We are analyzing https://link.springer.com/chapter/10.1007/978-4-431-56847-6_18.

Title:
Future of Muse Cells | SpringerLink
Description:
Discovered nearly 10 years ago by Professor Mari Dezawa and her colleagues, Muse cells are entering clinical trials faster than any other stem cell for three reasons. First, Muse cells have multiple fail-safe mechanisms to keep themselves from growing out of...
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ScholarlyArticle:
      headline:Future of Muse Cells
      pageEnd:315
      pageStart:309
      image:https://media.springernature.com/w153/springer-static/cover/book/978-4-431-56847-6.jpg
      genre:
         Biomedical and Life Sciences
         Biomedical and Life Sciences (R0)
      isPartOf:
         name:Muse Cells
         isbn:
            978-4-431-56847-6
            978-4-431-56845-2
         type:Book
      publisher:
         name:Springer Japan
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:Wise Young
            affiliation:
                  name:Rutgers, State University of New Jersey
                  address:
                     name:W. M. Keck Center for Collaborative Neuroscience, Rutgers, State University of New Jersey, Piscataway, USA
                     type:PostalAddress
                  type:Organization
            type:Person
      keywords:Muse, HLA-G, IDO, Tumor, Anti-immune, Tissue repair
      description:Discovered nearly 10 years ago by Professor Mari Dezawa and her colleagues, Muse cells are entering clinical trials faster than any other stem cell for three reasons. First, Muse cells have multiple fail-safe mechanisms to keep themselves from growing out of control and do not form tumors. In contrast, embryonic stem cells and induced pluripotent stem cells form tumors and must be differentiated before transplantation. Second, Muse cells possess potent anti-immune mechanisms, including human leukocyte antigen G and indoleamine 2,3-dioxygenase that prevent both cellular and humoral immunity. Muse cells engraft even though they do not match HLA antigens with the host. Third, Muse cells are able to determine what kind and how many cells they need to make for tissue repair. While the mechanisms responsible for these traits are not well understood, Muse cells are able to enter severely injured tissues of all kinds and repair them. Study of mechanisms underlying these traits of Muse cells is likely to yield new therapies for cancer prevention, autoimmune diseases, and repair of injured tissues. The future is bright for Muse cells.
      datePublished:2018
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Book:
      name:Muse Cells
      isbn:
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         978-4-431-56845-2
Organization:
      name:Springer Japan
      logo:
         url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
         type:ImageObject
      name:Rutgers, State University of New Jersey
      address:
         name:W. M. Keck Center for Collaborative Neuroscience, Rutgers, State University of New Jersey, Piscataway, USA
         type:PostalAddress
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Person:
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      affiliation:
            name:Rutgers, State University of New Jersey
            address:
               name:W. M. Keck Center for Collaborative Neuroscience, Rutgers, State University of New Jersey, Piscataway, USA
               type:PostalAddress
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