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We began analyzing https://www.nature.com/articles/ng.2667, but it redirected us to https://www.nature.com/articles/ng.2667. The analysis below is for the second page.

Title[redir]:
Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci | Nature Genetics
Description:
Matthew Brown and colleagues identify multiple susceptibility variants for ankylosing spondylitis through an association study based on high-density genotyping of immune-related loci. Their findings implicate numerous biological pathways in the pathogenesis of this disease and highlight shared risk factors with other autoimmune diseases. Ankylosing spondylitis is a common, highly heritable inflammatory arthritis affecting primarily the spine and pelvis. In addition to HLA-B*27 alleles, 12 loci have previously been identified that are associated with ankylosing spondylitis in populations of European ancestry, and 2 associated loci have been identified in Asians. In this study, we used the Illumina Immunochip microarray to perform a case-control association study involving 10,619 individuals with ankylosing spondylitis (cases) and 15,145 controls. We identified 13 new risk loci and 12 additional ankylosing spondylitis–associated haplotypes at 11 loci. Two ankylosing spondylitis–associated regions have now been identified encoding four aminopeptidases that are involved in peptide processing before major histocompatibility complex (MHC) class I presentation. Protective variants at two of these loci are associated both with reduced aminopeptidase function and with MHC class I cell surface expression.

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pubmed, article, google, scholar, cas, central, ankylosing, spondylitis, university, research, nature, arthritis, association, department, hospital, genet, study, rheumatology, nat, national, loci, medical, consortium, medicine, health, rheum, susceptibility, disease, genomewide, genetics, risk, variants, spondyloarthritis, wellcome, trust, funded, institute, supplementary, cell, access, diseases, canada, common, identifies, erap, australia, centre, norway, content, analysis,

Topics {✒️}

nature portfolio permissions reprints privacy policy alberta innovates–health solutions scientific research flanders australo-anglo-american spondyloarthritis consortium advertising jose luis fernandez-sueiro genome-wide meta-analysis increases carlos lopez-larrea van der horst-bruinsma regional visualization social media genome-wide association studies cedars-sinai medical center nature 489 nature 441 nature 488 nature 476 nature 467 nature 447 nature henri-jean garchon inger myrnes hansen bryan paul wordsworth & matthew genome-wide association study undergoes nonsense-mediated decay t-box proteins cooperate genome-wide association analysis cell-mediated immune mechanisms author information authors kyung bin joo spondyloarthritis research consortium raphael valle-oñate consuelo romero-sánchez tae-hwan kim hla-b27 positive patients hospital militar central final n-terminal trimming quantitative trait loci hla-b27 misfolding activates robust genotype-calling algorithm medical research council translational research institute cells lacking t-bet arthritis research uk population-based linkage analyses nord-trøndelag health study intramural research program national yang-ming university

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      description:Matthew Brown and colleagues identify multiple susceptibility variants for ankylosing spondylitis through an association study based on high-density genotyping of immune-related loci. Their findings implicate numerous biological pathways in the pathogenesis of this disease and highlight shared risk factors with other autoimmune diseases. Ankylosing spondylitis is a common, highly heritable inflammatory arthritis affecting primarily the spine and pelvis. In addition to HLA-B*27 alleles, 12 loci have previously been identified that are associated with ankylosing spondylitis in populations of European ancestry, and 2 associated loci have been identified in Asians. In this study, we used the Illumina Immunochip microarray to perform a case-control association study involving 10,619 individuals with ankylosing spondylitis (cases) and 15,145 controls. We identified 13 new risk loci and 12 additional ankylosing spondylitisâassociated haplotypes at 11 loci. Two ankylosing spondylitisâassociated regions have now been identified encoding four aminopeptidases that are involved in peptide processing before major histocompatibility complex (MHC) class I presentation. Protective variants at two of these loci are associated both with reduced aminopeptidase function and with MHC class I cell surface expression.
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