:Springer International Publishing
logo:
url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
type:ImageObject
type:Organization
author:
name:Mateo de las Casas-Engel
affiliation:
name:Consejo Superior de Investigaciones Científicas (CSIC)
address:
name:Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain
type:PostalAddress
type:Organization
type:Person
name:Angel L. Corbí
affiliation:
name:Consejo Superior de Investigaciones Científicas (CSIC)
address:
name:Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain
type:PostalAddress
type:Organization
email:[email protected]
type:Person
keywords:Inflammation, Macrophages, Psychiatric diseases, Serotonin, Serotonin receptor
description:Macrophages display a ample plethora of effector functions whose acquisition is promoted by the surrounding cytokine and cellular environment. Depending on the stimulus, macrophages become specialized (“polarized”) for either pathogen elimination, tissue repair and wound healing or immunosuppression. This “polarization” versatility allows macrophages to critically contribute to tissue homeostasis, as they promote initiation and resolution of inflammatory responses. As a consequence, deregulation of the tissue macrophage polarization balance is an etiological agent of chronic inflammation, autoimmune diseases, cancer and even obesity and insulin resistance. In the present review we describe current concepts on the molecular basis and the patho-physiological implications of macrophage polarization, and describe its modulation by serotonin (5-HT), a neurotransmitter that regulates inflammation and tissue repair via a large set of receptors (5-HTR1-7). 5-HT modulates the phenotypic and functional polarization of macrophages, and contributes to the maintenance of an anti-inflammatory state mainly via 5-HTR2B and 5-HTR7, whose activation has a great impact on macrophage gene expression profile. The identification of 5-HTR2B and 5-HTR7 as functionally-relevant polarization markers suggests their therapeutic value in inflammatory pathologies as well as their potential involvement in linking the immune and nervous systems.
datePublished:2014
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Book:
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isbn:
978-3-319-07320-0
978-3-319-07319-4
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name:Springer International Publishing
logo:
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type:PostalAddress
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address:
name:Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain
type:PostalAddress
type:Organization
name:Angel L. Corbí
affiliation:
name:Consejo Superior de Investigaciones Científicas (CSIC)
address:
name:Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain
type:PostalAddress
type:Organization
email:[email protected]
PostalAddress:
name:Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain
name:Centro de Investigaciones Biológicas, Consejo Superior de Investigaciones Científicas (CSIC), Madrid, Spain
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