Here's how DOI.ORG makes money* and how much!

*Please read our disclaimer before using our estimates.
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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. Hosting Providers

We began analyzing https://genesdev.cshlp.org/content/13/20/2678, but it redirected us to https://genesdev.cshlp.org/content/13/20/2678. The analysis below is for the second page.

Title[redir]:
Bmi-1 collaborates with c-Myc in tumorigenesis by inhibiting c-Myc-induced apoptosis via INK4a/ARF
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields

Matching Content Categories {📚}

  • Science
  • Education
  • Careers

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🚄 Respectable Traffic: 10k - 20k visitors per month


Based on our best estimate, this website will receive around 10,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Doi.org Make Money? {💸}

The income method remains a mystery to us.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Doi.org might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

bmi, inkaarf, apoptosis, author, tumorigenesis, submit, cmyc, myc, cold, spring, harbor, laboratory, press, article, home, subscribe, advertise, info, archive, contact, search, collaborates, cmycinduced, division, molecular, genetics, netherlands, oncogenes, lymphomagenesis, collaboration, critical, ability, induction, parf, heterozygosity, eμmyc, enhancing, observe, email, full, articles, issue, online, issn, skip, main, page, content, keyword, advanced,

Topics {✒️}

wild-type ink4a–arf allele inhibiting c-myc-induced apoptosis enhancing c-myc-induced apoptosis ink4a–arf dependent manner prohibiting myc-mediated induction issue alert ink4a/arf jacqueline induce apoptosis depends observe increased apoptosis dose-dependent regulator eμ–myc mice bmi-1−/− lymphoid organs eμ–myc transgene article » table jan-willem voncken primary embryo fibroblasts authors contributed equally bmi-1 inhibits lymphomagenesis netherlands cancer institute critical downstream target observe strong collaboration c-myc ink4a–arf 0890-9369 online issn author bmi-1 collaborates enhancing proliferation bmi-1 underlies prevent tumorigenesis netherlands abstract murine lymphomagenesis critical role apoptosis blanca scheijen karin kieboom anton berns molecular carcinogenesis molecular genetics biomedical genetics 1066 cx amsterdam recently identified results reinforce turn acts received august 3 accepted september 3 division bmi-1 myc' myc tumorigenesis

Questions {❓}

  • What's this?

External Links {🔗}(84)

Analytics and Tracking {📊}

  • Facebook Pixel
  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery

Emails and Hosting {✉️}

Mail Servers:

  • mx.zoho.eu
  • mx2.zoho.eu
  • mx3.zoho.eu

Name Servers:

  • josh.ns.cloudflare.com
  • zita.ns.cloudflare.com
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