Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4745266/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4745266/. The analysis below is for the second page.

Title[redir]:
Clearance Deficiency and Cell Death Pathways: A Model for the Pathogenesis of SLE - PMC
Description:
Alterations of cell death pathways, including apoptosis and the neutrophil specific kind of death called NETosis, can represent a potential source of autoantigens. Defects in the clearance of apoptotic cells may be responsible for the initiation of ...

Matching Content Categories {📚}

  • Science
  • Education
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We're unsure if the website is profiting.

Websites don't always need to be profitable; some serve as platforms for education or personal expression. Websites can serve multiple purposes. And this might be one of them. Ncbi.nlm.nih.gov has a secret sauce for making money, but we can't detect it yet.

Keywords {🔍}

sle, google, scholar, pubmed, doi, cells, lupus, apoptotic, patients, cell, clearance, systemic, pmc, article, free, erythematosus, nets, antibodies, death, autoantibodies, neutrophils, immunol, deficiency, autoantigens, disease, ifnα, crp, activity, apoptosis, immune, blood, macrophages, netosis, dnase, neutrophil, necrotic, chromatin, arthritis, impaired, complement, rheum, inflammation, proteins, dying, autoimmunity, production, human, extracellular, phagocytes, autoimmune,

Topics {✒️}

anti-sjögren’s-syndrome-related antigen song-chou hseih c-ly tyrosine kinase c-mer dna/anti-dna/ll37 complexes circulate monomeric c-reactive protein interferon-alpha mediates suppression pmc beta search phagocytose ig-coated beads pinto-da-silva lh crp/anti-crp antibodies assembly power – cell death induced foe – netting neutrophils immobilize opsonizing anti-beta2-glycoprotein dnase i-dependent degradation anti-neutrophil cytoplasmic antibody fc-receptor-mediated engulfment anti-neutrophil cytoplasmic antibodies ifn-α induced genes sle produce ifn-α apoptotic human t-cells surface code – biophysical signals cell-dependent immune response anti-ro/anti-ssa necrotic cell-derived chromatin subsequent anti-inflammatory response c-reactive protein mfg-e8-deficient mice anti-opsonin antibodies leads anti-ro/ssa antibodies anti-serum amyloid component chronic polyarthritis caused sphingosine-1-phosphate continually induces local stimulate macrophage chemotaxis double-stranded dna double stranded dna reported interferon-α activity receptor 9-dependent activation foster ifn-α production autoantibodies ifn-α signature normal opsonin-mediated clearance autophagy-related genes proinflammatory cytokines il-17a autophagy-related signaling reactive oxygen species anti-dnase antibodies compared significantly lower expression license information pmcid specific morphological features albumin-coated beads

Questions {❓}

  • Are they specific tools for the diagnosis of aPL syndrome?
  • Autoimmunity and glomerulonephritis in mice with targeted deletion of the serum amyloid P component gene: SAP deficiency or strain combination?
  • Interferon-alpha mediates suppression of C-reactive protein: explanation for muted C-reactive protein response in lupus flares?
  • Neutrophil extracellular traps: is immunity the second function of chromatin?

External Links {🔗}(273)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

4.01s.