We are analyzing https://link.springer.com/article/10.1007/s00018-017-2626-6.

Title:
Interaction and cross-talk between non-coding RNAs | Cellular and Molecular Life Sciences
Description:
Non-coding RNA (ncRNA) has been shown to regulate diverse cellular processes and functions through controlling gene expression. Long non-coding RNAs (lncRNAs) act as a competing endogenous RNAs (ceRNAs) where microRNAs (miRNAs) and lncRNAs regulate each other through their biding sites. Interactions of miRNAs and lncRNAs have been reported to trigger decay of the targeted lncRNAs and have important roles in target gene regulation. These interactions form complicated and intertwined networks. Certain lncRNAs encode miRNAs and small nucleolar RNAs (snoRNAs), and may regulate expression of these small RNAs as precursors. SnoRNAs have also been reported to be precursors for PIWI-interacting RNAs (piRNAs) and thus may regulate the piRNAs as a precursor. These miRNAs and piRNAs target messenger RNAs (mRNAs) and regulate gene expression. In this review, we will present and discuss these interactions, cross-talk, and co-regulation of ncRNAs and gene regulation due to these interactions.
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Keywords {🔍}

pubmed, article, google, scholar, cas, rna, central, noncoding, cancer, lncrna, cerna, rnas, cell, mirnas, long, mirna, expression, target, human, cells, res, binding, gene, wang, snornas, function, functions, interactions, small, interaction, lncrnas, regulation, found, zhang, table, liu, tumor, breast, microrna, hotair, snorna, sites, reported, ncrnas, mirp, targets, nucleic, acids, sequencing, protein,

Topics {✒️}

yuichiro tanaka & rajvir dahiya inhibit ras-erk1/erk2 signaling lncrna uca1-mir-507-foxm1 axis crispr/cas9-grna complex system rnai-mediated allelic trans-interaction mir-181b-12/15-lox signaling pathway site-specifically recruiting mll/compass generate piwi-interacting rnas article download pdf stress-response rna-binding protein pre-mir-133b hairpin structure lincrna h19–mir-106a interaction high-throughput rna sequencing high-throughput sequencing methodologies tumour suppressive mir-125b piwi-interacting rnas estrogen receptor-independent manner trf4–air2–mtr4 polyadenylation lincrna-ror induces epithelial negatively regulates mirna-130a interrupt gls1-mediated glutaminolysis c-myc activated driver mir-17-5p family member imprinted rtl1/peg11 locus trna-derived small rnas mir-148a regulates meg3 protein-coding rnas making human interleukin-4 pre-mrna malat1-mir-124-rbg2 axis mirna-mediated gene silencing rna-induced silencing complex mtor-stat3/microrna143 pathway mir-125a-5p decreases photoactivatable-ribonucleoside-enhanced crosslinking influence mirna-mediated repression snord-host rna zfas1 lncrna h19-derived mir-675 activate muscle-specific genes competing endogenous rna gwas-based association analyses exhibits pro-oncogenic activity small nucleolar rnas lincrna-nr_024015 exon identified triple-negative breast cancer hmga1p7-pseudogene regulates h19 documenting mirna–lncrna interactions studies including h19/mir-675 regulate lipid metabolism direct mirna–lncrna interactions direct mirna–lncrna interactions”

Questions {❓}

Huarte M, Rinn JL (2010) Large non-coding RNAs: missing links in cancer?

