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PMC . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
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  4. Monthly Traffic Estimate
  5. How Does Pmc.ncbi.nlm.nih.gov Make Money
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We are analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC5929477/.

Title:
The parabrachial nucleus: CGRP neurons function as a general alarm - PMC
Description:
The parabrachial nucleus (PBN), which is located in the pons and dissected by one of the major cerebellar output tracks, is known to relay sensory information (visceral malaise, taste, temperature, pain, itch) to forebrain structures including the ...
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Education
  • Science
  • Fitness & Wellness

Content Management System {📝}

What CMS is pmc.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pmc.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pmc.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Pmc.ncbi.nlm.nih.gov Make Money? {💸}

The income method remains a mystery to us.

Earning money isn't the goal of every website; some are designed to offer support or promote social causes. People have different reasons for creating websites. This might be one such reason. Pmc.ncbi.nlm.nih.gov has a secret sauce for making money, but we can't detect it yet.

Keywords {🔍}

doi, neurons, pubmed, google, scholar, cgrppbn, pmc, article, free, pbn, mice, parabrachial, nucleus, neurosci, agrp, lateral, pain, activation, amygdala, cgrp, brain, anorexia, food, receptor, feeding, sensory, responses, expression, gene, nat, response, circuit, activated, taste, appetite, cell, starvation, calcitonin, rat, regions, nts, neuron, activate, signaling, nature, palmiter, specific, fos, jax, visceral,

Topics {✒️}

calcitonin gene-related peptide calcitonin gene-related peptide pro-opiomelanocortin deficiency responds calcitonin gene-related protein pmc beta search initiate counter-regulatory responses melanocortin-4 receptor-regulated appetite making cre-driver lines express agouti-related protein cre-dependent gene encoding powerful gene-targeting strategies cbln4-ires-flp-cre glucose counter-regulatory signals superfamily cytokine mic-1/gdf15 high-throughput cre reporting cre-driver mouse lines crhr1-ires-flp-cre g-protein-coupled hard-wired neural circuits tissue-specific rna processing provide long-term control npy/agrp/gaba-expressing neurons adjacent kölliker-fuse nucleus allowed cre-dependent expression kölliker-fuse/parabrachial complex conditioned-taste aversion conditioned taste aversion gut-brain glucose signaling spino-parabrachial-amygdaloid pathway [11] interacting appetite-regulating pathways dbh-expressing neurons reside neighboring kölliker-fuse suggests cell-type-specific isolation conditioned taste aversions agouti-related protein excitatory hindbrain-forebrain communication bitter-responsive gustatory neurons concluding remarks parabrachial cgrp-expressing neurons hypercapnia-responsive parabrachial neurons photo-inactivation studies revealed situ hybridization techniques pbn cold-responsive neurons cre-driver lines agrp→cgrp neuron circuit vagal→nts→cgrppbn circuit cre-dependent channelrhodopsin establish robust foot-shock superior cerebellar peduncle positive-valence mechanism

Questions {❓}

  • Are the inputs to each of those brain regions coordinated to produce optimal behavioral responses?
  • Are there PBN neurons that relay the pleasurable signals and, if so, what are their downstream targets?
  • Are there additional PBN neurons that relay threat signals and, if so, how do they differ from the CGRPPBN neurons?
  • Can pharmacological approaches be developed that target the CGRPPBN to the extended amygdala circuit to treat eating, pain and/or anxiety disorders?
  • Do neurons within the PBN communicate with each other, either directly, or via feedback loops, and if so, for what purpose?
  • If CGRPPBN neurons are blind to the source of the excitatory input and thus serve as a general alarm to alert mammals of real or potential threats, then how do mammals mount appropriate behavioral and/or physiological responses to specific threats?
  • In particular, how do sensory cues that were associated with pain activate CGRPPBN neurons after conditioning?
  • The question then became, why do mice starve after AgRP-neuron ablation?
  • What is the molecular identity of PBN neurons that relay each of the five taste sensations to the thalamus?

Social Networks {👍}(6)

External Links {🔗}(426)

Analytics and Tracking {📊}

  • Google Analytics
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Libraries {📚}

  • jQuery
  • jQuery module (jquery-3.6.0)
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Emails and Hosting {✉️}

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CDN Services {📦}

  • Ncbi

3.99s.