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Keywords {🔍}

bmi, cells, doi, cancer, breast, pubmed, google, scholar, expression, stem, cell, nanog, figure, selfrenewal, pmc, knockdown, overexpression, article, stemness, nfκb, vector, free, control, mammary, epithelial, tumor, activity, normal, genes, pathway, properties, emt, role, study, luciferase, reduced, showed, mdamb, tissues, human, promoter, increased, primary, drug, shown, assay, levels, analysis, nblelp, regulating,

Topics {✒️}

twist1-induced epithelial-mesenchymal transition ikk-nuclear factor-kappab pathway nf-kappab/mmp-9 signaling pathway electronic supplementary material semi-quantitative rt-pcr analysis dual-luciferase assay kit inhibiting c-myc-induced apoptosis dual-luciferase assay showed late passage cells 25348805 abstract background nfκb promoter-luciferase plasmid pmc beta search induces epithelial-mesenchymal transition anti-α-tubulin antibody hrp-coupled secondary antibodies rt-pcr analysis showed pre-publication history ras/mapk signaling pathways promoter reporter assay activating nf-kappab signaling dbt-iisc partnership programme bd facs-aria ii ultra-low attachment plates undertook shrna-mediated knockdown license information pmcid chose nble-lp cells dual luciferase assay mu-myc transgenic mice nuclear factor-kappab providing pbabepuro-bmi1 construct figure 2b-left panel c-myc cooperating oncogene graphs showing fold supplementary materials 12885_2014_4966_moesm1_esm acute myeloid leukemia nanog promoter activity supplementary material stem cell factor embryonic stem cell nat rev cancer discussion bmi1 epithelial-mesenchymal transition firefly luciferase activity real time pcr mammary stem cell induced mesenchymal morphology nfκb promoter activity mammary epithelial cells bmi1 positively regulates ink4a-arf locus expression

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