PUBMED . NCBI . NLM . NIH . GOV {
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Title:
VDAC-dependent permeabilization of the outer mitochondrial membrane by superoxide induces rapid and massive cytochrome c release - PubMed
Description:
Enhanced formation of reactive oxygen species (ROS), superoxide (O2*-), and hydrogen peroxide (H2O2) may result in either apoptosis or other forms of cell death. Here, we studied the mechanisms underlying activation of the apoptotic machinery by ROS. Exposure of permeabilized HepG2 cells to O2*- eli …
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Keywords {🔍}
doi, pmid, mitochondrial, pubmed, cell, pmc, cytochrome, membrane, release, mitochondria, metabolism, apoptosis, ccr, bcl, figure, epub, death, cells, vdac, bax, family, proteins, free, article, articles, mesh, dec, permeability, oinduced, voltagedependent, anion, liposomes, caspase, main, ncbi, figures, similar, cited, references, information, resources, biol, outer, reactive, oxygen, species, ros, hydrogen, peroxide, activation,
Topics {✒️}
voltage-dependent anion channel hydrogen peroxide main page content similar articles cited literature sources bcl-2 protein family ca2+-dependent pore opening articles references adams oncogene outer mitochondrial membrane mitochondrial outer membrane mesh keyword mitochondrial membrane barrier mitochondrial membrane potential yeast mitochondria antibody vdac-dependent permeabilization permeabilized hepg2 cells cell death differ resources proapoptotic protein prevented o2--induced ccr liposomes proteins mitochondria bcl-xl bcl-2 family apoptosis-inducing activity intrinsic apoptosis pathway caspase activation hepg2 cells mitochondrial channel vdac united states government reactive oxygen species ptp blockers fail adenine nucleotide translocator fluorescent barrel-rosette antioxid redox signal species-specific control channel-forming activity cell death o2- triggers apoptosis mechanisms underlying activation o2--induced ccr o2- elicited rapid figures h2o2 promoted activation superoxide induces rapid cell biol cell survival
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