PMC . NCBI . NLM . NIH . GOV {
}
We are analyzing https://pmc.ncbi.nlm.nih.gov/articles/PMC2889094/.
Title:
Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy - PMC
Description:
Dysregulation of programmed cell death due to abnormal expression of Bcl-2 proteins is implicated in cancer, neurodegenerative diseases, and heart failure. Among Bcl-2 family members, BNip proteins uniquely stimulate cell death with features of both ...
Website Age:
27 years and 8 months (reg. 1997-10-02).
Title:
Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy - PMC
Description:
Dysregulation of programmed cell death due to abnormal expression of Bcl-2 proteins is implicated in cancer, neurodegenerative diseases, and heart failure. Among Bcl-2 family members, BNip proteins uniquely stimulate cell death with features of both ...
Website Age:
27 years and 8 months (reg. 1997-10-02).
Matching Content Categories {๐}
- Science
- Telecommunications
- Education
Content Management System {๐}
What CMS is pmc.ncbi.nlm.nih.gov built with?
Custom-built
No common CMS systems were detected on Pmc.ncbi.nlm.nih.gov, and no known web development framework was identified.
Traffic Estimate {๐}
What is the average monthly size of pmc.ncbi.nlm.nih.gov audience?
๐ Phenomenal Traffic: 5M - 10M visitors per month
Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
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How Does Pmc.ncbi.nlm.nih.gov Make Money? {๐ธ}
We're unsure if the website is profiting.
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Keywords {๐}
nix, death, cell, doi, mitochondrial, fig, apoptosis, pubmed, google, scholar, cells, pmc, reticular, caspase, article, ppif, mefs, programmed, expressing, tunel, cardiac, heart, annexin, mice, free, ablation, mptp, bnip, mitochondria, bax, bak, null, induced, pathways, failure, activation, inhibition, proteins, cytochrome, bars, labeling, transition, studies, staining, bcl, necrosis, permeability, upper, protected, apoptotic,
Topics {โ๏ธ}
org/cgi/content/full/0914013107/dcsupplemental er-nix activates bax/bak-independent conventional bax/bak-dependent apoptosis mitochondrial-free membrane fraction rabbit polyclonal anti-calnexin uniquely cyclophilin d-dependent erโmitochondrial necrotic pathway nix-cb5 expressing myocardium nix cb5-expressing cells er-mitochondrial crosstalk mediated pmc beta search nix-wt expressing mefs cells expressing nix-acta nix acta-expressing cells nix cb5-expressing hearts end-stage heart failure representative m-mode echocardiograms nix-mediated cell death er-enriched membranes labeled er-localized nix activates nix-mediated cardiac disease caspase inhibitor zvad-fmk wt nix-expressing hearts mitochondrial nix-acta hearts nix cb5-mediated opening mptp-dependent cell death organelle-specific mutants alexa fluor 546-phalloidin mitochondrial outer membranes outer membrane disruption ppif null mefs zvad-fmk attenuated annexin mptp-dependent death induced mitochondrial nix induces ฮฒ-adrenergic stimulation ฮฒ-gal control supporting information online permeability transition pore endoplasmic reticulum-mitochondria crosstalk cells expressing bax nix-expressing cells cardiac myocyte apoptosis ppif ablation eliminated cell live/dead assay mouse embryonic fibroblasts supplementary materials included nix transgenic mice assayed events leading organelle-specific effects er-mitochondrial crosstalk
Social Networks {๐}(6)
External Links {๐}(143)
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