Here's how LINK.SPRINGER.COM makes money* and how much!

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LINK . SPRINGER . COM {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Link.springer.com Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
  12. Libraries
  13. CDN Services

We are analyzing https://link.springer.com/article/10.1007/s13277-015-3762-y.

Title:
Metabolic reprogramming as a continuous changing behavior of tumor cells | Tumor Biology
Description:
Malignant cells resist microenvironment stress and migrate into surrounding tissues in order to divide with the need to adapt their metabolic program. These changes, often strengthened by the tremendous liaison between hypoxia, low glucose, and acidosis, are not yet completely understood. The aim of this perspective is to re-organize a possible comprehensive scenario useful to identify the metabolism occurring in various tumor cell subpopulations endowed with different capabilities.
Website Age:
28 years and 1 months (reg. 1997-05-29).

Matching Content Categories {๐Ÿ“š}

  • Health & Fitness
  • Science
  • Education

Content Management System {๐Ÿ“}

What CMS is link.springer.com built with?

Custom-built

No common CMS systems were detected on Link.springer.com, and no known web development framework was identified.

Traffic Estimate {๐Ÿ“ˆ}

What is the average monthly size of link.springer.com audience?

๐ŸŒ  Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 5,000,019 visitors per month in the current month.
However, some sources were not loaded, we suggest to reload the page to get complete results.

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How Does Link.springer.com Make Money? {๐Ÿ’ธ}

We can't figure out the monetization strategy.

Not every website is profit-driven; some are created to spread information or serve as an online presence. Websites can be made for many reasons. This could be one of them. Link.springer.com might be making money, but it's not detectable how they're doing it.

Keywords {๐Ÿ”}

pubmed, article, cancer, google, scholar, cas, tumor, metabolism, cells, central, cell, metabolic, hypoxia, privacy, cookies, reprogramming, calorini, nature, rev, content, publish, search, peppicelli, bianchini, access, glycolysis, nat, metastasis, data, information, log, journal, research, july, lido, microenvironment, discover, warburg, aerobic, experimental, res, biol, effect, lactate, download, author, springer, essential, function, optional,

Topics {โœ’๏ธ}

month download article/chapter francesca bianchiniย &ย lido calorini amp-activated protein kinase hypoxia-inducible factor 1alpha ancient energy gauge reciprocal metabolic reprogramming acidic extracellular microenvironment full article pdf privacy choices/manage cookies metabolic reprogramming article peppicelli enforce tumor regression tumor metabolism european economic area continuous changing behavior anomalous gene expression clowes memorial lecture antioxid redox signal gonzalez herrera kn istituto toscano tumori cancer cells give cancer metabolism nat rev cancer conditions privacy policy pericellular ph homeostasis clinical biomedical sciences cancer cell int mol cell biol nat cell biol cell cycle arrest cancer metastasis rev tumor stroma article log accepting optional cookies metabolism occurring glutamine metabolism intermediary metabolism glucose metabolism lido calorini main content log author information authors article cite high aerobic glycolysis reverse warburg effect journal finder publish tumor cells metabolic program metabolic systems check access gillies rj

Questions {โ“}

  • Why do cancers have high aerobic glycolysis?

Schema {๐Ÿ—บ๏ธ}

WebPage:
      mainEntity:
         headline:Metabolic reprogramming as a continuous changing behavior of tumor cells
         description:Malignant cells resist microenvironment stress and migrate into surrounding tissues in order to divide with the need to adapt their metabolic program. These changes, often strengthened by the tremendous liaison between hypoxia, low glucose, and acidosis, are not yet completely understood. The aim of this perspective is to re-organize a possible comprehensive scenario useful to identify the metabolism occurring in various tumor cell subpopulations endowed with different capabilities.
         datePublished:2015-07-10T00:00:00Z
         dateModified:2015-07-10T00:00:00Z
         pageStart:5759
         pageEnd:5762
         sameAs:https://doi.org/10.1007/s13277-015-3762-y
         keywords:
            Cancer metabolism
            Acidic microenvironment
            Hypoxia
            Cancer Research
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         isPartOf:
            name:Tumor Biology
            issn:
               1423-0380
               1010-4283
            volumeNumber:36
            type:
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               PublicationVolume
         publisher:
            name:Springer Netherlands
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               type:ImageObject
            type:Organization
         author:
               name:Silvia Peppicelli
               affiliation:
                     name:Florence University
                     address:
                        name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
                        type:PostalAddress
                     type:Organization
               type:Person
               name:Francesca Bianchini
               affiliation:
                     name:Florence University
                     address:
                        name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
                        type:PostalAddress
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               name:Lido Calorini
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                     name:Florence University
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                        name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
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      headline:Metabolic reprogramming as a continuous changing behavior of tumor cells
      description:Malignant cells resist microenvironment stress and migrate into surrounding tissues in order to divide with the need to adapt their metabolic program. These changes, often strengthened by the tremendous liaison between hypoxia, low glucose, and acidosis, are not yet completely understood. The aim of this perspective is to re-organize a possible comprehensive scenario useful to identify the metabolism occurring in various tumor cell subpopulations endowed with different capabilities.
      datePublished:2015-07-10T00:00:00Z
      dateModified:2015-07-10T00:00:00Z
      pageStart:5759
      pageEnd:5762
      sameAs:https://doi.org/10.1007/s13277-015-3762-y
      keywords:
         Cancer metabolism
         Acidic microenvironment
         Hypoxia
         Cancer Research
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs13277-015-3762-y/MediaObjects/13277_2015_3762_Fig1_HTML.gif
      isPartOf:
         name:Tumor Biology
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            1423-0380
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                     type:PostalAddress
                  type:Organization
            type:Person
            name:Francesca Bianchini
            affiliation:
                  name:Florence University
                  address:
                     name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
                     type:PostalAddress
                  type:Organization
            type:Person
            name:Lido Calorini
            affiliation:
                  name:Florence University
                  address:
                     name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
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         name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
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               name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
               type:PostalAddress
            type:Organization
      name:Francesca Bianchini
      affiliation:
            name:Florence University
            address:
               name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
               type:PostalAddress
            type:Organization
      name:Lido Calorini
      affiliation:
            name:Florence University
            address:
               name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
               type:PostalAddress
            type:Organization
      email:[email protected]
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      name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
      name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
      name:Department of Experimental and Clinical Biomedical Sciences, Section of Experimental Pathology and Oncology, Florence University, Florence, Italy
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External Links {๐Ÿ”—}(102)

Analytics and Tracking {๐Ÿ“Š}

  • Google Tag Manager

Libraries {๐Ÿ“š}

  • Clipboard.js
  • Prism.js

CDN Services {๐Ÿ“ฆ}

  • Crossref

4.08s.