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DOI . ORG {}

  1. Analyzed Page
  2. Matching Content Categories
  3. CMS
  4. Monthly Traffic Estimate
  5. How Does Doi.org Make Money
  6. Keywords
  7. Topics
  8. Questions
  9. Schema
  10. External Links
  11. Analytics And Tracking
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We began analyzing https://link.springer.com/article/10.1007/s10875-015-0147-3, but it redirected us to https://link.springer.com/article/10.1007/s10875-015-0147-3. The analysis below is for the second page.

Title[redir]:
Human Disease Phenotypes Associated With Mutations in TREX1 | Journal of Clinical Immunology
Description:
Considering that it is a single exon gene encoding a 314 amino acid protein, the genotype-phenotype landscape of TREX1 is remarkably complex. Here we briefly describe the human diseases so-far associated with mutations in TREX1, which include Aicardi-Goutières syndrome, familial chilblain lupus, systemic lupus erythematosus and retinal vasculopathy with cerebral leukodystrophy.

Matching Content Categories {📚}

  • Education
  • Science
  • Health & Fitness

Content Management System {📝}

What CMS is doi.org built with?

Custom-built

No common CMS systems were detected on Doi.org, and no known web development framework was identified.

Traffic Estimate {📈}

What is the average monthly size of doi.org audience?

🏙️ Massive Traffic: 50M - 100M visitors per month


Based on our best estimate, this website will receive around 80,479,999 visitors per month in the current month.

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How Does Doi.org Make Money? {💸}

We don’t know how the website earns money.

While many websites aim to make money, others are created to share knowledge or showcase creativity. People build websites for various reasons. This could be one of them. Doi.org could be secretly minting cash, but we can't detect the process.

Keywords {🔍}

pubmed, article, google, scholar, cas, trex, lupus, syndrome, central, aicardigoutieres, mutations, chilblain, crow, exonuclease, familial, disease, mutation, dna, human, systemic, erythematosus, vasculopathy, med, autoimmune, genet, cerebral, leekirsch, rice, gene, immunol, access, biol, perrino, nat, dominant, research, chem, cell, privacy, cookies, content, journal, clinical, retinal, leukodystrophy, harvey, hum, samhd, hollis, heterozygous,

Topics {✒️}

electronic supplementary material month download article/chapter include aicardi-goutières syndrome rare variants aicardi-goutieres syndrome masquerading aicardi-goutieres syndrome–report interferon-dependent autoimmune disease gene-targeted mice lacking dominant aicardi-goutieres syndrome systemic lupus erythematosus chilblain lupus erythematosus interferon-alpha reveals molecular central nervous system familial chilblain lupus familial chilblain lupus lee-kirsch ma full article pdf astrocytes produce interferon-alpha cutaneous lupus erythematosus stetson db aicardi-goutieres syndrome human disease phenotypes privacy choices/manage cookies van steensel ma severe chilblain lupus clinical immunology aims article journal c-terminal truncations dna degradation activity biomed res int trex1 exonuclease gene state subsidy managed cerebral leukodystrophy access national research agency dna degradation revealed japanese cohort study van den maagdenberg european research council crow yj article rice interferon-related biomarkers mazur dj autosomal dominant inheritance cell biochem biophys interferon-independent activation early hum dev polyarteritis nodosa vasculopathy related subjects autoimmune diseases machine learning

Questions {❓}

  • Aicardi-goutieres syndrome: a genetic microangiopathy?

Schema {🗺️}

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         headline:Human Disease Phenotypes Associated With Mutations in TREX1
         description:Considering that it is a single exon gene encoding a 314 amino acid protein, the genotype-phenotype landscape of TREX1 is remarkably complex. Here we briefly describe the human diseases so-far associated with mutations in TREX1, which include Aicardi-Goutières syndrome, familial chilblain lupus, systemic lupus erythematosus and retinal vasculopathy with cerebral leukodystrophy.
         datePublished:2015-03-04T00:00:00Z
         dateModified:2015-03-04T00:00:00Z
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            Aicardi-Goutières syndrome
            Familial chilblain lupus
            Systemic lupus erythematosus
            Retinal vasculopathy with cerebral leukodystrophy
            Immunology
            Infectious Diseases
            Internal Medicine
            Medical Microbiology
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      headline:Human Disease Phenotypes Associated With Mutations in TREX1
      description:Considering that it is a single exon gene encoding a 314 amino acid protein, the genotype-phenotype landscape of TREX1 is remarkably complex. Here we briefly describe the human diseases so-far associated with mutations in TREX1, which include Aicardi-Goutières syndrome, familial chilblain lupus, systemic lupus erythematosus and retinal vasculopathy with cerebral leukodystrophy.
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         Aicardi-Goutières syndrome
         Familial chilblain lupus
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         Infectious Diseases
         Internal Medicine
         Medical Microbiology
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                     name:INSERM UMR 1163, Laboratory of Neurogenetics and Neuroinflammation, Institut Imagine, Hôpital Necker, Paris Descartes – Sorbonne Paris Cité University, Paris, France
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            name:Paris Descartes – Sorbonne Paris Cité University
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            name:University of Manchester
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               name:Manchester Centre for Genomic Medicine, Institute of Human Development Faculty of Medical and Human Sciences, Manchester Academic Health Sciences Centre, University of Manchester, Manchester, UK
               type:PostalAddress
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            name:Paris Descartes – Sorbonne Paris Cité University
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      name:Manchester Centre for Genomic Medicine, Institute of Human Development Faculty of Medical and Human Sciences, Manchester Academic Health Sciences Centre, University of Manchester, Manchester, UK
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External Links {🔗}(286)

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Libraries {📚}

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Emails and Hosting {✉️}

Mail Servers:

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