Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers

We began analyzing http://genesdev.cshlp.org/content/27/7/725, but it redirected us to http://genesdev.cshlp.org/content/27/7/725. The analysis below is for the second page.

Title[redir]:
MYC/MAX control ERK signaling and pluripotency by regulation of dual-specificity phosphatases 2 and 7
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields

Matching Content Categories {๐Ÿ“š}

  • Science
  • Education
  • News & Politics

Content Management System {๐Ÿ“}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of doi.org audience?

๐Ÿ™๏ธ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 75,489,999 visitors per month in the current month.

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How Does Doi.org Make Money? {๐Ÿ’ธ}

We see no obvious way the site makes money.

Some websites aren't about earning revenue; they're built to connect communities or raise awareness. There are numerous motivations behind creating websites. This might be one of them. Doi.org might be cashing in, but we can't detect the method they're using.

Keywords {๐Ÿ”}

erk, dusp, author, article, signaling, myc, submit, mycmax, pluripotency, activity, online, copyright, cold, spring, harbor, laboratory, press, home, subscribe, advertise, info, archive, contact, search, dualspecificity, phosphatases, georgia, maintenance, pluripotent, max, mechanism, complexes, binding, resulting, loss, mapkerk, pathway, email, supplemental, material, facebook, full, articles, issue, issn, skip, main, page, content, keyword,

Topics {โœ’๏ธ}

supplemental material myc/max complexes achieve extracellular signal-regulated kinase mapk/erk signaling pathway myc-binding partner max mapk/erk pathway /stat3 signaling reaches authors contributed equally dual-specificity phosphatase dephosphorylating residues required leukemia inhibitory factor org/cgi/doi/10 dual-specificity phosphatases 2 psc maintenance involving erk activity article doi 0890-9369 online issn article ยป table catalytic activity issue alert ectopic dusp2 max article erk search 7 james chappell1 yuhua sun1 amar singh molecular biology coverdell center health sciences abstract suppression absolute requirement transcriptionally regulating dusps function inactivating erk1 dusp7 transcriptional activation findings elucidate regulatory role directly inhibit accepted march 18 copyright ยฉ 2013 details tweeted myc signaling sustain pluripotency dusp2 online maintenance

Questions {โ“}

  • What's this?

External Links {๐Ÿ”—}(89)

Analytics and Tracking {๐Ÿ“Š}

  • Facebook Pixel
  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {๐Ÿ“š}

  • jQuery

Emails and Hosting {โœ‰๏ธ}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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