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We began analyzing https://www.nature.com/articles/6695007, but it redirected us to https://www.nature.com/articles/6695007. The analysis below is for the second page.

Title[redir]:
Molecular cytogenetic analysis of 11 new breast cancer cell lines | British Journal of Cancer
Description:
We describe a survey of genetic changes by comparative genomic hybridization (CGH) in 11 human breast cancer cell lines recently established in our laboratory. The most common gains took place at 8q (73%), 1q (64%), 7q (64%), 3q (45%) and 7p (45%), whereas losses were most frequent at Xp (54%), 8p (45%), 18q (45%) and Xq (45%). Many of the cell lines displayed prominent, localized DNA amplifications by CGH. One-third of these loci affected breast cancer oncogenes, whose amplifications were validated with specific probes: 17q12 (two cell lines with ERBB2 amplifications), 11q13 (two with cyclin-D1), 8p11–p12 (two with FGFR1) and 10q25 (one with FGFR2). Gains and amplifications affecting 8q were the most common genetic alterations in these cell lines with the minimal, common region of involvement at 8q22–q23. No high-level MYC (at 8q24) amplifications were found in any of the cell lines. Two-thirds of the amplification sites took place at loci not associated with established oncogenes, such as 1q41–q43, 7q21–q22, 7q31, 8q23, 9p21–p23, 11p12–p14, 15q12–q14, 16q13–q21, 17q23, 20p11–p12 and 20q13. Several of these locations have not been previously reported and may harbour important genes whose amplification is selected for during cancer development. In summary, this set of breast cancer cell lines displaying prominent DNA amplifications should facilitate discovery and functional analysis of genes and signal transduction pathways contributing to breast cancer development.

Matching Content Categories {📚}

  • Education
  • Health & Fitness
  • Science

Content Management System {📝}

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Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🌠 Phenomenal Traffic: 5M - 10M visitors per month


Based on our best estimate, this website will receive around 9,135,289 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We can't tell how the site generates income.

Not all websites are made for profit; some exist to inform or educate users. Or any other reason why people make websites. And this might be the case. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

cancer, breast, article, google, scholar, cas, kallioniemi, human, cell, nature, ethier, lines, res, pubmed, amplification, growth, cells, content, genes, amplified, tanner, cookies, molecular, amplifications, privacy, analysis, open, comparative, genomic, hybridization, cgh, development, isola, karhu, gene, research, data, journal, access, forozan, ammerman, genetic, dna, carcinoma, usa, treat, press, tirkkonen, factor, advertising,

Topics {✒️}

nature portfolio privacy policy nature med 4 nature advertising 0/ reprints breast cancer development high-throughput molecular profiling c-erbb-2 gene amplification social media selective media sum-159pt cells personal data human breast cancer molecular cytogenetics breast cancer cells breast carcinoma cells molecular cytogenetic analysis primary breast cancer data protection cancer development human breast cancers permissions breast cancer based drosophila aurora kinase functional analysis human colorectal cancers dna sequence amplification privacy localized dna amplifications chromosomal gains cancer biology cell lines explore content similar content epidermal growth factor hormone receptor positive comparative genomic hybridization amplified dna sequences journals search log high-level myc tyrosine phosphorylated erbb-2 european economic area metstatic lymph node progressively elevated levels michigan medical school predicts poor outcome comparative transcriptional analyses amplification-related gain breast cancer

