Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC4620423/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC4620423/. The analysis below is for the second page.

Title[redir]:
Common stemness regulators of embryonic and cancer stem cells - PMC
Description:
Pluripotency of embryonic stem cells (ESCs) and induced pluripotent stem cells is regulated by a well characterized gene transcription circuitry. The circuitry is assembled by ESC specific transcription factors, signal transducing molecules and ...

Matching Content Categories {📚}

  • Education
  • Science
  • Telecommunications

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't see how the site brings in money.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Ncbi.nlm.nih.gov has a revenue plan, but it's either invisible or we haven't found it.

Keywords {🔍}

doi, google, scholar, pubmed, cells, stem, cell, cancer, pmc, article, free, embryonic, differentiation, cscs, signaling, pluripotency, expression, human, selfrenewal, escs, oct, breast, nanog, sox, csc, role, mouse, tumor, genes, wang, dna, transcription, factors, notch, biol, res, development, nature, pathway, klf, prostate, methylation, epigenetic, proliferation, histone, nat, wnt, pluripotent, inhibition, tet,

Topics {✒️}

epcam+ esa+cd44+cd24-/lowlin- phenotype o-linked n-acetylglucosamine transferase tgf-βr ii-null phenotype[156] cbp/β-catenin antagonist icg-001 integrin/focal adhesion kinase tgf-beta-receptor-mediated signaling wnt/beta-catenin signaling pathway sry-related hmg-box factors integrin/fak-dependent tumorigenic effects activate wnt/β-catenin signaling tgfβ-sox4-sox2 signaling appears tgfbeta/activin-mediated smad signaling human colon-cancer-initiating cells germ-line-competent embryonic stem beta-catenin regulates expression pmc beta search tgf-β/activin/nodal pathway sry-related hmg box wnt/β-catenin signalling ser/thr kinase type stage-specific embryonic antigen tgf-β agonists/antagonists[133] histone k4 tri-methyltransferase anti-cancer-stem-cell therapy o-linked n-acetylglucosaminetransferase restriction enzyme digestion-mediated tgf-β cues underlie β-catenin-dependent transcription tyrosine kinase c-met cd44⁺cd24⁻ stem cell lipoprotein receptor-related protein wnt/β-catenin signaling janus-family tyrosine kinase fgf-induced erk1/2 signalling probing transcription-specific outputs wnt/β-catenin branch paracrine fgf/tbx3 signaling tet-family-dependent chromatin remodeling stage-specific embryonic antigens wnt/β-catenin pathway active β-catenin interacts induces epithelial-mesenchyme transition sonic hedgehog-induced medulloblastomas c-kit proto-oncogene[62] tumor-initiating stem cells g-protein-coupled receptor methylated dna immunoprecipitation-chip breast tumor-initiating cells β-catenin promote exit mmtv-wnt1 transgenic mice[102]

Questions {❓}

  • CD133: to be or not to be, is this the real question?
  • Can cancer cells be reprogrammed to pluripotency and then induced to differentiate?
  • Cancer: pathological nuclear reprogramming?
  • PRC1 complex diversity: where is it taking us?
  • Pharmacogenomics and cancer stem cells: a changing landscape?
  • Tackling the cancer stem cells - what challenges do they pose?
  • Targeting Wnt signaling: can we safely eradicate cancer stem cells?
  • Wnt: what’s needed to maintain pluripotency?
  • P63 and the epithelial stem cell: more than status quo?

Social Networks {👍}(6)

External Links {🔗}(956)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Knockout.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

6.85s.