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cells, cancer, don, treatment, pubmed, article, pancreatic, cell, google, scholar, gemr, glutamine, proteins, protein, miapaca, cas, glycosylation, signaling, hours, fig, treated, pathways, analysis, egfr, proliferation, ros, resistance, untreated, data, cellular, exosomes, gem, drug, disruption, exosome, central, gemcitabine, pathway, including, nature, metabolic, redox, metabolism, expression, tumor, showed, glycan, bead, proteomics, biosynthesis,

Topics {✒️}

nature portfolio scientific instrument services activating hab18g/cd147-egfr-pstat3 signaling privacy policy phospho-stat3 kits advertising nature 496 nature 316 nature chemical tools software solutions reprints author information authors ubiquitin-mediated proteasomal degradation drug-resistant human adenocarcinoma pancreatic ductal adenocarcinoma sensitize chemo-resistant pdac 6-diazo-5-oxo-l-norleucine alexa568 goat anti-rabbit stable-isotope dimethyl labeling disrupting n-linked glycosylation original author serum-free media atop gemcitabine-metal–organic frameworks n-linked glycosylation sites serine/threonine-protein kinases provide udp-n-acetylglucosamine receptor tyrosine kinases phospho-p44/p42 mapk vivo drug-induced apoptosis 100 mg/ml fibronectin overnight tandem mass spectrometry thermo fisher scientific mass spectrometry analysis permissions smad3/cmyc axis key gemcitabine-metabolising enzyme potential anti-cancer activity kras-regulated metabolic pathway hexosamine biosynthetic pathway chemoresistant pancreatic cancer full size image trans-proteomic pipeline egfr-related signaling pathways suppress udp-glcnac generation phospho-s6 ribosomal protein site-specific glycosylation occupancy mass spectrometric data proteomics data analysis pancreatic cancer survival

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• ER stress and hexosamine pathway during tumourigenesis: A pas de deux?
• Reactive oxygen species and cancer paradox: To promote or to suppress?

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