Schema {🗺️}

WebPage:
      mainEntity:
         headline:Molecular cytogenetic analysis of 11 new breast cancer cell lines
         description:We describe a survey of genetic changes by comparative genomic hybridization (CGH) in 11 human breast cancer cell lines recently established in our laboratory. The most common gains took place at 8q (73%), 1q (64%), 7q (64%), 3q (45%) and 7p (45%), whereas losses were most frequent at Xp (54%), 8p (45%), 18q (45%) and Xq (45%). Many of the cell lines displayed prominent, localized DNA amplifications by CGH. One-third of these loci affected breast cancer oncogenes, whose amplifications were validated with specific probes: 17q12 (two cell lines with ERBB2 amplifications), 11q13 (two with cyclin-D1), 8p11–p12 (two with FGFR1) and 10q25 (one with FGFR2). Gains and amplifications affecting 8q were the most common genetic alterations in these cell lines with the minimal, common region of involvement at 8q22–q23. No high-level MYC (at 8q24) amplifications were found in any of the cell lines. Two-thirds of the amplification sites took place at loci not associated with established oncogenes, such as 1q41–q43, 7q21–q22, 7q31, 8q23, 9p21–p23, 11p12–p14, 15q12–q14, 16q13–q21, 17q23, 20p11–p12 and 20q13. Several of these locations have not been previously reported and may harbour important genes whose amplification is selected for during cancer development. In summary, this set of breast cancer cell lines displaying prominent DNA amplifications should facilitate discovery and functional analysis of genes and signal transduction pathways contributing to breast cancer development.
         datePublished:1999-11-26T00:00:00Z
         dateModified:2011-11-16T00:00:00Z
         pageStart:1328
         pageEnd:1334
         sameAs:https://doi.org/10.1038/sj.bjc.6695007
         keywords:
            molecular cytogenetics
            FISH
            CGH
            chromosomal aberrations
            oncogene amplification
            Biomedicine
            general
            Cancer Research
            Epidemiology
            Molecular Medicine
            Oncology
            Drug Resistance
         image:
         isPartOf:
            name:British Journal of Cancer
            issn:
               1532-1827
               0007-0920
            volumeNumber:81
            type:
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               PublicationVolume
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         author:
               name:F Forozan
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                     address:
                        name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:R Veldman
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                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                     address:
                        name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                        type:PostalAddress
                     type:Organization
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               name:C A Ammerman
               affiliation:
                     name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
                     address:
                        name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:N Z Parsa
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                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                     address:
                        name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:A Kallioniemi
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                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                     address:
                        name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                        type:PostalAddress
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                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                     address:
                        name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                        type:PostalAddress
                     type:Organization
               type:Person
               name:S P Ethier
               affiliation:
                     name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
                     address:
                        name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
                        type:PostalAddress
                     type:Organization
               type:Person
         isAccessibleForFree:1
         type:ScholarlyArticle
      context:https://schema.org
ScholarlyArticle:
      headline:Molecular cytogenetic analysis of 11 new breast cancer cell lines
      description:We describe a survey of genetic changes by comparative genomic hybridization (CGH) in 11 human breast cancer cell lines recently established in our laboratory. The most common gains took place at 8q (73%), 1q (64%), 7q (64%), 3q (45%) and 7p (45%), whereas losses were most frequent at Xp (54%), 8p (45%), 18q (45%) and Xq (45%). Many of the cell lines displayed prominent, localized DNA amplifications by CGH. One-third of these loci affected breast cancer oncogenes, whose amplifications were validated with specific probes: 17q12 (two cell lines with ERBB2 amplifications), 11q13 (two with cyclin-D1), 8p11–p12 (two with FGFR1) and 10q25 (one with FGFR2). Gains and amplifications affecting 8q were the most common genetic alterations in these cell lines with the minimal, common region of involvement at 8q22–q23. No high-level MYC (at 8q24) amplifications were found in any of the cell lines. Two-thirds of the amplification sites took place at loci not associated with established oncogenes, such as 1q41–q43, 7q21–q22, 7q31, 8q23, 9p21–p23, 11p12–p14, 15q12–q14, 16q13–q21, 17q23, 20p11–p12 and 20q13. Several of these locations have not been previously reported and may harbour important genes whose amplification is selected for during cancer development. In summary, this set of breast cancer cell lines displaying prominent DNA amplifications should facilitate discovery and functional analysis of genes and signal transduction pathways contributing to breast cancer development.
      datePublished:1999-11-26T00:00:00Z
      dateModified:2011-11-16T00:00:00Z
      pageStart:1328
      pageEnd:1334
      sameAs:https://doi.org/10.1038/sj.bjc.6695007
      keywords:
         molecular cytogenetics
         FISH
         CGH
         chromosomal aberrations
         oncogene amplification
         Biomedicine
         general
         Cancer Research
         Epidemiology
         Molecular Medicine
         Oncology
         Drug Resistance
      image:
      isPartOf:
         name:British Journal of Cancer
         issn:
            1532-1827
            0007-0920
         volumeNumber:81
         type:
            Periodical
            PublicationVolume
      publisher:
         name:Nature Publishing Group UK
         logo:
            url:https://www.springernature.com/app-sn/public/images/logo-springernature.png
            type:ImageObject
         type:Organization
      author:
            name:F Forozan
            affiliation:
                  name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                  address:
                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:R Veldman
            affiliation:
                  name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                  address:
                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:C A Ammerman
            affiliation:
                  name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
                  address:
                     name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:N Z Parsa
            affiliation:
                  name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                  address:
                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:A Kallioniemi
            affiliation:
                  name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                  address:
                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:O-P Kallioniemi
            affiliation:
                  name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
                  address:
                     name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
                     type:PostalAddress
                  type:Organization
            type:Person
            name:S P Ethier
            affiliation:
                  name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
                  address:
                     name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
                     type:PostalAddress
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      name:British Journal of Cancer
      issn:
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      volumeNumber:81
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      name:Nature Publishing Group UK
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      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
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         name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
         type:PostalAddress
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
      address:
         name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
         type:PostalAddress
      name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
      address:
         name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
         type:PostalAddress
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
      address:
         name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
         type:PostalAddress
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
      address:
         name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
         type:PostalAddress
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
      address:
         name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
         type:PostalAddress
      name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
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            name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
            address:
               name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
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            name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
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               name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
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            name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
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               name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
               type:PostalAddress
            type:Organization
      name:N Z Parsa
      affiliation:
            name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
            address:
               name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
               type:PostalAddress
            type:Organization
      name:A Kallioniemi
      affiliation:
            name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
            address:
               name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
               type:PostalAddress
            type:Organization
      name:O-P Kallioniemi
      affiliation:
            name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health
            address:
               name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
               type:PostalAddress
            type:Organization
      name:S P Ethier
      affiliation:
            name:University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr
            address:
               name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
               type:PostalAddress
            type:Organization
PostalAddress:
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
      name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
      name:Cancer Genetics Branch, National Human Genome Research Institute, National Institutes of Health, Bethesda, USA
      name:Department of Radiation Oncology, Division of Radiation and Cancer Biology, University of Michigan Medical School, 7312 Cancer Center, 1500 E. Medical Center Dr, Ann Arbor, USA

Social Networks {👍}(1)

External Links {🔗}(181)

Analytics and Tracking {📊}

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Libraries {📚}

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  • Zoom.js

Emails and Hosting {✉️}

Mail Servers:

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Name Servers:

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CDN Services {📦}

  • Crossref

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