We are analyzing https://link.springer.com/article/10.1007/s10565-019-09496-2.

Title:
Cell death mechanisms in eukaryotes | Cell Biology and Toxicology
Description:
Like the organism they constitute, the cells also die in different ways. The death can be predetermined, programmed, and cleanly executed, as in the case of apoptosis, or it can be traumatic, inflammatory, and sudden as many types of necrosis exemplify. Nevertheless, there are a number of cell deaths—some of them bearing a resemblance to apoptosis and/or necrosis, and many, distinct from each—that serve a multitude of roles in either supporting or disrupting the homoeostasis. Apoptosis is coordinated by death ligands, caspases, b-cell lymphoma-2 (Bcl-2) family proteins, and their downstream effectors. Events that can lead to apoptosis include mitotic catastrophe and anoikis. Necrosis, although it has been considered an abrupt and uncoordinated cell death, has many molecular events associated with it. There are cell death mechanisms that share some standard features with necrosis. These include methuosis, necroptosis, NETosis, pyronecrosis, and pyroptosis. Autophagy, generally a catabolic pathway that operates to ensure cell survival, can also kill the cell through mechanisms such as autosis. Other cell-death mechanisms include entosis, ferroptosis, lysosome-dependent cell death, and parthanatos.
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28 years and 1 months (reg. 1997-05-29).

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Keywords {🔍}

cell, death, pubmed, article, google, scholar, cas, apoptosis, cells, central, cancer, necrosis, mechanisms, sci, biol, autophagy, mol, zhang, necroptosis, ferroptosis, mitochondrial, immunol, human, signaling, differ, entosis, apoptotic, activation, induces, caspase, liu, programmed, molecular, methuosis, pyroptosis, induction, methods, permeability, chem, rep, res, protein, stress, med, wang, inflammatory, pathway, autosis, parthanatos, access,

Topics {✒️}

ros–er–calcium–mitochondria signal pathway reactive oxygen species methuosis-inducing indole-based chalcones grace nirmala & manu lopus month download article/chapter hydrogen peroxide-induced apoptosis valent iron-based nanotherapeutics chalcone-related small molecule lysosome-dependent cell death radiation-induced mitotic catastrophe aβ-induced cell death interleukin-1β-producing monocytes ferrous-supply-regeneration nanoengineering phospho-ripk3 immunohistochemical labeling nonapoptotic cell death necroptosis-dependent signaling pathway granzyme b-mediated apoptosis human tumor cells imaging-guided photodynamic therapy ischemia/reperfusion-induced damage article cell biology cathepsin b-independent necrosis factor 2-dependent mechanism tnf receptor superfamilies full article pdf mitochondrial permeability transition human macrophages infected membrane permeability transition de oliveira si mediated necrosis independently ferroptotic cell death cell death induced necrotic cell death cancer-cell-specific ferroptosis article nirmala ripk3-dependent necroptosis calpain-mediated cleavage endoplasmic reticulum stress programmed cell death mitochondrial calcium overload privacy choices/manage cookies diverse species silmitasertib promotes methuosis lysosomal cell death measuring er stress neutrophil extracellular trap regulated cell death cell death dis cell death mechanisms propofol inhibits parthanatos

Questions {❓}

NETosis: how vital is it?

Schema {🗺️}

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