Here's how PUBMED.NCBI.NLM.NIH.GOV makes money* and how much!

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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/18440987/.

Title:
Mitochondrial fission mediates high glucose-induced cell death through elevated production of reactive oxygen species - PubMed
Description:
These findings indicate that the fission-mediated fragmentation of mitochondrial tubules is causally associated with enhanced production of mitochondrial ROS and cardiovascular cell injury in hyperglycaemic conditions.
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {πŸ“š}

  • Science
  • Health & Fitness
  • Fitness & Wellness

Content Management System {πŸ“}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {πŸ“ˆ}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {πŸ’Έ}

We don’t know how the website earns money.

The purpose of some websites isn't monetary gain; they're meant to inform, educate, or foster collaboration. Everyone has unique reasons for building websites. This could be an example. Pubmed.ncbi.nlm.nih.gov could be getting rich in stealth mode, or the way it's monetizing isn't detectable.

Keywords {πŸ”}

mitochondrial, doi, fission, cell, high, pmid, pmc, glucose, pubmed, mitochondria, free, article, death, reactive, oxygen, species, ros, conditions, nih, metabolism, states, role, cells, sustained, figure, heart, epub, cardiovascular, drug, hhsunited, main, articles, mesh, jul, production, yoon, morphology, levels, protein, increased, cardiac, kim, effects, ncbi, conflict, interest, similar, cited, references, types,

Topics {βœ’οΈ}

united states government hhs main page content funding linkout reactive oxygen species hyperglycaemia-induced cell injury inhibiting mitochondrial fission stem-cell-derived cardiomyocytes animal dose-response relationship resources cardiovasc res literature sources similar articles cited cellular ros levels pubchem substance grants fission protein dlp1 drug dynamin mesh keyword fission-mediated fragmentation / metabolism endothelium promoting mitochondrial fission high glucose mitochondrial cell death cardiovascular cell injury subsequent cell death mitochondrial permeability transition physiol endocrinol metab mol biol cell pubmed bereiter-hahn protein dlp1 shey-shing sheu antioxid redox signal atrophied skeletal muscle leduc-gaudet jp int rev cytol heart fail rev evaluated mitochondrial morphology st-jean pelletier st-pierre dh mitochondrial fission maintaining tubular mitochondria glucose transport cultured disease models cell death mitochondrial ros mitochondrial tubules mitochondrial dysfunction aged mice increased production bovine cells cells incubated

External Links {πŸ”—}(64)

Analytics and Tracking {πŸ“Š}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {πŸ“š}

  • Clipboard.js
  • jQuery
  • PhotoSwipe

CDN Services {πŸ“¦}

  • Ncbi

3.8s.