
DOI . ORG {
}
Title[redir]:
Bmi-1 collaborates with c-Myc in tumorigenesis by inhibiting c-Myc-induced apoptosis via INK4a/ARF
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields
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Keywords {🔍}
bmi, inkaarf, apoptosis, author, tumorigenesis, submit, cmyc, myc, cold, spring, harbor, laboratory, press, article, home, subscribe, advertise, info, archive, contact, search, collaborates, cmycinduced, division, molecular, genetics, netherlands, oncogenes, lymphomagenesis, collaboration, critical, ability, induction, parf, heterozygosity, eμmyc, enhancing, observe, email, full, articles, issue, online, issn, skip, main, page, content, keyword, advanced,
Topics {✒️}
wild-type ink4a–arf allele inhibiting c-myc-induced apoptosis enhancing c-myc-induced apoptosis ink4a–arf dependent manner prohibiting myc-mediated induction issue alert ink4a/arf jacqueline induce apoptosis depends observe increased apoptosis dose-dependent regulator eμ–myc mice bmi-1−/− lymphoid organs eμ–myc transgene article » table jan-willem voncken primary embryo fibroblasts authors contributed equally bmi-1 inhibits lymphomagenesis netherlands cancer institute critical downstream target observe strong collaboration c-myc ink4a–arf 0890-9369 online issn author bmi-1 collaborates enhancing proliferation bmi-1 underlies prevent tumorigenesis netherlands abstract murine lymphomagenesis critical role apoptosis blanca scheijen karin kieboom anton berns molecular carcinogenesis molecular genetics biomedical genetics 1066 cx amsterdam recently identified results reinforce turn acts received august 3 accepted september 3 division bmi-1 myc' myc tumorigenesis
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