GENESDEV . ORG {
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We began analyzing https://genesdev.cshlp.org/content/27/7/725, but it redirected us to https://genesdev.cshlp.org/content/27/7/725. The analysis below is for the second page.
Title[redir]:
MYC/MAX control ERK signaling and pluripotency by regulation of dual-specificity phosphatases 2 and 7
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields
Title[redir]:
MYC/MAX control ERK signaling and pluripotency by regulation of dual-specificity phosphatases 2 and 7
Description:
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fields
Matching Content Categories {📚}
- Science
- Education
- News & Politics
Content Management System {📝}
What CMS is genesdev.org built with?
Custom-built
No common CMS systems were detected on Genesdev.org, and no known web development framework was identified.
Traffic Estimate {📈}
What is the average monthly size of genesdev.org audience?
🚄 Respectable Traffic: 10k - 20k visitors per month
Based on our best estimate, this website will receive around 10,019 visitors per month in the current month.
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How Does Genesdev.org Make Money? {💸}
We can't see how the site brings in money.
Not all websites focus on profit; some are designed to educate, connect people, or share useful tools. People create websites for numerous reasons. And this could be one such example. Genesdev.org might be plotting its profit, but the way they're doing it isn't detectable yet.
Keywords {🔍}
erk, dusp, author, article, signaling, myc, submit, mycmax, pluripotency, activity, online, copyright, cold, spring, harbor, laboratory, press, home, subscribe, advertise, info, archive, contact, search, dualspecificity, phosphatases, georgia, maintenance, pluripotent, max, mechanism, complexes, binding, resulting, loss, mapkerk, pathway, email, supplemental, material, facebook, full, articles, issue, issn, skip, main, page, content, keyword,
Topics {✒️}
supplemental material myc/max complexes achieve extracellular signal-regulated kinase mapk/erk signaling pathway myc-binding partner max mapk/erk pathway /stat3 signaling reaches authors contributed equally dual-specificity phosphatase dephosphorylating residues required leukemia inhibitory factor org/cgi/doi/10 dual-specificity phosphatases 2 psc maintenance involving erk activity article doi 0890-9369 online issn article » table catalytic activity issue alert ectopic dusp2 max article erk search 7 james chappell1 yuhua sun1 amar singh molecular biology coverdell center health sciences abstract suppression absolute requirement transcriptionally regulating dusps function inactivating erk1 dusp7 transcriptional activation findings elucidate regulatory role directly inhibit accepted march 18 copyright © 2013 details tweeted myc signaling sustain pluripotency dusp2 online maintenance
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