Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/19706792/.

Title:
Apoptotic signaling activated by modulation of the F0F1-ATPase: implications for selective killing of autoimmune lymphocytes - PubMed
Description:
7-Chloro-5-(4-hydroxyphenyl)-1-methyl-3-(napthalen-2-ylmetyl)-4,5,-dihydro-1H-benzo[b][1,4]diazepin-2(3H)-one (Bz-423) is a proapoptotic 1,4-benzodiazepine that potently suppresses disease in the murine model of lupus by selectively killing pathogenic lymphocytes. In MRL/MpJ-Fas(lpr) (MRL-lpr) mice, …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Science
  • Health & Fitness
  • Education

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We're unsure how the site profits.

While profit motivates many websites, others exist to inspire, entertain, or provide valuable resources. Websites have a variety of goals. And this might be one of them. Pubmed.ncbi.nlm.nih.gov might be cashing in, but we can't detect the method they're using.

Keywords {🔍}

doi, pmid, pmc, pubmed, free, opipari, glick, cells, apoptosis, article, epub, mitochondrial, cell, bak, mice, death, kinase, fig, nih, states, signaling, selective, mrllpr, protein, superoxide, bzinduced, activation, articles, mesh, apoptotic, autoimmune, lymphocytes, benzodiazepine, disease, signals, effects, review, hhsunited, main, ncbi, similar, cited, references, resources, aug, activated, ffatpase, wahl, lupus, pathogenic,

Topics {✒️}

united states government oligomycin-sensitivity-conferring protein component funding linkout jun n-terminal kinase pubchem substance grants t-leukemic cell lines antiapoptotic phosphatidylinositol 3-kinase main page content disease-selective effects similar articles cited arrests graft-versus-host disease t-cell activation resources hhs bz-423 inactivates akt autoimmune lymphocytes thomas antiapoptotic pi3k-akt signaling bz-423-induced superoxide contributes mesh keyword cell death articles references astoul pathogenic b220+ double-negative autoimmune mrl/lpr mice fas death receptor jurkat cells bz-423-induced bak activation cell leukemia line mol cell cardiol cell apoptosis fas-deficient mice sensitize autoreactive cd4 lymphocytes mrl-lpr cd4 mrl-lpr mice akt signaling lyssiotis ca byersdorfer ca bak-dependent apoptosis permeability transition pore jurkat cell-type death mechanism immunomodulatory benzodiazepine bz-423 pubmed wordmark pubmed logo bz-423-induced sci transl med bz-423 activates bax mrl/mpj-fas dihydro-1h-benzo[

Social Networks {👍}(6)

External Links {🔗}(77)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery
  • PhotoSwipe

CDN Services {📦}

  • Ncbi

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