Here's how NCBI.NLM.NIH.GOV makes money* and how much!

*Please read our disclaimer before using our estimates.
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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC3660717/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC3660717/. The analysis below is for the second page.

Title[redir]:
The Role of Estrogens in Control of Energy Balance and Glucose Homeostasis - PMC
Description:
Estrogens play a fundamental role in the physiology of the reproductive, cardiovascular, skeletal, and central nervous systems. In this report, we review the literature in both rodents and humans on the role of estrogens and their receptors in the ...

Matching Content Categories {📚}

  • Health & Fitness
  • Science
  • Fitness & Wellness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We find it hard to spot revenue streams.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Ncbi.nlm.nih.gov has a revenue plan, but it's either invisible or we haven't found it.

Keywords {🔍}

google, scholar, pubmed, doi, estrogen, erα, insulin, muscle, receptor, pmc, free, article, mice, women, diabetes, resistance, tissue, expression, skeletal, glucose, adipose, energy, metabolism, metabolic, effects, action, estrogens, fat, postmenopausal, effect, body, endocrinol, increased, neurons, physiol, glut, therapy, intake, protein, female, sensitivity, food, obesity, regulation, activation, liver, homeostasis, lipid, cell, role,

Topics {✒️}

pro-opiomelanocortin peroxisome proliferator-activated receptor high-fat-diet-induced insulin resistance suppress srebp-1c gene c-jun-n-terminal kinase nucleus tractus solitarius g-protein-coupled estrogen receptor srebp-1c mrna amp-activated protein kinase high-fat diet-fed mice agouti-related peptide stz-induced β-cell destruction srebp-1c protein-coupled estrogen receptor diet-induced insulin resistance oxidized ldl-induced expression low-density lipoprotein signal transducer tissue-selective estrogen complex gq-coupled membrane receptor estrogen response element lipogenesis including acetyl-coenzyme ligand-activated er dissociates ligand-activated transcription factors conjugated equine estrogen high fat diet atp-binding cassette transporter functional mef2 element contraction-stimulated glucose uptake oestrogen receptor-α plays chaperone heat-shock protein protein-gated inwardly rectifying macrophage ppar γ 17β-estradiol-induced killing age-matched wild-type counterparts estrogen receptor-α genotypes insulin-stimulated glucose uptake insulin-stimulated glucose disposal estradiol-induced insulin secretion preventing stz-induced diabetes stz-induced diabetic rats lipoprotein lipase gene muscle-atrophy ubiquitin ligases β-cell destruction induced hepatic glucose production sex hormone-binding globulin conjugate binds glp-1r extranuclear estrogen receptor-α activating glp-1r signaling high-impact physical exercise

Questions {❓}

  • Chief among these is how can we enhance the metabolic actions of E2 in chronic therapies aimed at prevention and treatment of metabolic diseases?
  • Does obesity arise from loss of ERα within adipocytes in particular, or can it be driven as a secondary phenotype resulting from a loss of ERα in brain, skeletal muscle, liver, or even in selective immune cells?
  • Removal of visceral fat prevents insulin resistance and glucose intolerance of aging: an adipokine-mediated process?
  • Skeletal muscle triglycerides, diacylglycerols, and ceramides in insulin resistance: another paradox in endurance-trained athletes?

Social Networks {👍}(6)

External Links {🔗}(712)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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