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Interaction and cross-talk between non-coding RNAs
         description:Non-coding RNA (ncRNA) has been shown to regulate diverse cellular processes and functions through controlling gene expression. Long non-coding RNAs (lncRNAs) act as a competing endogenous RNAs (ceRNAs) where microRNAs (miRNAs) and lncRNAs regulate each other through their biding sites. Interactions of miRNAs and lncRNAs have been reported to trigger decay of the targeted lncRNAs and have important roles in target gene regulation. These interactions form complicated and intertwined networks. Certain lncRNAs encode miRNAs and small nucleolar RNAs (snoRNAs), and may regulate expression of these small RNAs as precursors. SnoRNAs have also been reported to be precursors for PIWI-interacting RNAs (piRNAs) and thus may regulate the piRNAs as a precursor. These miRNAs and piRNAs target messenger RNAs (mRNAs) and regulate gene expression. In this review, we will present and discuss these interactions, cross-talk, and co-regulation of ncRNAs and gene regulation due to these interactions.
         datePublished:2017-08-24T00:00:00Z
         dateModified:2017-08-24T00:00:00Z
         pageStart:467
         pageEnd:484
         license:http://creativecommons.org/licenses/by/4.0/
         sameAs:https://doi.org/10.1007/s00018-017-2626-6
         keywords:
            Long non-coding RNA
            Circular RNA
            MicroRNA
            Competing endogenous RNA
            Interaction
            Small nucleolar RNA
            PIWI-interacting RNAs
            Cell Biology
            Biomedicine
            general
            Life Sciences
            Biochemistry
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                     address:
                        name:Department of Urology, University of California, San Francisco, San Francisco, USA
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ScholarlyArticle:
      headline:Interaction and cross-talk between non-coding RNAs
      description:Non-coding RNA (ncRNA) has been shown to regulate diverse cellular processes and functions through controlling gene expression. Long non-coding RNAs (lncRNAs) act as a competing endogenous RNAs (ceRNAs) where microRNAs (miRNAs) and lncRNAs regulate each other through their biding sites. Interactions of miRNAs and lncRNAs have been reported to trigger decay of the targeted lncRNAs and have important roles in target gene regulation. These interactions form complicated and intertwined networks. Certain lncRNAs encode miRNAs and small nucleolar RNAs (snoRNAs), and may regulate expression of these small RNAs as precursors. SnoRNAs have also been reported to be precursors for PIWI-interacting RNAs (piRNAs) and thus may regulate the piRNAs as a precursor. These miRNAs and piRNAs target messenger RNAs (mRNAs) and regulate gene expression. In this review, we will present and discuss these interactions, cross-talk, and co-regulation of ncRNAs and gene regulation due to these interactions.
      datePublished:2017-08-24T00:00:00Z
      dateModified:2017-08-24T00:00:00Z
      pageStart:467
      pageEnd:484
      license:http://creativecommons.org/licenses/by/4.0/
      sameAs:https://doi.org/10.1007/s00018-017-2626-6
      keywords:
         Long non-coding RNA
         Circular RNA
         MicroRNA
         Competing endogenous RNA
         Interaction
         Small nucleolar RNA
         PIWI-interacting RNAs
         Cell Biology
         Biomedicine
         general
         Life Sciences
         Biochemistry
      image:
         https://media.springernature.com/lw1200/springer-static/image/art%3A10.1007%2Fs00018-017-2626-6/MediaObjects/18_2017_2626_Fig1_HTML.gif
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         name:Springer International Publishing
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            type:ImageObject
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            url:http://orcid.org/0000-0002-1660-156X
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                     name:Department of Urology, University of California, San Francisco, San Francisco, USA
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                     name:Department of Urology, University of California, San Francisco, San Francisco, USA
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                  address:
                     name:Department of Urology, University of California, San Francisco, San Francisco, USA
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                  address:
                     name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
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      affiliation:
            name:University of California, San Francisco
            address:
               name:Department of Urology, University of California, San Francisco, San Francisco, USA
               type:PostalAddress
            type:Organization
            name:San Francisco Veterans Affairs Medical Center
            address:
               name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
               type:PostalAddress
            type:Organization
      email:[email protected]
      name:Mitsuho Imai-Sumida
      affiliation:
            name:University of California, San Francisco
            address:
               name:Department of Urology, University of California, San Francisco, San Francisco, USA
               type:PostalAddress
            type:Organization
            name:San Francisco Veterans Affairs Medical Center
            address:
               name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
               type:PostalAddress
            type:Organization
      name:Yuichiro Tanaka
      affiliation:
            name:University of California, San Francisco
            address:
               name:Department of Urology, University of California, San Francisco, San Francisco, USA
               type:PostalAddress
            type:Organization
            name:San Francisco Veterans Affairs Medical Center
            address:
               name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
               type:PostalAddress
            type:Organization
      name:Rajvir Dahiya
      affiliation:
            name:University of California, San Francisco
            address:
               name:Department of Urology, University of California, San Francisco, San Francisco, USA
               type:PostalAddress
            type:Organization
            name:San Francisco Veterans Affairs Medical Center
            address:
               name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Department of Urology, University of California, San Francisco, San Francisco, USA
      name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
      name:Department of Urology, University of California, San Francisco, San Francisco, USA
      name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
      name:Department of Urology, University of California, San Francisco, San Francisco, USA
      name:San Francisco Veterans Affairs Medical Center, San Francisco, USA
      name:Department of Urology, University of California, San Francisco, San Francisco, USA
      name:San Francisco Veterans Affairs Medical Center, San Francisco, USA

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