We are analyzing https://link.springer.com/article/10.1186/s12943-024-01934-y.

Title:
Selective autophagy in cancer: mechanisms, therapeutic implications, and future perspectives | Molecular Cancer
Description:
Eukaryotic cells engage in autophagy, an internal process of self-degradation through lysosomes. Autophagy can be classified as selective or non-selective depending on the way it chooses to degrade substrates. During the process of selective autophagy, damaged and/or redundant organelles like mitochondria, peroxisomes, ribosomes, endoplasmic reticulum (ER), lysosomes, nuclei, proteasomes, and lipid droplets are selectively recycled. Specific cargo is delivered to autophagosomes by specific receptors, isolated and engulfed. Selective autophagy dysfunction is closely linked with cancers, neurodegenerative diseases, metabolic disorders, heart failure, etc. Through reviewing latest research, this review summarized molecular markers and important signaling pathways for selective autophagy, and its significant role in cancers. Moreover, we conducted a comprehensive analysis of small-molecule compounds targeting selective autophagy for their potential application in anti-tumor therapy, elucidating the underlying mechanisms involved. This review aims to supply important scientific references and development directions for the biological mechanisms and drug discovery of anti-tumor targeting selective autophagy in the future.
Website Age:
28 years and 1 months (reg. 1997-05-29).

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Science
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Custom-built

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🌠 Phenomenal Traffic: 5M - 10M visitors per month

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Keywords {🔍}

pubmed, autophagy, cancer, article, google, scholar, cas, cell, cells, mitophagy, central, protein, selective, erphagy, atg, death, mitochondrial, proteins, mitochondria, anticancer, xenophagy, role, membrane, stress, drug, wang, lipid, targeting, development, receptor, promotes, process, effect, lysosomal, biol, famb, chen, liu, receptors, resistance, tumor, lipophagy, degradation, expression, parkin, fig, zhang, domain, mol, lysosomes,

Topics {✒️}

skp1/cul1/f-box protein parkin-ubiquitin-p62-mediated mitochondrial priming branched-chain fatty acyl-coas promotes pink1-prkn/parkin-dependent mitophagy high alpha-methylacyl-coa racemase rab10-ehbp1-ehd2 complex essential hypoxia-inducible factor 1-alpha ifn-gamma-inducible lrg-47 alk-independent anti-cancer mechanism ubiquitin-conjugating enzyme e2q adherent-invasive escherichia coli n-terminal phox-bem1 domain inhibits parkin/pink1-mediated mitophagy atm serine/threonine kinase alpha-methylacyl-coa racemase ring-ch-type finger 5 article download pdf molecular cell biology small molecule compounds srebp-2/pnpla8 axis improves gamma-aminobutyric acid receptor influence parkin/pink1-mediated mitophagy targeting 6-phosphofructo-2-kinase/fructose-2 cardiac-expressed death factors induce fam134b-mediated er-phagy initiates fundc1-mediated autophagy wild-type mz-54 cells bcl2l1-pgam5-fundc1 axis plays soluble er-phagy receptor mitochondrial iron-mediated immunometabolism exploiting nanomaterial-mediated autophagy reducing nod1-dependent activation perk-eif2 signaling pathway anchorage-dependent cells split her2-positive breast cancer sirt1-foxo3-bnip3 axis acyl-coa binding protein dnm1l-mediated mitochondrial fission huntingtin-induced cell death pexophagic receptor-protein complex tumor-related signaling pathways double-membrane spherical vesicles peroxisome proliferator-activated receptors age-related macular degeneration parl-pgam5-pink1 axis lop-induced er-phagy akt/mtor signaling pathways pink1-prkn-mediated mitophagy salmonella-targeted xenophagy blockade acyl-coa-binding domain

Questions {❓}

How the Warburg effect supports aggressiveness and drug resistance of cancer cells?
Metabolic adaption of cancer cells toward autophagy: is there a role for ER-phagy?

Schema {🗺️}

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      name:Innovation Center of Nursing Research, Nursing Key Laboratory of Sichuan Province, State Key Laboratory of Biotherapy and Cancer Center, National Clinical Research Center for Geriatrics, West China Hospital, Sichuan University /West China School of Nursing, Sichuan University, Chengdu, China
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      name:Innovation Center of Nursing Research, Nursing Key Laboratory of Sichuan Province, State Key Laboratory of Biotherapy and Cancer Center, National Clinical Research Center for Geriatrics, West China Hospital, Sichuan University /West China School of Nursing, Sichuan University, Chengdu, China
      name:Innovation Center of Nursing Research, Nursing Key Laboratory of Sichuan Province, State Key Laboratory of Biotherapy and Cancer Center, National Clinical Research Center for Geriatrics, West China Hospital, Sichuan University /West China School of Nursing, Sichuan University, Chengdu, China
      name:Innovation Center of Nursing Research, Nursing Key Laboratory of Sichuan Province, State Key Laboratory of Biotherapy and Cancer Center, National Clinical Research Center for Geriatrics, West China Hospital, Sichuan University /West China School of Nursing, Sichuan University, Chengdu, China
      name:Innovation Center of Nursing Research, Nursing Key Laboratory of Sichuan Province, State Key Laboratory of Biotherapy and Cancer Center, National Clinical Research Center for Geriatrics, West China Hospital, Sichuan University /West China School of Nursing, Sichuan University, Chengdu, China

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