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NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Social Networks
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. Hosting Providers
  14. CDN Services

We began analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC11190820/, but it redirected us to https://pmc.ncbi.nlm.nih.gov/articles/PMC11190820/. The analysis below is for the second page.

Title[redir]:
Understanding the complexity of p53 in a new era of tumor suppression - PMC
Description:
p53 was discovered 45 years ago as a SV40 large T antigen binding protein, coded by the most frequently mutated TP53 gene in human cancers. As a transcription factor, p53 is tightly regulated by a rich network of post-translational modifications to ...

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of ncbi.nlm.nih.gov audience?

🚀🌠 Tremendous Traffic: 10M - 20M visitors per month


Based on our best estimate, this website will receive around 10,538,143 visitors per month in the current month.

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How Does Ncbi.nlm.nih.gov Make Money? {💸}

We can't see how the site brings in money.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Ncbi.nlm.nih.gov could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

google, scholar, doi, pubmed, pmc, article, free, cell, cancer, tumor, cells, mutant, activity, suppression, mutations, nat, gene, protein, functions, dna, nature, function, activation, suppressor, targeting, role, regulation, mdm, apoptosis, ferroptosis, genes, mol, wang, normal, stem, mouse, liu, target, expression, metabolic, mice, response, pathway, biol, transcription, damage, targets, development, inhibition, aging,

Topics {✒️}

p53-mediated anti-tumor activity p53-mediated cell-cycle arrest pmc beta search monocytic antigen-presenting cells fine-tuning differentiation route p53 prevents r-loop c-terminal regulatory domain autologous tumor-infiltrating lymphocytes sirtuin1-p53 regulatory axis p53-mediated pro-survival activity t-cell receptor-engineered brd4-dependent csf-1 expression tumor-suppressive immune microenvironment p53-derived peptide-mhcs anti-tumor immune response blocking e6-mediated degradation p53-dependent metabolic remodelling e1b gene-attenuated adenovirus stapled α-helical peptide chromosomal-instable stem cells p53-mediated ferroptosis caused tumor-suppressive immune network uninfected embryonal carcinoma-cells full-length p53 protein p53-mediated tumor suppression anti-apoptotic survivin gene p53-dependent tumor suppression c-terminal lysine residues central dna-binding domain potent tumor-suppressive capabilities compromising anti-tumor efficacy n-terminal transactivation domain α-ketoglutarate links p53 p53–5kr mutants recapitulates p53-mediated senescence impairs p53-mediated tumour suppression inhibiting mdm2-mediated degradation exhibits tf-independent function specific response element genome-wide cheater screen inducing puma-dependent suppression carcinogen-induced colorectal tumors induce anti-tumor activity tumor type-specific studies bispecific antibody h2-scdb cell type-specific delivery small-molecule p53 activator p53-null female mice cell-free dna activates p53-dependent apoptosis

Questions {❓}

  • 105,142,296 So how does basal p53 work in unstressed cells?
  • 196,197,216,217 A tightly relevant question is, among the LOF, DNE, and GOF of p53 alteration, which is the predominant mechanism underpinning p53-related tumor initiation and progression?
  • 317–320 What are the unidentified targets and functions of p53?
  • Ageing - The price of tumour suppression?
  • Are the non-cell-autonomous activities of p53 viable targets for cancer treatment?
  • Considering the frequent side effects, are there other elements in the p53 pathway that could serve as safer targets than MDM2, without compromising anti-tumor efficacy?
  • Fifthly, is targeting p53 truly a practical approach for treating cancers?
  • Firstly, what is the function of basal p53?
  • Fourthly, what are the underpinning mechanisms and biological relevance of the context-dependent activity of p53?
  • P53 Dysfunction in Neurodegenerative Diseases - The Cause or Effect of Pathological Changes?
  • Secondly, what is the contribution of p53’s repressive target genes to its function?
  • Thirdly, are genetically engineered mouse models (GEMMs) sufficiently effective for p53 research?

Social Networks {👍}(6)

External Links {🔗}(652)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)

Emails and Hosting {✉️}

Mail Servers:

  • nihcesxway.hub.nih.gov
  • nihcesxway2.hub.nih.gov
  • nihcesxway3.hub.nih.gov
  • nihcesxway4.hub.nih.gov
  • nihcesxway5.hub.nih.gov

Name Servers:

  • dns1-ncbi.ncbi.nlm.nih.gov
  • dns2-ncbi.ncbi.nlm.nih.gov
  • lhcns1.nlm.nih.gov
  • lhcns2.nlm.nih.gov

CDN Services {📦}

  • Ncbi

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