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article, google, scholar, cas, cell, nature, jnk, histone, phosphorylation, biol, access, stem, cells, kinase, protein, data, kinases, development, content, nat, mol, research, map, open, embryonic, cookies, differentiation, signaling, transcription, nfy, regulation, science, manuscript, privacy, function, analysis, stadler, schübeler, pathway, neurons, chromatin, expression, genes, gene, signal, genet, sci, basel, european, information,

Topics {✒️}

nature portfolio permissions reprints privacy policy open access database nf-κb–dependent gene expression open software development c-jun nh2-terminal kinase author information authors c-jun n-terminal kinases advertising cytokine-induced gene expression european research council social media novartis research foundation jun n-terminal kinase dominant-negative nf-ya reduces nature 461 nature 438 nature 423 nature tools lineage-specific polycomb targets italy biomedical research mitogen-activated protein kinases stress-activated kinase 1/2 msk-dependent h3k27me3s28 phosphorylation cell type-dependent control transcription factor-binding profiles map kinase–mediated phosphorylation research springerlink instant access myc-dependent transcriptional activation genome-wide location analysis mitogen-activated protein permissions hp1-chromatin binding p38 mapk pathways annexin a1 expression stem cell differentiation quantitative genomics facility friedrich miescher institute iκb kinase-α drosophila thorax development early embryonic neurogenesis privacy scaffold protein revealed dominant-negative analogs transcription factor nf short dna sequences

Questions {❓}

• AP-1—the Jun proteins: oncogenes or tumor suppressors in disguise?
• Histone H3 phosphorylation: universal code or lineage specific dialects?
• Phosphorylation of serine 10 in histone H3, what for?

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