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PUBMED . NCBI . NLM . NIH . GOV {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Pubmed.ncbi.nlm.nih.gov Make Money
  6. Keywords
  7. Topics
  8. Social Networks
  9. External Links
  10. Analytics And Tracking
  11. Libraries
  12. CDN Services

We are analyzing https://pubmed.ncbi.nlm.nih.gov/15800627/.

Title:
Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death - PubMed
Description:
Mitochondria play a critical role in mediating both apoptotic and necrotic cell death. The mitochondrial permeability transition (mPT) leads to mitochondrial swelling, outer membrane rupture and the release of apoptotic mediators. The mPT pore is thought to consist of the adenine nucleotide transloc …
Website Age:
27 years and 8 months (reg. 1997-10-02).

Matching Content Categories {📚}

  • Science
  • Telecommunications
  • Fitness & Wellness

Content Management System {📝}

What CMS is pubmed.ncbi.nlm.nih.gov built with?

Custom-built

No common CMS systems were detected on Pubmed.ncbi.nlm.nih.gov, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of pubmed.ncbi.nlm.nih.gov audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Pubmed.ncbi.nlm.nih.gov Make Money? {💸}

We can't tell how the site generates income.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Pubmed.ncbi.nlm.nih.gov might have a hidden revenue stream, but it's not something we can detect.

Keywords {🔍}

cell, death, mitochondrial, doi, pmid, cyclophilin, mice, permeability, transition, pmc, protein, metabolism, nature, mitochondria, ppif, epub, free, article, gene, cited, swelling, proteins, articles, mesh, null, oxidative, review, drug, calcium, cardiac, research, cyclophilins, main, ncbi, abstract, similar, resources, mar, role, apoptotic, membrane, isomerase, protected, fibroblasts, pubmed, shimizu, tsujimoto, stress, sep, genetics,

Topics {✒️}

tumour-necrosis factor-alpha-induced death drug-induced liver injury protein atractyloside cytochromes ischaemia/reperfusion-induced cell death main page content mitochondrial-dependent cell death drug metab rev voltage-dependent anion channel calcium signaling protein protein mesh keyword books gene gene cell death christopher spontaneous cell death cell death mechanisms mice overexpressing cyclophilin necrotic cell death apoptotic cell death mitochondrial swelling prolyl isomerase located resources nature ppif gene product ppif null mice mitochondrial permeability transition inhibiting oxidative stress cell death cardiac mitochondria ppif protein similar articles cited ppif null fibroblasts improving mitochondrial dysfunction nat cell biol mol cell cardiol research support mitochondria isolated outer membrane rupture united states government adenine nucleotide translocator focal cerebral ischemia biochim biophys acta medgen nucleotide omim biomedical research knockout mice mitochondria fibroblasts isolated mitochondrial matrix permeability transition cell rep abstract oxidant stress

External Links {🔗}(87)

Analytics and Tracking {📊}

  • Google Analytics
  • Google Analytics 4
  • Google Tag Manager

Libraries {📚}

  • Clipboard.js
  • jQuery

CDN Services {📦}

  • Ncbi

3.89s.