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Title:
The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins - PubMed
Description:
Early in programmed cell death (apoptosis), mitochondrial membrane permeability increases. This is at least in part due to opening of the permeability transition (PT) pore, a multiprotein complex built up at the contact site between the inner and the outer mitochondrial membranes. The PT pore has be …
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Keywords {🔍}
doi, mitochondrial, pore, pmid, apoptosis, kroemer, pubmed, proteins, pmc, permeability, caspases, zamzami, cell, bcl, susin, figure, transition, complex, membrane, protein, free, article, metabolism, articles, cited, mesh, exp, med, marzo, brenner, death, liposomes, ptpc, effect, review, main, ncbi, figures, similar, references, information, resources, regulation, reed, early, programmed, opening, calcium, reconstituted, mitochondria,
Topics {✒️}
proto-oncogene bcl-2 main page content bcl-2-related proteins mutated bcl-2 proteins similar articles cited adenine nucleotide translocator mesh keyword voltage-dependent anion channel literature sources similar articles articles references zamzami intact mitochondria proteins isolated mitochondria liposomes exhibit bcl-2-inhibited effect apoptosis-related endoproteases bcl-xl augment permeability transition apoptotic program liposomes purified protein complexes mouse bcl2l1 protein programmed cell death programmed lymphocyte death lonidamine triggers apoptosis pt pore complex apoptosis-inhibitory effect thiol-reactive agents critical thiol residue outer mitochondrial membranes multiprotein complex built resources mitochondrial potential constitutes mitochondrial transmembrane potential bcl-xl cell-free extracts united states government reactive oxygen species functional behavior comparable dimensional gel electrophoresis biochem soc symp de pablo ma early irreversible step books pubchem compound pt modulatory capacity reconstituted ptpc pores recombinant bcl-2 bcl-2 family figures
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